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Research projects (13000 - 13500 of 26984)

Research projects (13000 - 13500 of 26984)

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• Jan Vossaert<br />

Tuition fee voor o.a. Bayan Sapargaliyeva<br />

Vrije Universiteit Brussel<br />

Abstract: Medicine - Pharmacy<br />

Organisations:<br />

• Human Ecology<br />

<strong>Research</strong>ers:<br />

• David PROOT<br />

• LUC HENS<br />

Mechanisms <strong>of</strong> cross protection against H3N2 influenza virus in pig and human (FLUCROSS)<br />

Ghent University<br />

Abstract: It is studied whether immunity obtained by previous infection or vaccination with a human or swine influenza virus protects against<br />

subsequent infection with antigenically related or unrelated swine influenza virus. We focus on H3N2 swine influenza virus, a likely candidate for a<br />

future pandemic. Furthermore, we aim to improve the interpretation <strong>of</strong> serological tests against swine influenza viruses in humans.<br />

Organisations:<br />

• Departement <strong>of</strong> Virology, parasitology and immunology<br />

<strong>Research</strong>ers:<br />

• Eric Cox<br />

• Kristien Van Reeth<br />

Record <strong>of</strong> fees and compensations: judicial DNA-research<br />

Universiteit Hasselt<br />

Abstract: This project represents a formal service agreement between the parties BIOMED (research institute at Hasselt University), and on the<br />

other hand, 'het Gerecht van Tongeren'. BIOMED provides 'het Gerecht van Tongeren' research results on the judicial DNA-research under the<br />

conditions as stipulated in the present contract.<br />

The work covers all states <strong>of</strong> the month March 2010<br />

Organisations:<br />

• Immunology - Biochemistry<br />

• Biomedical <strong>Research</strong> Institute<br />

<strong>Research</strong>ers:<br />

• Luc MICHIELS<br />

Immunogenicity and Safety <strong>of</strong> the Influenza Vaccine (Split Virion, Inactivated), Northern Hemisphere 2010-2011<br />

Formulation (Intradermal Route).<br />

University <strong>of</strong> Antwerp<br />

Abstract: this is a vaccin trial in 65 healthy subjects, with a intradermal injection <strong>of</strong> a flu vaccine; Composition <strong>of</strong> the flu vaccins corresponds with the<br />

recommended composition by the WHO for the winter 2010-2011.<br />

Organisations:<br />

• VAXINFECTIO<br />

<strong>Research</strong>ers:<br />

• Pierre Van Damme<br />

Implementation <strong>of</strong> novel diagnostic technologies and classification <strong>of</strong> DNA variants <strong>of</strong> unknown pathogenicity in<br />

hereditary breast and/or ovariancancer<br />

K.U.Leuven<br />

Abstract: No English Abstract<br />

Organisations:<br />

• Department <strong>of</strong> Human Genetics<br />

<strong>Research</strong>ers:<br />

• Eric Legius<br />

• Gert Matthijs<br />

• Geneviève Michils<br />

Mappingsonderzoek architectuurcultuurbeleid<br />

University College Ghent<br />

Abstract: Abstract not yet available<br />

Organisations:<br />

• Faculty <strong>of</strong> Business Administration and Public Administration<br />

• Department <strong>of</strong> Administration and Management<br />

<strong>Research</strong>ers:<br />

• Ellen Wayenberg<br />

• Junior Burssens<br />

LRRK2 signalling pathways: validation and evaluation <strong>of</strong> the physiologicrelevance <strong>of</strong> candidate substrates for LRRK2<br />

protein<br />

K.U.Leuven<br />

Abstract: Parkinsons disease (PD) is the most common neurodegenerative movement disorder in the world. The clinical features <strong>of</strong> this disease are<br />

caused by an unexplained loss <strong>of</strong> the nigrostriatal dopaminergic neurons that are important for controlling voluntary movements. Recent studies<br />

have identified genes that are mutated in families with PD, and the functional characterization <strong>of</strong> these PD-related genes has provided important<br />

clues on the potential pathobiological mechanisms leading to disease. Mutationsin LRRK2 have been recognized as the major prevalent<br />

geneticcause <strong>of</strong> familial and sporadic PD. Not much is known about the function<strong>of</strong> this protein, but strong evidences point its kinase activity as<br />

crucial for LRRK2-driven toxicity. Therefore, the identification and characterization <strong>of</strong> the substrates <strong>of</strong> LRRK2 are needed, and the aim <strong>of</strong> this<br />

project is to tackle this issue by using relevant cell and animal models available, in combination with a set <strong>of</strong> optimized biochemical protocols. This<br />

wil<br />

Organisations:<br />

• Department <strong>of</strong> Human Genetics<br />

<strong>Research</strong>ers:

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