What Really Causes Alzheimer's Disease - Soil and Health Library

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14. Chiu, H.F.K., and Zhang, M. (2000). Dementia research in China. International Journal of Geriatric Psychiatry, 15, 947-953. 15. David, A.R. (1997). Disease in Egyptian mummies: The contribution of new technologies. Lancet, 349(9067), 1760-1763. 16. Ibid. 17. Doctor’s Guide. Global Edition (1995). Unlocking mummies’ secrets to study today’s diseases. http://www.pslgroup.com/dg/2c6ae.htm 18. Mummies: Unwrapped: The mysterious world of mummies. http:// www.tvdailyonline.com/tvguide/announce/coming10.htm 19. Gomez i Prat, J., and de Souza, S.M. (2003). Preehistoric tuberculosis in America: Adding comments to a literature review. Memorias do Instituto Oswaldo Cruz, 98 Suppl 1, 151-159. 20. Fusegawa, H., Wang, B.H., Sakurai, K., Nagasawa, K., Okauchi, M., and Nagakura, K. (2002). Outbreak of tuberculosis in a 2000-yearold Chinese population. Kansenshogaku Zasshi, 77(3), 146-149. 21. Reinhard, K.J., and Buikstra, J. (2003). Louse infection of the Chiribaya culture, Southern Peru: Variation in prevalence by age and sex. Memorias do Instituto Oswaldo Cruz, 98 Suppl 1, 173-179. 22. Cockburn, T.A., Cockburn, E., and Reyman, T.A. (eds.) (1998). Mummies, disease and ancient cultures. Cambridge: Cambridge University Press. 23. Filer, J. (1995). Disease. London: Egyptian Bookshelf, British Museum Press. 24. Foster, H.D. (2002). Why the preeminent risk factor in sporadic Alzheimer’s disease cannot be genetic. Medical Hypotheses, 59(1), 57-61. 25. Office of Technology Assessment, op. cit. 26. Schoenberg, B.S. (1981). Methodologic approaches to the epidemiologic study of dementia. In J.A. Mortimer and L.M. Schuman (eds.), The epidemiology of dementia: Monographs in epidemiology and biostatistics (pp. 117-131). New York: Oxford University Press. 27. Fotuhi, M. (2003). The memory cure. New York: McGraw-Hill. 28. Ineichen, B. (1998). The geography of dementia: An approach through epidemiology. Health & Place, 4(4), 383-394. 29. Arizona Department of Health Services. Public Health Services. Mortality from Alzheimer’s disease Among Arizona Residents, 1990-2000. http://www.hs.state.az.us/plan/report/mfad/mfad00/figures00. htm 23
30. Missouri Department of Health Center for Health Information Management & Epidemiology (1998). Missouri Monthly Vital Statistics, Provisional Statistics, 32(8). Jefferson City: Missouri. http://www. health.state.mo.us/MonthlyVitalStatistics/oct98vol32no8.pdf 31. Arizona Department of Health Services, op. cit. 32. Hagnell, O., Lanke, J., Rorsman, B., and Ojesjo, L. (1981). Does the incidence of age psychoses increase? Neuropsychobiology, 7, 201-211. 33. Ibid. 34. Centers for Disease Control. Mortality from Alzheimer’s disease - United States, 1979-1987 (1991). Journal of the American Medical Association, 265(3), 313-317. 35. Centers for Disease Control and Prevention (2001). Mortality declines for several leading causes of death in 1999. Media Relations Press Release (404). http://www.organicconsumers.org/madcow/ mortality62601.cfm 36. National Center for Health Statistics (2003). HHS News. HHS study finds life expectancy in the US rose to 77.1 years in 2001. http:// www.cdc.gov/nchs/releases/03news/lifeex.htm 37. Centers for Disease Control (1991), op. cit. 38. Jorm, A.F., Henderson, A.S., and Jacomb, P.A. (1989). Regional differences in mortality from dementia in Australian analysis of death certificate data. Acta Psychiatrica Scandinavica, 79, 179-185. 39. Neuman, S.C., and Bland, R.C. (1987). Canadian trends in mortality from mental disorders, 1965-1983. Acta Psychiatrica Scandinavica, 76, 1-7. 40. Martyn, C.N., and Pippard, E.C. (1988). Usefulness of mortality data in determining the geography and time trends in dementia. Journal of Epidemiology and Community Health, 42, 134-137. 41. Flaten, T.P. (1989). Mortality from dementia in Norway, 1969-1983. Journal of Epidemiology and Community Health, 43, 285-289. 42. Shenk, op. cit. 43. Fotuhi, op. cit. 24
Page 2 and 3: What Really Causes Alzheimer’s Di
Page 4 and 5: Dedicated to Grey Friar’s Bobby i
Page 6 and 7: ACKNOWLEDGEMENTS The unusual cover
Page 8 and 9: WHAT REALLY CAUSES ALZHEIMER’S DI
Page 10 and 11: mutations involved in promoting the
Page 12 and 13: the amount of sediment in the water
Page 14 and 15: poor source of minerals, such as ca
Page 16 and 17: 17. Flaten, T.P. (1990). Geographic
Page 18 and 19: TABLE OF CONTENTS 1 Who Was I? ....
Page 20 and 21: WHO WAS I? 1 LEAR: FOOL: Does any h
Page 22 and 23: population. 8-9 David Shenk, 10 in
Page 24 and 25: saw this as a death sentence. “Te
Page 26 and 27: patients deteriorate. That is, the
Page 28 and 29: REFERENCES 1. Henderson, A.S. (1986
Page 30 and 31: 35. Garruto, R.M., Swyt, C., Yanagi
Page 32 and 33: IN THE BEGINNING: A HISTORY 2 I am
Page 34 and 35: In 1901 there was certainly nothing
Page 36 and 37: falciparum, which causes malaria, a
Page 38 and 39: techniques, medical researchers mus
Page 40 and 41: USA age-adjusted mortality rates fo
Page 44 and 45: THE BRAIN DRAIN: PATHOLOGY AND GENE
Page 46 and 47: with other threads of tau and becom
Page 48 and 49: The most extreme of the geneticists
Page 50 and 51: SUMMARY Major pathologic changes oc
Page 52 and 53: PUTTING THE DISEASE IN ITS PLACE 4
Page 54 and 55: smoking, over-consumption of alcoho
Page 56 and 57: eported more commonly from Russia,
Page 58 and 59: These differences in the spatial di
Page 60 and 61: years and older, the dementia preva
Page 62 and 63: REFERENCES 1. Hippocrates On Airs,
Page 64 and 65: Alzheimer’s disease in two commun
Page 66 and 67: BIOCHEMICAL ABNORMALITIES 5 What we
Page 68 and 69: aspartate, normally exist in low co
Page 70 and 71: types of glutamate receptors in the
Page 72 and 73: There is also growing evidence that
Page 74 and 75: of Alzheimer’s disease and so res
Page 76 and 77: SUMMARY It is apparent from this sh
Page 78 and 79: 14. Kirschmann, J.D. with Dunn, L.J
Page 80 and 81: 39. Symons, M.C.R., and Gutteridge,
Page 82 and 83: OTHER RISKS, FURTHER HAZARDS 6 I ca
Page 84 and 85: Cholesterol Whether or not high cho
Page 86 and 87: Homocysteine The essential amino ac
Page 88 and 89: placebos. As has been described ear
Page 90 and 91: may be stronger in women. Gustafson
Page 92 and 93:
disease. The first of these followe
Page 94 and 95:
Head Trauma Controversy continues o
Page 96 and 97:
Osteoporosis A recent longitudinal
Page 98 and 99:
Aging If, as has been previously ar
Page 100 and 101:
REFERENCES 1. Fotuhi, M. (2003). Th
Page 102 and 103:
25. Lim, G.P., Chu, T., Yang, F., B
Page 104 and 105:
54. Ibid. 55. Wilson, R.S., Barnes,
Page 106 and 107:
78. D’Alessandro, R., Gallassi, R
Page 108 and 109:
DOWN TO EARTH: LOCATION, LOCATION,
Page 110 and 111:
oad salt was used extensively and s
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of aluminum and potassium) to staun
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that, within psychiatric nursing ho
Page 116 and 117:
The importance of monomeric (single
Page 118 and 119:
would also promote this form of dem
Page 120 and 121:
correctly able to identify whether
Page 122 and 123:
and teeth. As a consequence, under
Page 124 and 125:
and coworkers 82 also established t
Page 126 and 127:
17. Crapper, D.R., Krishnan, S.S.,
Page 128 and 129:
45. Perl, D.P., and Good, P.F. (198
Page 130 and 131:
78. Jacqmin, H., Commenges, D., Let
Page 132 and 133:
CHECKING OUT ALTERNATIVES 8 Knowing
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in Japan, China, and Korea. Some of
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All appear favourable and Galantami
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changes. These observations may exp
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and behaviour. The reason for this
Page 142 and 143:
15. Thal, L.J., Salmon, D.P., Laske
Page 144 and 145:
43. Pettegrew, J.W., Klunk, W.E., P
Page 146 and 147:
CONVENTIONAL MEDICAL WISDOM 9 We’
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expected, a deficiency of this neur
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There may be a case for the use of
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of Alzheimer’s patients. 28 Birks
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M., Thomas, R., and Thal, L.J. (200
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BACK FROM THE ABYSS 10 Abide with m
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Louis Blank could no longer recogni
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UNDERSTANDING THE SYSTEM: DEMENTIA
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gene that is responsible. 3 There i
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misfolded proteins that have the un
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those who do not carry the APO E4 a
Page 168 and 169:
REFERENCES 1. Hoffman, R. (2003). W
Page 170 and 171:
IT TAKES TWO (OR MORE) TO TANGO 12
Page 172 and 173:
and increased risk for cardiovascul
Page 174 and 175:
of drinking water and this form of
Page 176 and 177:
These high levels of aluminum were
Page 178 and 179:
acid and elevated aluminum. 40 Unde
Page 180 and 181:
accompanied by a build-up of both b
Page 182 and 183:
11. Bishop and Waldholz, op. cit. 1
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changes in calmodulin-implications
Page 186 and 187:
A BARRIER TO ANTAGONISM 13 It all l
Page 188 and 189:
aluminum enters neurons and alone i
Page 190 and 191:
It is at this level that the geneti
Page 192 and 193:
REFERENCES 1. Koestler, A. (1959).
Page 194 and 195:
OTHER ENZYMES - FURTHER PROBLEMS 14
Page 196 and 197:
mechanisms for triggering and direc
Page 198 and 199:
of the phospholipids that concentra
Page 200 and 201:
neurofibrillary tangles of Alzheime
Page 202 and 203:
REFERENCES 1. Heinlein, R. cited by
Page 204 and 205:
lets of patients with Alzheimer’s
Page 206 and 207:
THE ACID TEST: DO THE PIECES FIT? 1
Page 208 and 209:
Table 1: The Pieces of the Jigsaw P
Page 210 and 211:
Chapter 8. Checking Out Alternative
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Alzheimer’s patients are very glu
Page 214 and 215:
There is no doubt that, globally, s
Page 216 and 217:
genetic aberration can remove beta-
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also that diabetics would benefit f
Page 220 and 221:
We do not need a giant, time wastin
Page 222 and 223:
Table 2: Alzheimer’s Disease and
Page 224 and 225:
this process occurs more slowly if
Page 226 and 227:
patients. 83 It has been establishe
Page 228 and 229:
sampled materials established that
Page 230 and 231:
damage before Alzheimer’s symptom
Page 232 and 233:
Not only is the aluminum burden of
Page 234 and 235:
phosphofructokinase. All the necess
Page 236 and 237:
REFERENCES 1. Brown, S.R. (2003). S
Page 238 and 239:
27. Kawahara, M., Kato, M., and Kur
Page 240 and 241:
adenylate cyclase, guanylate cyclas
Page 242 and 243:
adrenergic hypersensitivity and emo
Page 244 and 245:
115. Marlowe, M., and Bliss, L.B. (
Page 246 and 247:
GOOD-BYE TO THE LONG GOOD-BYE 16
Page 248 and 249:
It would seem to be in the best int
Page 250 and 251:
cancer, and diabetes mellitus. 16 S
Page 252 and 253:
in the Developed World, by governme
Page 254 and 255:
poor source of minerals, including
Page 256 and 257:
SUMMARY There is currently a global
Page 258 and 259:
25. Peak Performance, op.cit. 26. L
Page 260 and 261:
A Index A1-calmodulin complex 159 a
Page 262 and 263:
lood pressure 63-66, 80-81, 208 blo
Page 264 and 265:
E Early Onset Alzheimer’s disease
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hardening of the arteries 7 harmful
Page 268 and 269:
miners 97, 108 MIRAGE 74, 85, 223 M
Page 270 and 271:
etired 1 retrogenesis vii-viii, 2-3
Page 272 and 273:
vitamin B 12 67, 70, 147, 203 vitam
Page 274 and 275:
of the boards of the Journal of Ort
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