Equine ERU - University of Florida

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Uveitis starts as blood-ocularbarrier compromise!• Blood vessels of iris,CB and choroidbecome thickened,congested and “leaky”• Cells and mediatorsenter the eye– PMNs then LCs– Inflammatorycytokines
Lymphocyte infiltration• Heavy influx of LCinto uveal and othereye tissues• Clusters of LCresembling follicles inciliary body• T-LCs predominate,MHC Class II reactive
Page 1: Equine “Recurrent” Uveitis is a
Page 10 and 11: Testing for Leptospirosis• Most s
Page 12 and 13: Breed and UveitisColor pattern more
Page 14 and 15: The more pigment, the less ERU, and
Page 16 and 17: 150952
Page 18 and 19: PMNs give a green appearance in ERU
Page 20 and 21: ERU and fibrin (Cookie)
Page 22 and 23: Fibrin is removed by TPA
Page 24 and 25: Tissue Plasminogen Activator• Cat
Page 26 and 27: Endotheliitis: precursor of glaucom
Page 28 and 29: Tear film is unstable with edema
Page 31 and 32: Fibrin plaque in pupil.ERU causes c
Page 33 and 34: Moon
Page 35 and 36: Detachment of Retina
Page 37 and 38: Band Keratopathy: chronic ERU- 6% o
Page 39 and 40: Differentials for ERUERU resembles
Page 41 and 42: KEY POINTS: Treatment• But eventu
Page 43 and 44: KEY POINTS: Outcome• Tell owners
Page 45 and 46: • ERU Medical Therapy- Systemic N
Page 47 and 48: Intracameral Medications for ERU•
Page 49 and 50: Intracameral Administration• Trem
Page 51 and 52: 48 hrs post Rx
Page 53 and 54:
• Vitrectomy- Europe: 98% have le
Page 55 and 56:
Cyclosporine A implants• (Slow re
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Equine ERU - University of Florida

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