AGPI - Lipides et signalisation cellulaire

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DHA Accumulation in the Brain is Early and Rapid
Étude épidémiologique, effectuée à Hyderabad, Inde Diabète de type II
Page 1 and 2: 1. Acides gras polyinsaturés (AGPI
Page 4 and 5: % of total fatty acids FAT SOURCE n
Page 6 and 7: PUFA : “Pharmaconutrients”
Page 8 and 9: - AGPI n-3 dans le lait maternel
Page 12 and 13: PUFA/EPAX dietary treatment diminis
Page 14 and 15: PUFA, n-6 : “Immunostimulators”
Page 16 and 17: Effects of n-3 rich diet macrophage
Page 18 and 19: Psoriasis Dematose, érythémato-sq
Page 20 and 21: - Lupus - Polyarthrite rhumatoïde
Page 22 and 23: Sclérose en plaque
Page 24 and 25: Huile de poisson… un bon ttt ? 16
Page 26 and 27: Les bienfaits de l’huile de poiss
Page 28 and 29: Th1 phenotype is involved in type 1
Page 30 and 31: Système immunitaire & Hypertension
Page 33 and 34: Cyclooxygénase Lipoxygénase méca
Page 35 and 36: Phosphatidylcholine (PC) est la cib
Page 37 and 38: - Sécrétées - Cytosoliques Phosp
Page 39 and 40: AGPI, n-3 PL DAG - DHA DAG - EPA DA
Page 41 and 42: CalB domain ATP PKC kinase Thr-497
Page 43 and 44: Différentes isoformes de PKC GROUP
Page 45 and 46: Activation des différentes isoform
Page 47 and 48: PIP 2 PLC IP3 “CROSS - TALK” -
Page 49 and 50: L’acide arachidonique induit la p
Page 51 and 52: EPA and DHA diminish PMA-induced ER
Page 53 and 54: β-actin, mRNA IL-2, mRNA PMA + ion

AGPI - Lipides et signalisation cellulaire

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