Der NO/ONOO Der NO/ONOO- Zyklus : Grundlagen des ...

Reported apparent genetic susceptibility in fibromyalgia
Lowered
Consistent with action of glucocorticoids including
glucocorticoid cortisol in repressing iNOS induction; consistent with
receptor activity
role of nitric oxide in fibromyalgia
MEFV gene effects
OLETF rat as a
fibromyalgia model
COMT metabolism
of catecholamines
Adrenergic
receptor gene
Interpreted in terms of inflammatory cytokine,
including IL-1 role in initiating cases of fibromyalgia.
Phenotype is produced by increased angiotensin II
action, leading to increased superoxide; effect of
gene is blocked by angiotensin antagonists of
superoxide dismutase mimic
Suggests neurological role
Suggests neurological role
Dopamine receptor
Suggests neurological role
gene