Programme & Abstracts - Society for Endocrinology

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12:30 Lunch and poster viewing Reception Room 13:30 Debate: “The NHS can no longer afford to pay for endocrine ‘lifestyle’ conditions” Introduction: Dr Anna Crown (Brighton) For: Dr John Quin (Consultant Endocrinologist, Brighton) Against: Dr Tom Scanlon (Director of Public Health Brighton & Hove) Clinical cases II: Endocrine neoplasia & neuroendocrine tumours Chairs: Professor Colin Dayan (Cardiff) and Dr John Newell-Price (Sheffield) 14:15 A case of malignant bladder phaeochromocytoma Grant ,P. Carroll, P. Agustsson, T. St. Thomas’ Hospital, London 14:30 RET C620R, the janus mutation Roplekar, R. 1 , Carroll, PV. 2 1 King’s College London, Guy’s and St Thomas’ Hospital NHS Trust, London 2 Guy’s and St Thomas’ Hospital NHS Trust, London 14:45 Symptomatic hypoglycaemia due to high IGF-2 levels in a patient with adrenocortical carcinoma Doumouchtsis,K. Mishra,N. Schulte,K-M. Aylwin, SJB. King’s College Hospital, London 15:00 A clinically silent invasive adrenocortical tumour: charting steroid excretion before tumour removal, and after mitotane therapy and repeat surgery Taylor, N F. 1 , Ghataore, L. 1 , Schulte, K-M . 2 Aylwin, SJB. 3 King’s College Hospital, London 15:15 Pancreatic neuroendocrine tumor secreting glucagon and GLP-1 causing diabetes and subsequent hyperinsulinaemic hypoglycaemia Roberts, RE._ Zhao, M. 2 Whitelaw, BC. 3 Ramage, J. 4 Rela, M. 4 Diaz-Cano, SJ. 5 , Quaglia, A. 4 Huang, GC. 2 Aylwin, SJB. 3 1, 3-5 King’s College Hospital London School of Medicine, London 2 King's College London School of Medicine, London 15:30 Tea and poster viewing 16:00 Lecture 3: ‘Pseudo-endocrinology’: unorthodox endocrine remedies Dr John Miell (London) Introduction by: Dr Simon Alywin (London) 16:30 Lecture 4: Clinical vignettes from the endocrine cancer to MDMs Dr Pauline Kane (London) and Dr Simon Aylwin (London) Introduction by: Dr Anna Crown (Brighton) 17:00 Evening reception and awards ceremony Reception Room Two oral presentations will be awarded prizes: £250 for the first prize and £150 for the runner up prize. The two overall highest scoring posters will also receive a prize of £100 each.
Hypophysitis: The diagnostic challenges of an inflammatory pituitary mass Liu,Y. Boleat,E. Agustsson, T. Kariyawasam, D. Department of Endocrinology, Guy’s and St. Thomas Hospital NHS Foundation Trust London, Westminster Bridge Road, London SE1 7EH Case History: A 24 year old student from Pakistan presented to A&E with 6 days of severe headache and vomiting. He complained of chronic headaches and short-term memory loss 6 months prior to presentation. There was no visual disturbance or neck stiffness. He denied a history of tuberculosis (with a negative testing prior to entry to UK), and had no respiratory symptoms, fever or night-sweats. There was weight loss of approximately 17kg in the preceding months but no symptoms of polyuria or polydipsia and no nocturia. The past medical history consisted only of congenital nystagmus. Neurological examination was normal. CT brain raised the concern of a circle of Willis aneurysm; however a subsequent MRA revealed the abnormality as an enlarged pituitary with compression of the optic chiasm. On further questioning, he reported a six months history of nausea, vomiting and reduced energy with lowered libido but intact erectile function. Investigations and methods: Pituitary screen revealed a TSH of 0.50mIU/L, FT4 6.2pmol/L, FT3 2.5pmol/L, morning cortisol of 60nmol/l, ACTH 20ng/L, IGF-1 17.4nmol/L, GH 1.7ug/L, prolactin 223 mU/L, testosterone 2nmol/l, LH 2.8IU/L, FSH 5.4IU/L. Plasma osmolality was 286 mOsmol and sodium was 140 mmol/L. Pituitary MRI showed a bulky diffusely avidly enhancing pituitary with convex upper border and a thickened, enhancing stalk. A right inferotemporal defect was found on formal visual field testing. Chest X-ray and CT of chest, abdomen and pelvis were normal. He was considered to have partial hypopituitarism with TSH, gonadotrophin and possibly ACTH deficiency. Mantoux test was positive at 20mms. ACE levels were normal in serum. CRP was
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