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HBOT and <strong>Suit</strong> <strong>Therapy</strong><br />

improvements in both physical function and cognitive ability.<br />

So, significant brain recovery is still possible later in life. The<br />

reality that most of us have or will experience is that the<br />

physicians that treat those with cerebral palsy and other brain<br />

injuries have had no exposure to Hyperbaric Medicine during<br />

their careers, so they do not prescribe HBOT.<br />

How can HBOT improve the <strong>Cerebral</strong> Palsy condition<br />

and other brain conditions?<br />

When cells in the brain die, either from trauma or lack of<br />

oxygen, blood plasma leaks out into the surrounding brain<br />

tissue causing swelling and reducing blood flow. These<br />

otherwise normal cells go dormant because they can’t<br />

function without the appropriate amount of oxygen. HBOT<br />

dramatically increases the oxygen carried in the blood<br />

plasma and other bodily fluids (which is normally quite<br />

low) by up to 2,000% making oxygen available to heal<br />

damaged capillary walls, preventing plasma leakage and<br />

reducing swelling. As the swelling decreases, normal blood<br />

flow can be restored to the dormant tissue<br />

(neovascularization) and these cells then have the potential<br />

to go back “on-line” and function normally.<br />

It is at this point, you would expect to see improvement in<br />

the areas where there are deficits. Un<strong>for</strong>tunately, there<br />

are no factors (age, condition, length of time from injury,<br />

etc.) that absolutely predict or guarantee the degree of<br />

improvement. However, what we have experienced at our<br />

facility in treating neurological conditions (infants through<br />

seniors) with HBOT is that 67% received such significant<br />

benefit, that they chose to return <strong>for</strong> additional treatments…<br />

% of Patients who Returned to HHI <strong>for</strong> Additional HBOT<br />

<strong>Cerebral</strong> Palsy 74%<br />

Multiple Sclerosis 71%<br />

PDD / Autism 63%<br />

Stroke 58%<br />

Our findings are similar or greater than some of the more<br />

<strong>for</strong>mal studies done on HBOT and its effect on <strong>Cerebral</strong><br />

Palsy individuals. The following study was presented in<br />

November of 2000.<br />

The Cornell Study (condensed)<br />

Dr Maurine Packard Division of Child Development<br />

and Pediatric Neurology - Study Design<br />

This study was designed as a randomized, delayed entry<br />

trial of the effects of HBOT on children ages 1 to 5 years<br />

with moderate to severe CP. Enrollment criteria were 1)<br />

age between 1 and 5 years; 2) moderate to severe CP; 3)<br />

no evidence of brain mal<strong>for</strong>mation; 4) developmental<br />

delay of at least 33% in one area; 5) no active seizures<br />

<strong>for</strong> the previous 6 months. The protocol consisted of 40<br />

one-hour sessions of HBOT at 1.5 ATA. The sessions were<br />

scheduled twice a day, five days a week <strong>for</strong> four weeks.<br />

We did not design a double blind study, in which some<br />

children would receive placebo treatments, <strong>for</strong> several<br />

reasons. First, this was a pilot study to see if there was<br />

any evidence of benefit <strong>for</strong> these children. Second we<br />

purposefully enrolled children of various ages and<br />

disability levels to evaluate the efficacy of HBOT in a<br />

range of affected children. Third, as time in the chamber<br />

is very expensive, we wanted as much in<strong>for</strong>mation about<br />

treatment effects as possible. Finally, it seemed unethical<br />

to have parents devote so much time and energy to a<br />

potentially ineffective treatment.<br />

Demographics<br />

The study population included 26 children, ages 15<br />

months to 5 years, with cerebral palsy secondary to<br />

prenatal insults, premature birth, birth asphyxia, and<br />

post-natal hemorrhage. The subjects were enrolled at a<br />

rate of 4 per month and matched roughly to age and<br />

severity. The average age at enrollment was 30 months.<br />

The average motor age was 7.5 months; the average<br />

cognitive age and language age were both l2 months.<br />

Nine had cortical visual impairments.<br />

Assessment<br />

Intake assessments included a neuro-developmental<br />

assessment, Bayley II (cognitive assessment), Preschool<br />

Language Scale (language assessment), the Peabody<br />

Motor Scales (an assessment of gross and fine motor<br />

skills), Pediatric Evaluation of Disabilities Inventory<br />

(PEDI), a parental report of specific skill in mobility, selfcare,<br />

and social interactions. Assessments were<br />

conducted at four time points: T1 - at enrollment; T2 -<br />

<strong>Cerebral</strong> Palsy Magazine-December 2004 49

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