- Page 1: Weinstein Cardiovascular Developmen
- Page 4 and 5: Welcome to the 2012 Chicago Weinste
- Page 8 and 9: Wednesday, May 2 nd 12:00 pm Meetin
- Page 10 and 11: 4:25 pm S2.2 Epigenetic regulation
- Page 12 and 13: 10:00-10:30 am Coffee Break 10:30-1
- Page 14 and 15: 4:45 pm S5.4 A Tbx5-Scn5a Molecular
- Page 16 and 17: 1:00-3:30 pm Poster Session 3 2:15-
- Page 18 and 19: Keynote Presentation Clifford J. Ta
- Page 20 and 21: Annual Business Meeting Each Weinst
- Page 22 and 23: Sponsors The 2012 Weinstein Cardiov
- Page 24 and 25: S1.2 Complex trait analysis of vent
- Page 26 and 27: S1.4 The Genetic Basis of Isolated
- Page 28 and 29: S1.6 Identification of Novel Mutati
- Page 30 and 31: S2.2 Epigenetic regulation of cardi
- Page 32 and 33: S2.4 CIP, a novel cardiac nuclear p
- Page 34 and 35: S2.6 A Transitional Extracellular M
- Page 36 and 37: S3.2 TU-tagging in mice defines dyn
- Page 38 and 39: S3.4 The novel mechanism of histone
- Page 40 and 41: S3.6 Drosophila microRNA-1 Genetica
- Page 42 and 43: S4.2 The Role of Fibronectin - Inte
- Page 44 and 45: S4.4 The neural crest contributes t
- Page 46 and 47: S4.6 Nephronectin regulates axial v
- Page 48 and 49: S5.2 Wnt signaling in the developin
- Page 50 and 51: S5.4 A Tbx5-Scn5a Molecular Network
- Page 52 and 53: S5.6 Activation of voltage-dependen
- Page 54 and 55: S6.2 Clonally dominant cardiomyocyt
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S6.4 A Cdc42 associated genetic net
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S6.6 Distinct origin and commitment
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S7.2 Zebrafish second heart field d
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S7.4 Mutations in the NOTCH pathway
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S7.6 Retinol Dehydrogenase 10: Role
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S8.2 Tie1 is Required for Semilunar
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S8.4 Filamin-A Regulates Tissue Rem
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S8.6 Endothelial nitric oxide signa
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could provide the basis for a thera
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variants likely to be homozygous ba
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promote and repress BMP signaling a
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Calcific aortic valve disease (CAVD
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Supported in part by Primary Childr
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VACTERL, a constellation of malform
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future studies, we will test whethe
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found in 59 mutant lines. Double ou
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2 Children’s Hospital of Pittsbur
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cardiovascular phenotyping, and imp
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positional cloning, we identified a
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PIK3CB was validated to be a direct
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positive cardiomyocytes, epicardium
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affecting term viability of mutants
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observed in the hearts of these ani
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S3.4 The novel mechanism of histone
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to the FHF, and Tbx5 is required in
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genes. In addition to zebrafish, MM
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3.20 Cell surface chemoproteomics f
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3.25 Combinatorial Regulation of Ca
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ultimate steps of NCC differentiati
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S4.6 Nephronectin regulates axial v
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and yolk sac. The later group is de
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These NCC depletions were also obse
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elationship. This timeline will ben
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development, TnC is highly upregula
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was measured two weeks later. MCT g
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earlier steps in coronary vasculoge
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murine cardiomyocytes, demonstratin
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necessary for CPC AP generation are
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conduction genes and thereby defini
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The epicardium plays an important r
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hpf and 96 to 120 hpf) stopped the
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S6.2 Clonally dominant cardiomyocyt
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6.7 Dynamic Mesp1 regulation direct
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looping, and the splice-site MO phe
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increase of hoxb5b at least in part
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diffusion but can label hundreds of
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6.27 Vascular Endothelial And Endoc
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S7.2 Zebrafish second heart field d
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S7.6 Retinol Dehydrogenase 10: Role
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7.11 Mef2c Regulates Transcription
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Cyp26c1, are expressed in the anter
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vitro double transgenic mouse repor
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S8.2 Tie1 is Required for Semilunar
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demonstrate a novel molecular pathw
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angiogenic factors (VEGF) at the ju
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studied before and after (3-6 month
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8.21 4-D Shear Stress Maps of the D
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progression in developing valves an
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8.31 Twist1 Homodimer and Heterodim
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Burchf, John S8.5 Burkhard, Silja 5
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Hu, Jiangyong 3.18 Huang, Can 1.22
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Mohun, Timothy 1.9, 3.28 Molero, Ma
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Tao, Jiayi 5.23 Targoff, Kimara S7.
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Registrants Radwan Abu-Issa Departm
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Rachel Brunner Department of Biolog
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Steven Fisher Department of medicin
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Chuanchau Jou Department of Pediatr
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Ching-Ling (Ellen) Lien Department
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Mariya Mollova Department of Cardio
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Stacey Rentschler Department of Med
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Mardi Sutherland Department of Mole
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Hisato Yagi Department of Developme
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