Professor W. Daniel Stamer - University of Arizona

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Curriculum Vitae of W. Daniel Stamer Revised 2/4/2011 87. RD McCarty, RM Beverley, MC Giovingo, MJ Nolan, BYJT. Yue, WD Stamer, PA Knepper. Differential Pattern of Junctional Complex Lipid Raft Proteins in Human Conventional Outflow. Invest. Ophthalmol. Vis Sci, ARVO abstract #3204, 2010. 88. GM Sumida and WD Stamer. S1P1 receptor activation is insufficient to mimic S1P-mediated effects on aqueous humor. FASEB, abstract #1640, 2010. 89. WD Stamer, D Piwnica and DF Woodward. Bioactive lipids modulate contractility of ocular outflow cells. Keystone Symposia Conference. Bioactive lipids: Biochemistry and Disease. Kyoto, Japan, June 6-11. 90. EH Zhou, R Krishnan, WD Stamer, KM Perkumas, K Rajendran, JJ Fredberg and M Johnson. . Stiffness and Traction Forces of Schlemm's Canal Endothelial Cells in Vitro. Abstract# 817. XIX International Congress of Eye Research, Montreal, Canada, July, 2010. 91. LH Millard, K Watanabe, A Shimada, DF Woodward and WD Stamer. Prostaglandin/Prostamide F Synthase Expression in Human Eyes. Abstract #500. XIX International Congress of Eye Research, Montreal, Canada, July, 2010. Grants and Contracts: Federal Grants 2009-2014 Co-Principal Investigator, Bioengineering Research Program Grant (Administrative PI: Mark Johnson). National Eye Institute, R01 EY19696, “The Mechanical Basis of Primary Open-Angle Glaucoma”. 2009-2014 Co-Investigator, (Civan, PI), National Eye Institute, R01 EY013624, “Aqueous Humor Outflow Control and Intraocular Pressure”. 2006-2012 Principal Investigator, National Eye Institute, R01 EY17007, “Cadherin Dynamics and Glaucoma”. 2001-2011 Principal Investigator, National Eye Institute, R01 EY12797, “Molecular Mechanisms of Myocilin Function in the Human Eye”. 2003-2006 Principal Investigator, National Eye Institute, R03 EY15115 “Seleniuminduced Glaucoma”. 2008 Principal Investigator, National Institute for General Medical Sciences, R01 GM59986, “Regulation of Ion Channel Function of Aquaporin-1”. 2003-2007 Co-Principal Investigator, (AJ Yool, Co-P.I.) National Institute for General Medical Sciences, R01 GM59986, “Regulation of Ion Channel Function of Aquaporin-1”. 2002-2005 Principal Investigator, National Eye Institute, R03 EY13861, “Cadherin Regulation of Aqueous Humor Outflow”. 26
Curriculum Vitae of W. Daniel Stamer Revised 2/4/2011 State Grants 2000-2001 25% Effort, Principal Investigator, Dean’s Research Council (DRCG), “Fluid Flow Routes across Schlemm’s Canal”; Received Dean’s commendation for outstanding progress 2/20/02. Private Industry Grants/Contracts 2000 Unrestricted Research Grant, Allergan, Inc. 2003 Unrestricted Research Grant, Allergan, Inc. 2004 Unrestricted Research Grant, Allergan, Inc. 2005 Unrestricted Research Grant, Allergan, Inc. 2006 Unrestricted Research Grant, Allergan, Inc. 2006 Research Contract, Pfizer Pharmaceuticals 2007 Unrestricted Research Grant, Allergan, Inc. 2007 Research Contract, Pfizer Pharmaceuticals 2007 Research Contract, Allergan, Inc. 2008 Unrestricted Research Grant, Allergan, Inc. 2008 Research Contract, Allergan, Inc. 2009 Unrestricted Research Grant, Allergan, Inc. 2010 Unrestricted Research Grant, Allergan, Inc. 2010 Research Contract, GlaxoSmithKline Private Foundation Grants 2010 Principal Investigator, Research to Prevent Blindness Foundation, Research Sabbatical Award. 2001-2003 25% Effort, Principal Investigator, American Health Assistance Foundation (AHAF), G2001-026, National Glaucoma Research Award, “Role of Aquaporin-1 in the Permeability of Schlemm’s Canal”. 1999-2000 25% Effort, Principal Investigator, Glaucoma Research Foundation (GRF), “Effect of mutations in myocilin on cellular secretion”. 1998-1999 25% Effort, Co-Principal Investigator, American Health Assistance Foundation (AHAF), National Glaucoma Research Award, “Role of TIGR/MYOC Protein in Human TM Cells”. Training Grants 2006-2008 Thesis mentor for Renata Ramos, National Eye Institute, F31-EY16887, “Biomechanical factors that regulate outflow facility”. 27
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Professor W. Daniel Stamer - University of Arizona

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