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PHASE II: INFLAMMATION • Inflammatory cells infiltrate the myocardium • Cytokine release • (Ex. interferons, interleukins, tumor necrosis factor) • Macrophages, lymphocytes, and endothelial cell activation • Inflammatory mediators = myocarditis • Vascular endothelium disrupted: • Endothelial dysfunction • Vasoconstriction • Necrosis of myocardial tissues
PHASE III: CELL-MEDIATED IMMUNITY • Recruitment of immune cells to infected myocardium • T helper cells + cytotoxic T clear the virally infected cells • Activated T lymphocytes lysis virus-infected cardiomyocytes • Viral clearing • Detrimental effects: • Myocyte signal transduction • Calcium homeostasis • Cardiac bioenergetics
Page 1 and 2: A CASE REPORT: ECMO SUPPORT FOR FUL
Page 3 and 4: MYOCARDITIS: DEFINITION • Myocard
Page 5 and 6: CLASSIFICATION • Etiology • Ex:
Page 7: Phase I: Viral Infection • Cardio
Page 11 and 12: FIGURE 1 PATHOGENESIS OF VIRAL MYOC
Page 13 and 14: HISTORY & PHYSICAL • 24 year old
Page 15: ICU AND RIGHT HEART CATH • VTach
Page 19 and 20: MUSC-ECMO •Equipment: •Stockert
Page 21: • Cardiac Stun: ECHO: DAY 1 OF EC
Page 26 and 27: ECHO: DAY 6 OF ECMO • Trial Off-
Page 28 and 29: 6 14 20 34 38 46 54 62 70 82 90 102
Page 30 and 31: fib (mg/dL) 215 191 208 229 233 306
Page 35 and 36: EF (%) 80 Patient EF Through Hospit
Page 37 and 38: CLINICAL PRESENTATION OF MYOCARDITI
Page 39 and 40: METHODS OF FM DIAGNOSIS • Echocar
Page 41 and 42: LABORATORY EVIDENCE FOR FM VS. ACUT
Page 43 and 44: Fulminant Chronic HISTOLOGICAL CLAS
Page 46: LONG-TERM OUTCOME OF FULMINANT MYOC
Page 49 and 50: EXPERIENCE AND RESULT OF ECMO IN TR
Page 51: CONCLUSION Teamwork and aggressive
Page 54 and 55: Initial Tn levels were elevated (21
Page 56 and 57: #3 JOURNAL OF THE AMERICAN COLLEGE
Page 58 and 59:
FAVOURABLE CLINICAL OUTCOME IN PATI
Page 60 and 61:
SPINALE IDEAS • Myocardial stanni
Page 62 and 63:
FIGURE 2. A CASE OF FULMINANT MYOCA
Page 64 and 65:
CALL FOR ABSTRACTS ABSTRACTS FOR CA
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