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<strong>Medical</strong> <strong>Emergencies</strong> <strong>in</strong> <strong>the</strong><br />

<strong>Dental</strong> <strong>Office</strong><br />

<strong>Tomas</strong> J. Barrios DDS<br />

Associate Cl<strong>in</strong>ical Professor of<br />

Oral & Maxillofacial Surgery,<br />

St. Josephs Regional <strong>Medical</strong> Center,<br />

Jersey City <strong>Medical</strong> Center


• Prevention & Preparation<br />

• Syncope<br />

• Hypoglycemia<br />

• Epileptic episode<br />

• Ang<strong>in</strong>a<br />

• Myocardial <strong>in</strong>farct<br />

• Anes<strong>the</strong>tic overdose<br />

• Drug allergy anaphylaxis<br />

• Asthma


Prevention & Preparation<br />

• BLS Certification doctors and staff<br />

• ACLS certification : any type of sedation<br />

techniques<br />

• Emergency Kits<br />

• Staff drills


Emergency Kits<br />

• Contents Vary<br />

• Purchase from any<br />

manufacturer<br />

• Refill


Components of emergency kits


Emergency drills<br />

• Most important : Keep it<br />

simple !<br />

• Designate a specific task to<br />

each staff member<br />

1. Assistant : gets kit and assist<br />

Dr.<br />

2. Receptionist : calls 911 and<br />

make sure EMS arrives ,<br />

clears area for patients<br />

3. Additional staff : go between,<br />

records vitals,<br />

event tim<strong>in</strong>g


ABC of any emergency<br />

• Sup<strong>in</strong>e position<br />

• 100 % Oxygen<br />

• Evaluate Airway, breath<strong>in</strong>g , circulation<br />

• Vitals


Emergency management &<br />

resuscitation plan<br />

• Primary survey<br />

1. ABCDE<br />

2. Purpose : Identify &<br />

treat life threaten<strong>in</strong>g<br />

problems<br />

3. History<br />

4. Resuscitation measures<br />

are <strong>in</strong>stituted<br />

• Secondary survey<br />

1. Head to toe by region<br />

exam<br />

2. Purpose : Identify &<br />

treat life threaten<strong>in</strong>g<br />

problems<br />

3. History : data ga<strong>the</strong>r<strong>in</strong>g<br />

4. System specific test<br />

5. Re evaluation: repeat<br />

surveys untill cause is<br />

identified<br />

6. Def<strong>in</strong>itive care<br />

6.


Vitals, Cl<strong>in</strong>ical signs and<br />

symptoms of potential illness


Tenets of primary survey<br />

• Proceed rapidly<br />

• Err on <strong>the</strong> side of aggressiveness<br />

• When <strong>in</strong> doubt “do”<br />

• Stay <strong>in</strong> sequence<br />

• Know what to look for , recognize and treat<br />

• Look for likely, treatable problems<br />

• Make decisions based on direct exam<strong>in</strong>ation<br />

• Initiate only simple test and procedures


Generalized treatment protocol


Documentation<br />

• Brief history of <strong>the</strong> event<br />

• Positive f<strong>in</strong>d<strong>in</strong>gs of primary & secondary<br />

survey<br />

• Treatment provided<br />

• Time of important events<br />

• Disposition


<strong>Medical</strong> legal issues<br />

• What if its not a patient <strong>in</strong> <strong>the</strong> office <br />

• Death or Hospital transfer by EMS , is a<br />

reportable event to <strong>the</strong> State Board


Syncope


Types of syncope


Vasodepressor Syncope<br />

• Most common medical emergency <strong>in</strong><br />

dentistry<br />

• 30 % of adult population<br />

• Accounts for 3% of ER visits


Etiology<br />

• Decreased cerebral blood flow (CBF)


Differential diagnosis<br />

Anxiety attacks<br />

hyperventilation syndrome<br />

MI<br />

Hypoglycemia<br />

Epilepsy<br />

Hypotension


Cl<strong>in</strong>ical manifestation presyncopal<br />

• Early<br />

Nausea<br />

Warmth<br />

Perspiration<br />

loss of color<br />

Basel<strong>in</strong>e Blood press<br />

Tachycardia<br />

• Late<br />

Hypotension<br />

Bradycardia<br />

Hyperpnea<br />

Pupillary dilation<br />

Peripheral coldness<br />

Visual disturbance<br />

Loss of consciousness


Syncopal phase<br />

• All secondary to decreased CBF<br />

• Loss of consciousness<br />

• Loss of postural tone<br />

• Any syncope last<strong>in</strong>g > a few m<strong>in</strong>utes can<br />

<strong>in</strong>duce seizures and cerebral ischemia


Treatment<br />

• Trendelenburg position<br />

• Pregnant patient lateral decubitus<br />

• Asses consciousness<br />

• ABC<br />

• 100 % oxygen<br />

• Spirits of ammonia<br />

• Vitals ( Bradycardia < 60 adm<strong>in</strong>ister Atrop<strong>in</strong>e .5mg IV<br />

1mg IM every 5 m<strong>in</strong>utes until max dose of 3 mg<br />

• EMS if loss of consciousness is > 5 m<strong>in</strong> or if recovery is<br />

> 20 m<strong>in</strong>


Postsyncope<br />

• Evaluate discharge home with escort or<br />

EMS<br />

Dependent on recovery and Vitals<br />

Recovery > 20 m<strong>in</strong><br />

Underly<strong>in</strong>g medical conditions


Hypotension


Hypotension<br />

• Follow<strong>in</strong>g syncope it is <strong>the</strong> most common<br />

cause of loss of consciousness <strong>in</strong> <strong>the</strong><br />

dental office


What affects perfusion


Causes of hypotension


Orthostatic Hypotension<br />

• Most common cause of hypotension <strong>in</strong> <strong>the</strong><br />

dental office<br />

• It is Syncope when <strong>the</strong> patient is placed<br />

quickly from a sup<strong>in</strong>e to upright position<br />

( < CBF )


Why Most likely <strong>in</strong> elderly <br />

• Ag<strong>in</strong>g decreases baroreflex mechanism<br />

which impairs cardioacceleratory response<br />

to preload reduction dur<strong>in</strong>g upright<br />

posture<br />

• May be on medications<br />

• Most susceptible


Vasovagal Hypotension (syncope)<br />

• Initiated by stressful physical ,<br />

psychological or surgical stimuli ( cough<strong>in</strong>g<br />

pa<strong>in</strong>, gagg<strong>in</strong>g )<br />

• The impulses are transmitted directly to<br />

<strong>the</strong> medulla <strong>in</strong> area closely related to <strong>the</strong><br />

nuclei of <strong>the</strong> vagus nerve


Cl<strong>in</strong>ical sign and symptoms<br />

• Bradycardia results from Vagal stimulation<br />

and parasympa<strong>the</strong>tic tone<br />

• Vasodilation results from dim<strong>in</strong>ished<br />

sympa<strong>the</strong>tic tone


Treatment<br />

• Remov<strong>in</strong>g <strong>the</strong> <strong>in</strong>itiat<strong>in</strong>g stimuli<br />

• Trendelenburg position<br />

• Oxygen<br />

• Vitals


Rout<strong>in</strong>e treatment for a patient<br />

with hypotension and <strong>in</strong>adequate<br />

perfusion


Treatment<br />

• Place <strong>in</strong> Trendelenburg position<br />

• Oxygen<br />

• Vitals<br />

• ABC<br />

• Evaluate BP<br />

( if no BP monitor present , remember palpate<br />

pulse, correlated to a systolic of: Radial 80 mm<br />

Hg , Brachial 70 mm Hg , Carotid 60 m Hg )<br />

• Adm<strong>in</strong>ister: Phenylephr<strong>in</strong>e spray 0.25-0.5 0.5 mg IV<br />

2-3mg IM , Ephedr<strong>in</strong>e 10-25 mg IV


• What if patients are receiv<strong>in</strong>g B-Blockers B Blockers <br />

Isoproterenol 0.2mg IV slowly at 1 m<strong>in</strong><br />

<strong>in</strong>terval and monitor patients response


Hypoglycemia


Diabetes


Epidemiology<br />

• Incidence 15.7 million or 5.9% of<br />

population of U.S<br />

• Incidence of undiagnosed 5.4 million or<br />

34% of diabetic population


Why is glucose important <br />

• Primary energy<br />

substrate for all<br />

functions


Pathophysiology<br />

• Type 1 IDDM : little or no <strong>in</strong>sul<strong>in</strong> is secreted<br />

uptake of glucose or conversion <strong>in</strong>to glycogen <strong>in</strong><br />

<strong>the</strong> liver does not occur, <strong>the</strong>refore liver glucose<br />

production is elevated. Gluconeogenesis<br />

accelerates<br />

• Type 2 NIIDM : Insul<strong>in</strong> resistance causes <strong>the</strong><br />

liver to cont<strong>in</strong>ue glucose production and<br />

prohibits glucose uptake by muscles


Cl<strong>in</strong>ical factors of diabetes


Type II NIDDM Meds


Type I IDDM Meds<br />

• Need to know or quick reference


Monitor<strong>in</strong>g<br />

• Hemoglob<strong>in</strong> A1C<br />

• Fructosam<strong>in</strong>e<br />

• Home monitor<strong>in</strong>g


How are patients go<strong>in</strong>g to become<br />

hypoglycemic <br />

• Too much <strong>in</strong>sul<strong>in</strong><br />

• Alcohol consumption<br />

• Excessive exercise<br />

• Missed delayed meals<br />

• Reduced meals<br />

• Medication error<br />

• O<strong>the</strong>r illness


Symptoms<br />

• Autonomic<br />

1. Sweat<strong>in</strong>g<br />

2. Trembl<strong>in</strong>g<br />

3. Palpitations<br />

4. Anxiety<br />

5. Nausea<br />

5.<br />

• Neuroglycopenic<br />

1. Dizz<strong>in</strong>ess<br />

2. Confusion<br />

3. Difficulty speak<strong>in</strong>g<br />

4. Headache<br />

5. Inability to concentrate<br />

6. Weakness<br />

7. Blurred vision<br />

7.


Treatment


Alternative TX<br />

• Glucose tablets<br />

• 4 teaspoons of sugar <strong>in</strong> water<br />

• 5 oz of regular soft dr<strong>in</strong>k<br />

• Orange juice<br />

• Glucagon dosage : 0.5-1mg IM or IV


Seizures


Seizures<br />

• Manifestation of bra<strong>in</strong><br />

dysfunction<br />

• Excessive neuronal<br />

cortical discharge<br />

• Secondary to tox<strong>in</strong>s,<br />

drugs, cerebral<br />

hypoxia, or metabolic<br />

disturbances


Prevention & preparation<br />

• History<br />

1. What type of seizure disorder do you have <br />

2. Are you on any medications for <strong>the</strong> disorder <br />

3. Are you tak<strong>in</strong>g <strong>the</strong> medications as prescribed <br />

4. Have you had serum level of <strong>the</strong> medication done If<br />

so when <br />

5. When was your last seizure <br />

6. What provokes it <br />

7. Do you have an Aura <br />

8. Where you hospitalized <br />

9. How long was your seizure <br />

9.


Treatment protocol<br />

• Most seizures last < 2 m<strong>in</strong><br />

• EMS activated<br />

• Assure patient & staff safety<br />

• Adm<strong>in</strong>ister oxygen<br />

• Manage airway<br />

• Monitor vitals , pulse oxymetry<br />

• Suction available<br />

• If seizure is last<strong>in</strong>g > 2 m<strong>in</strong>utes , establish IV,<br />

adm<strong>in</strong>ister Meds


Benzodiazep<strong>in</strong>e<br />

• Diazepam<br />

Adult : 5 to 10 mg<br />

IV/IM<br />

Pediatric : 0.2 to 0.5<br />

mg/kg IV/IM<br />

• Midazolam<br />

0.05 to 0.1 mg/kg IV<br />

0.2 mg/kg IM ( Max<br />

10 mg)


Pharmacologic management<br />

• EMS not arrived > 5 m<strong>in</strong><br />

Adult : Dextrose 50 ml bolus of 50%<br />

glucose<br />

Pediatric : 2ml/kg 25% dextrose solution<br />

• Evaluate airway ma<strong>in</strong>tenance<br />

• Evaluate cardiac rhythm


Postictal<br />

• Lethargy<br />

• Disorientation<br />

• Apnea, obstructed airway<br />

• Cardiac arrhythmias<br />

• Evaluate patient <strong>in</strong>jury


Chest pa<strong>in</strong>


• Ang<strong>in</strong>a : Lat<strong>in</strong> for spasmodic , chok<strong>in</strong>g or<br />

suffocat<strong>in</strong>g pa<strong>in</strong><br />

• Pectoris :Lat<strong>in</strong> for chest


Differential Diagnosis<br />

• Ang<strong>in</strong>a<br />

• Myocardial <strong>in</strong>farction<br />

• Dyspepsia, GERD<br />

• Musculoskeletal<br />

• Pulmonary embolus<br />

• Spontaneous pneumothorax<br />

• Aortic dissection<br />

• Esophageal rupture<br />

• Panic disorder


Relevant factors<br />

• Onset : time, associated<br />

event<br />

• Location<br />

• Radiation absence or site<br />

• Type of pa<strong>in</strong>: deep<br />

visceral, superficial,<br />

pleuritic<br />

• Exacerbat<strong>in</strong>g or<br />

alleviat<strong>in</strong>g factors


What occurs <br />

• Increased Myocardial Demand<br />

1. Elevated heart rate<br />

2. Elevated BP<br />

3. Elevated endogenous catecholam<strong>in</strong>es<br />

3.<br />

• Decreased Myocardial Oxygen delivery<br />

1. Decreased diastolic fill<strong>in</strong>g<br />

2. Myocardial vessel occlusion<br />

3. Hypoxia<br />

4. anemia<br />

4.


Treatment<br />

• ABC<br />

• oxygen<br />

• Position patient comfort<br />

• Vitals<br />

• EMS<br />

• Nitroglycer<strong>in</strong> : spray or tab .4mg repeat three<br />

times every 5 m<strong>in</strong> ( systolic BP>90 mm Hg )<br />

• Aspir<strong>in</strong>


Myocardial <strong>in</strong>farct<br />

• If chest pa<strong>in</strong> > 20 m<strong>in</strong> consider MI<br />

• Cardiac monitor<br />

• Morph<strong>in</strong>e 2 – 4 mg IV<br />

• EMS transport


Adverse drug reactions with<br />

local anes<strong>the</strong>tics


Types of local anes<strong>the</strong>tic reactions<br />

• Local anes<strong>the</strong>tic toxicity<br />

• Drug <strong>in</strong>teractions<br />

• Vasoconstrictor <strong>in</strong>teractions<br />

• Me<strong>the</strong>moglob<strong>in</strong>emia


Dosages


How Anes<strong>the</strong>tic overdose can<br />

occur


Cl<strong>in</strong>ical Signs


Treatment<br />

• ABC<br />

• Oxygen<br />

• Vitals<br />

• EMS<br />

• Monitor seizures<br />

• Monitor respiration<br />

• Cardiac monitor


Drug <strong>in</strong>teractions


Vasoconstrictor <strong>in</strong>teractions


Treatment<br />

• ABC<br />

• Patient comfort<br />

• Vitals<br />

• EMS<br />

• Reassurance reaction will pass<br />

• If BP becomes >170 systolic consider<br />

nitroglycer<strong>in</strong>


Me<strong>the</strong>moglob<strong>in</strong>emia<br />

• Dose dependent reaction<br />

• Adm<strong>in</strong>istration of Nitrates, amide<br />

conta<strong>in</strong><strong>in</strong>g drugs ( priloca<strong>in</strong>e, Benzoca<strong>in</strong>e )<br />

• Pathophysiology : oxidation of <strong>the</strong> iron<br />

with<strong>in</strong> hemoglob<strong>in</strong> produc<strong>in</strong>g<br />

me<strong>the</strong>moglob<strong>in</strong>


Cl<strong>in</strong>ical signs<br />

• Cyanosis at me<strong>the</strong>moglob<strong>in</strong> levels of 10%<br />

to 20%<br />

• Dyspnea and tachycardia at metHb level<br />

of 35% to 40%


Treatment<br />

• ABC<br />

• Oxygen<br />

• EMS<br />

• Monitor patient vitals , Cardiac<br />

• Most healthy adults drugs and metabolites<br />

are elim<strong>in</strong>ated<br />

• Methylene blue 1-21<br />

2 mg/kg IV


Airway<br />

Allergy<br />

Obstruction<br />

Asthma<br />

Hyperventilation


Allergy and anaphylaxis


Drug allergy & Anaphylaxis<br />

• Adverse drug reactions occur <strong>in</strong> 1% to<br />

15% of drug regimens<br />

• Drug allergy < 2% overall except for some<br />

common agents : penicill<strong>in</strong> ,<br />

cephalospor<strong>in</strong>, and trimethoprim-<br />

sulfamethoxazole ( Sulfa )


Risks Factors<br />

• Multiple <strong>in</strong>termittent exposures<br />

• Parenteral vs oral<br />

• Children less chance of develop<strong>in</strong>g reactions to<br />

meds because of shorter exposure times<br />

• Women higher <strong>in</strong>cidence of cutaneous reactions<br />

secondary to <strong>the</strong>ir <strong>in</strong>creased exposure to<br />

cosmetics and latex gloves<br />

• Individuals with multiple illnesses ,<br />

polypharmacy<br />

• Allergies to foods


Gell & Coombs Classification<br />

• Type 1 ( IgE – Mediated Hypersensitivity)<br />

most life threaten<strong>in</strong>g<br />

few m<strong>in</strong>utes<br />

• Type 2 ( Cytotoxic / Cytolytic antibody<br />

mediated) IgM or IgG antibodies mediate<br />

• Type 3 ( Immnune complex mediated )<br />

1- 4 weeks, IgM – IgG soluble metabolite<br />

• Type 4 (delayed Hypersensitivity )<br />

sensitized T cell lymphocytes


Signs & Symptoms of m<strong>in</strong>or allergic<br />

reactions


Signs & Symptoms of Anaphylaxis


Treatment<br />

• ABC<br />

• Establish reaction type<br />

• Activate EMS<br />

• IV access


Medications for treatment


M<strong>in</strong>or reactions


Anaphylaxis


Management of allergic scenarios


Medications<br />

• Diphenhydram<strong>in</strong>e 50 mg IM , IV<br />

• Ep<strong>in</strong>ephr<strong>in</strong>e: .3ml 1/1000 ( 0.3mg )<br />

• Dexamethasone : 20 mg IM , IV


Obstructed airway


Etiology<br />

• Foreign body aspiration<br />

• Laryngeal edema


Basics treatment of obstructed<br />

airway


Cricothyrotomy


Asthma


Types of Asthma<br />

• Extr<strong>in</strong>sic : allergic asthma, younger<br />

patients , Type 1 hypersensitivity Rx<br />

• Intr<strong>in</strong>sic : older patients, nonallergic<br />

factors , cold temperatures, exercise,<br />

stress


Asthma medications


What is asthma <br />

• Basically it is slow<br />

progress<strong>in</strong>g<br />

Bronchospasm


Treatment<br />

• Term<strong>in</strong>ate <strong>the</strong>rapy<br />

• Position patient<br />

• Adm<strong>in</strong>ister B agonist spray Albuterol<br />

• Oxygen<br />

• EMS<br />

• Ep<strong>in</strong>ephr<strong>in</strong>e SC or IM 0.3ml ( 1/1000<br />

dilution) Epipen


Hyperventilation


• Usually a patient which suffers from:<br />

panic, phobias, psychiatric disorder<br />

• Identify patient early


Signs and symptoms<br />

• Sigh<strong>in</strong>g<br />

• Tachypnea<br />

• Shortness of breath<br />

• Pa<strong>in</strong> on respiration<br />

• Tachycardia<br />

• Nonradiat<strong>in</strong>g chest pa<strong>in</strong><br />

• Lungs clear to<br />

auscultation<br />

• Normal oxygen saturation<br />

• Dizz<strong>in</strong>ess<br />

• fa<strong>in</strong>tness<br />

• Altered consciousness<br />

• Muscle cramp<br />

• Tremor<br />

• Myalgia


Treatment<br />

• Reassurance<br />

• Slow down breath<strong>in</strong>g<br />

• Comfortable position<br />

• Remove any visual stimuli<br />

• Vitals<br />

• Full rebreath<strong>in</strong>g bag<br />

• Anxiolytic meds , Diazepam


Bibliography<br />

• Handbook of <strong>Medical</strong> <strong>Emergencies</strong> <strong>in</strong> <strong>the</strong><br />

<strong>Dental</strong> <strong>Office</strong>, Stanley F. Malamed<br />

• <strong>Medical</strong> <strong>Emergencies</strong> <strong>in</strong> Dentistry, Jeffrey<br />

D. Bennett , Morton B. Rosenberg<br />

• ACLS provider manual , American Heart<br />

Association

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