Neopl-L1-15

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Hyperplasia• increased organ/tissue mass due to increased number of cells.• recall, hypertrophy and hyperplasia are not mutually exclusive.a) Etiologyi) Physiologic Hyperplasia• physiologic hormonal stimulation• compensatory hyperplasiaii) Pathologic Hyperplasia• excessive hormonal stimulation• chronic irritation (via growth factors)
Hyperplasiab) Mechanisms / Biochemistry increased production of growth factors / hormones. increased expression of growth factor receptors. activation of specific intracellular signaling pathways.• It is reversible, it regresses when stimulus is removed (unlikeneoplasia).• pathologic hyperplasia is a “fertile soil” for the development ofneoplasia.
Page 1 and 2: General Pathology VPM 152Disorders
Page 3 and 4: Complementary textbooks:• Kumar,
Page 5 and 6: NORMAL TISSUE GROWTH AND CELL PROLI
Page 7 and 8: Stem Cellsb) Primordial Germ Cells
Page 9 and 10: Adult Stem CellsBStem cell niches i
Page 11 and 12: Aplasia• failure of an organ / ti
Page 13 and 14: Unilateral hypoplasia (right sided)
Page 15: Causes of Developmental Anomalies
Page 19 and 20: TGoiter, thyroid gland, goat fetus.
Page 21 and 22: Pathologic Basis of Veterinary Dise
Page 23 and 24: Nodular hyperplasia, liver, dog. Si
Page 25 and 26: Nodular adrenal cortical hyperplasi
Page 27 and 28: Metaplasia• one adult cell type i
Page 29 and 30: CRobbins and Cotran Pathologic Basi
Page 31 and 32: Joseph Merrick photographed in 1889

Neopl-L1-15

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