Types of Homotoxins

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Chemical poisoning: principal pollutants • Organic compounds... • Non-halogenated compounds: • Aliphatic compounds (formaldehyde, methane) • Aromatic compounds (benzene, toluene) • Halogenated compounds: • Aliphatic compounds (chlorofluorocarbons, trichloroethane) • Aromatic compounds (chlorobenzene, dioxins) © IAH 2007 14 Between the chemicals also a lot of halogen and non-halogen compounds are extremely toxic and even can cancerogenous. Because formaldehyde resins are used in many construction materials, including plywood, carpet, and spray-on insulating foams, and because these resins slowly give off formaldehyde over time, formaldehyde is one of the more common indoor air pollutants. At concentrations above 0.1 mg/kg in air, inhaled formaldehyde can irritate the eyes and mucous membranes, resulting in watery eyes, headache, a burning sensation in the throat, and difficulty breathing. Benzene exposure has serious health effects. Breathing high levels of benzene can result in death, while low levels can cause drowsiness, dizziness, rapid heart rate, headaches, tremors, confusion, and unconsciousness. Eating or drinking foods containing high levels of benzene can cause vomiting, irritation of the stomach, dizziness, sleepiness, convulsions, rapid heart rate, and death. Chlorobenzene has been used in the manufacture of certain pesticides, most notably DDT by reaction with chloral (trichloroacetaldehyde). It also once found use in the production of phenol. However, use of these manufacturing processes has declined significantly in the past few decades. Today the major use of chlorobenzene is as an intermediate in the production of nitrochlorobenzenes and diphenyl oxide, which are important in the production of commodities such as herbicides, dyestuffs, and rubber Dioxins build up in living tissue (bioaccumulation) over time, so even small exposures may accumulate to dangerous levels. TCDD has a half-life of ~7 years in humans, but at high concentrations, the elimination rate is enhanced by metabolism. The health effects of dioxins are mediated by their action on a cellular receptor, the Aryl Hydrocarbon Receptor (AhR). Exposure to high levels of dioxin in humans causes a severe form of persistent acne, known as chloracne. Other effects may include: developmental abnormalities in the enamel of children's teeth, damage to the immune system, endometriosis, birth defect, COPD, diabetes and at least in laboratory animals, increased rates of liver and thyroid cancer are observed 14
Chemical poisoning: principal pollutants • Other substances... • Asbestos • Carbon dioxide • Carbon monoxide © IAH 2007 15 Other substances, better known by the public but extremely toxic too are substances like asbestos, carbon dioxide and carbon monoxide. Although great efforts are done in Europe to get asbestos banned out of building materials and fire protection materials, it is still in mass in the environment as it remains for years. The hook-like particles of asbestos can intrude in the deepest parts of the lungs and are known to be cancerogenic. CO 2 and CO both are polluating toxic gasses that are very common in modern societies, especially living in heavy traffic and industrial regions. 15
Page 1 and 2: Types of Homotoxins IAH AC Types of
Page 3 and 4: Agenda: • Definition of homotoxin
Page 5 and 6: Types of homotoxins • Exogenous h
Page 7 and 8: Homotoxins: • Chemical toxins •
Page 9 and 10: Chemical poisoning: toxicological c
Page 11 and 12: Chemical poisoning: toxicological c
Page 13: Chemical poisoning: principal pollu
Page 17 and 18: Factors determining homotoxic overl
Page 19 and 20: Atmosphere - pollution • Pollutan
Page 21 and 22: Metals and metalloids • Classific
Page 23 and 24: Metals and metalloids • Toxicity
Page 25 and 26: Toxicity of metals according to dos
Page 27 and 28: Factors determining homotoxic overl
Page 29 and 30: Insidious homotoxins… © IAH 2007
Page 31 and 32: Electromagnetic fields in everyday
Page 33 and 34: Geopathies • In geopathogenic zon
Page 35 and 36: Sick-building-syndrome • Causes:
Page 37 and 38: Noise pollution • At levels in ex
Page 39 and 40: Food homotoxicity • Industrialize
Page 41 and 42: Aggressive therapies • Long-term
Page 43 and 44: Aggressive therapies: antibiotics
Page 45 and 46: Aggressive therapies • Anti-cance
Page 47 and 48: Chemotherapy • Modulation • Tra
Page 49 and 50: Interference field: histology •
Page 51 and 52: Psychotoxicity © IAH 2007 An homot
Page 53 and 54: Psychotoxins • Modulation: • Ce

Types of Homotoxins

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