Topical Management of arterial and venous ulcers - VascularWeb

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Common final pathway in chronicwounds: Unregulated inflammation• Sources– venous hypertension– Chronic pressure– Bacterial overgrowthTreatment methods– Compression0.020.0180.0160.0140.0120.010.0080.0060.0040.002– Contact cast 0TNF-a in venous ulcerNormalBefore
Rx protocol for VLU• Compression• Wound debridement/elimination ofbacteria• Exudate control• Adjuvant therapies to accelerate closure• Prevention of recurrence
Page 1 and 2: Professor of Surgery Chief, Divisio
Page 3 and 4: DisclosuresScientific Consultant•
Page 6 and 7: Venous/lymphatic ulcer• Common as
Page 8 and 9: • May be inatypicallocations• T
Page 10: Causes of Atypical WoundsInfectious
Page 13: When to biopsy a• Long durationch
Page 17 and 18: Coexistent arterial insufficiency:H
Page 19 and 20: Topical therapy• Wound debridemen
Page 21 and 22: Special considerations forpatients
Page 23 and 24: How much to debride?
Page 25 and 26: The necrotic toe
Page 27 and 28: Wound bacterial load• Bacterial c
Page 29 and 30: Biofilm/bacterial burden
Page 31 and 32: Wound bacteria data - chronicVLU•
Page 33 and 34: Vulcan trial• Michaels et al Br J
Page 35 and 36: What to put on the wound?• Over 5
Page 37 and 38: Demonstrated effects of chronicwoun
Page 39 and 40: Exudate Management:DressingsNone Lo
Page 41 and 42: Hydrocolloids• Provide barrier if
Page 43 and 44: Foams, Specialty absorbents• High
Page 45 and 46: Negative pressure compared tomoist

Topical Management of arterial and venous ulcers - VascularWeb

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