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MEDICINSKA ISTRAŽIVANJA VOL. 45 / SVESKA 1 / 2011 Figure 4a. APMPPE, central retinal vein occlusion, occurring after APMPPE in the left eye and evolving towards secondary glaucoma Figure 4b. APMPPE of the right eye, one year later, after the evolution of the disease in the left eye Case No. 5 (F age 32). This patient was diagnosed with APMPPE in both eyes. After a month, the disease evolved in the left eye towards Birdshot retinopathy and papilloedema of the optic nerve. 34 Figure 5a. APMPPE in the left eye Figure 5b. Birdshot retinopathy and papilloedema, after one month of the disease evolution Case No. 6 (M age 55) The patient had two clinical forms of White dot syndrome in the same eye: APMPPE and serpiginous choroidopathy. Placoid lesions appeared in various forms of the disease evolution. APMPPE and serpiginous choroidopathy evolved towards the severe form of serpiginous choroidopathy.
MEDICINSKA ISTRAŽIVANJA VOL. 45 / SVESKA 1 / 2011 Figure 6a. APMPPE on the right eye, placoid lesions in different stages of evolution (2003) Figure 6b. Serpiginous choroidopathy and APMPPE in the right eye, FA (2003) Figure 6c. Severe form of serpiginous choroidopathy two years after the first occurrence of the disease (2006) 35 Case No. 7 (F age 47) The patient suffered from multifocal choroiditis and panuveitis located bilaterally, preceded by juxtapapillar subretinal neovascularisation. In the observed term in the right eye placoid lesions were diagnosed of APMPPE type, equatorially and on the periphery. In the left eye the disease evolved into the severe form of serpiginous choroidopathy. Figure 7a. Juxtapapillar neovascular membrane in the right eye in White dot syndrome (2003) Figure 7b. Multifocal choroiditis and AMPPE in the right eye two years later (2005)
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