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<strong>Acute</strong> <strong>myocardial</strong> <strong>infarction</strong>, <strong>Definition</strong>, E...<br />

by garest - http://garest.net/acute-<strong>myocardial</strong>-<strong>infarction</strong>-definition-etiology-signs-and-symptoms-acute-<strong>myocardial</strong>-in<br />

farction.html<br />

<strong>Acute</strong> <strong>myocardial</strong> <strong>infarction</strong>, Defi<br />

nition, <strong>Etiology</strong>, <strong>SIGNS</strong> <strong>AND</strong> <strong>SYMPTOMS</strong> <strong>Acute</strong> <strong>myocardial</strong><br />

<strong>infarction</strong><br />

<strong>Acute</strong> <strong>myocardial</strong> <strong>infarction</strong><br />

A. <strong>Acute</strong> <strong>myocardial</strong> <strong>infarction</strong> DEFINITIONS<br />

Myocardial <strong>infarction</strong> refers to the process of the destruction of heart tissue due to<br />

inadequate blood supply so that coronary blood flow is reduced.<br />

(Brunner & Sudarth, 2002)<br />

<strong>Acute</strong> <strong>myocardial</strong> <strong>infarction</strong> is <strong>myocardial</strong> necrosis caused by blood flow to the heart muscle<br />

impaired. (Suyono, 1999)<br />

B. <strong>Acute</strong> <strong>myocardial</strong> <strong>infarction</strong> <strong>Etiology</strong> (cassowaries, 2002)<br />

1. factor:<br />

a. Reduced <strong>myocardial</strong> oxygen supply due to 3 factors:<br />

Vascular factors: Atherosclerosis,Spasms,arteritis,<br />

Factors circulation: Hypotension, Stenosos aurta,insufficiency,<br />

Blood Factor: Anemia,Hypoxaemia, polycythemia<br />

b. Increased cardiac output:<br />

- Excessive activity<br />

- Emotions<br />

- Eating too much<br />

- Hypertiroidisme<br />

c. Increased <strong>myocardial</strong> oxygen demand:<br />

- Damage miocard<br />

- Hypertropimiocard<br />

- Hypertension diastolic<br />

2. Predisposing factors:<br />

a. biological risk factors can not be changed:<br />

- Age over 40 years<br />

- Gender: a high incidence in men, whereas in women increases after menopause<br />

- Heredity<br />

- Race: higher incidence in blacks.<br />

b. Risk factors that can be changed:<br />

- Mayor: hyperlipidemia, hypertension, Smoking, Diabetes, Obesity, A diet high in saturated<br />

fat, calories<br />

- Minor: Physical In actifity, pattern type A personality (emotional, aggressive, ambitious,<br />

competitive),excessive psychological stress.<br />

C. <strong>Acute</strong> <strong>myocardial</strong> <strong>infarction</strong> <strong>SIGNS</strong> <strong>AND</strong> <strong>SYMPTOMS</strong><br />

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<strong>Acute</strong> <strong>myocardial</strong> <strong>infarction</strong>, <strong>Definition</strong>, E...<br />

by garest - http://garest.net/acute-<strong>myocardial</strong>-<strong>infarction</strong>-definition-etiology-signs-and-symptoms-acute-<strong>myocardial</strong>-in<br />

farction.html<br />

Signs and symptoms of <strong>myocardial</strong> <strong>infarction</strong> (TRIAD) is:<br />

1. Pain:<br />

a. Chest pain that occurs suddenly and constantly not subside, usually above the sternal<br />

region and upper abdomen, this is the main symptom.<br />

b. The severity of pain can increase secaara settled until the pain becomes unbearable.<br />

c. Pain is very sick, such as punctured and needles that can spread to the shoulder and<br />

continues down the arm (usually the left arm).<br />

d. The pain began spontaneously (not happening after the activity or emotional disturbance),<br />

persist for several hours or days, and not relieved by rest or nitroglycerin assistance (NTG).<br />

e. The pain may spread to the jaw and neck.<br />

f. Pain is often accompanied by shortness of breath, pale, cold, severe diaphoresis, dizziness<br />

or head was floating, and nausea and vomiting.<br />

g. Patients with diabetes mellitus will not experience severe pain because of neuropathy that<br />

accompanies diabetes can interfere neuroreseptor (accumulated experience of pain).<br />

2. Laboratory<br />

Examination Cardiac enzymes:<br />

a. CPK-MB/CPK<br />

Isoenzymes were found in the heart muscle to increase between 4-6 hours, peaks within 12-24<br />

hours, returning to normal within 36-48 hours.<br />

b. LDH / HBDH<br />

Increased within 12-24 hours dam takes longer to return to normal<br />

c. AST / SGOT<br />

Increases (less real / special) occurs within 6-12 hours, culminating in 24 hours, returning to<br />

normal within 3 or 4 days<br />

d. Troponin<br />

3. ECG<br />

ECG changes that occur in the early phase of the T wave height and symmetrical. After this<br />

there is ST segment elevation. Change happens then is the wave Q / QS which indicates<br />

necrosis<br />

Pain scores <strong>Acute</strong> <strong>myocardial</strong> <strong>infarction</strong> according to White:<br />

0 = no pain<br />

1 = pain on one side without disturbing activity<br />

2 = more pain in one place and lead to disruption of activities, such as difficulty getting out of<br />

bed, it’s hard to bend the head and others.<br />

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