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Mental Illness and Serious Harm to Others - University of Liverpool

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NHS National Programme on Forensic <strong>Mental</strong> Health Research <strong>and</strong> Development<br />

1992). It may be, <strong>to</strong>o, that in a treated sample, treatment has an effect. Another<br />

possibility may be real international or community differences. Although studies are<br />

stronger on correlates than causes <strong>of</strong> violence in the context <strong>of</strong> illness, there are some<br />

which do begin <strong>to</strong> disentangle cause <strong>and</strong> effect (Taylor, 1993; Taylor et al, 1994) <strong>and</strong><br />

possible pathways (e.g. Steadman <strong>and</strong> Silver, 2000). There is good evidence that violent<br />

<strong>of</strong>fending specifically is more likely than not <strong>to</strong> post-date the onset <strong>of</strong> illness (Taylor <strong>and</strong><br />

Hodgins, 1994).<br />

3.3 Contemporaneous substance misuse <strong>and</strong> other comorbidities<br />

Few doubt that through the 1990s, <strong>and</strong> maybe also the 1980s, alcohol <strong>and</strong> illicit drug use<br />

have been associated with higher rates <strong>of</strong> violence among people with mental illness, as it<br />

has among those without (Soyka, 2000), but there most certainty ends. Swanson et al<br />

(1990) may have been the first <strong>to</strong> advance epidemiological evidence from the community<br />

for the association, but most subsequent epidemiological studies concurred, regardless <strong>of</strong><br />

jurisdiction. At about the same time that this evidence was emerging, it was also becoming<br />

apparent that people with schizophrenia were four times as likely <strong>to</strong> misuse such<br />

substances as their healthy peers, <strong>and</strong> people with mania six times as likely (Regier at al,<br />

1990). Changed availability <strong>of</strong> substances <strong>of</strong> misuse may be one <strong>of</strong> the most important<br />

effects <strong>of</strong> the shift <strong>to</strong> community care <strong>of</strong> people with mental disorder, <strong>and</strong> could account for<br />

any changes in violence rates. Through the 1950s, ‘60s, <strong>and</strong> ‘70s, a number <strong>of</strong> independent<br />

studies in Europe <strong>and</strong> the USA had shown an inverse relationship between substance<br />

misuse <strong>and</strong> violence among people with schizophrenia (Häfner <strong>and</strong> Böker, 1973;<br />

Virkkunen, 1974; Tardiff <strong>and</strong> Sweillam, 1980; Taylor, 1993). Further, changes in fashion or<br />

availability <strong>of</strong> illicit drugs may lead <strong>to</strong> varying trends in association. The nature <strong>and</strong><br />

extent <strong>of</strong> influence on behaviour is not equivalent between drugs.<br />

Tiihonen <strong>and</strong> Swartz (2000) provide perhaps the best framework for underst<strong>and</strong>ing or<br />

setting hypotheses about the interactions. They list:<br />

❑ Substance abuse reduces impulse control.<br />

❑ Substance abuse exacerbates psychotic <strong>and</strong> other symp<strong>to</strong>ms.<br />

❑ Substance abuse is a proxy measure for personality disorder.<br />

❑ Medication side-effects lead <strong>to</strong> self-medication with illicit substances.<br />

❑ Medication non-compliance leads <strong>to</strong> substance use.<br />

❑ Psychotic <strong>and</strong> other symp<strong>to</strong>ms lead <strong>to</strong> self-medication.<br />

❑ Exposure <strong>to</strong> adverse social environments leads <strong>to</strong> substance use <strong>and</strong> aggressive<br />

behaviour.<br />

❑ Boredom <strong>and</strong> lack <strong>of</strong> structure lead <strong>to</strong> substance use.<br />

There is no epidemiological evidence <strong>to</strong> support any one <strong>of</strong> these hypotheses over any<br />

other. Indeed, Tiihonen <strong>and</strong> Swartz plausibly suggest that the hypotheses are not<br />

mutually exclusive. They might be applied with advantage <strong>to</strong> test relative risks in each<br />

individual case. Mueser et al (1998b), in a review <strong>of</strong> aetiological theories, were<br />

unimpressed only with evidence for the self-medication model. Mueser <strong>and</strong> colleagues<br />

(1998a) have also summarised the problems <strong>of</strong> 'traditional' treatment approaches for<br />

people who have major mental disorder <strong>and</strong> substance abuse problems, <strong>and</strong> reviewed<br />

9

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