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Testing Gateway Theory: do cigarette prices affect illicit drug use?

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692 M. Beenstock, G. Rahav / Journal of Health Economics 21 (2002) 679–698<br />

Table 4<br />

Comparison between 2SP and 2SBP<br />

Variable 2SP 2SBP<br />

Cigarette <strong>prices</strong> −0.00553 (7.119) −0.00538 (7.133)<br />

ρ 0 0.57 (27.1)<br />

PPS 3.228 (13.058) 3.402 (14.300)<br />

The t-values are shown in parentheses.<br />

two reasons. First, beca<strong>use</strong> PPS is an estimate of its true value. Secondly, beca<strong>use</strong> in any<br />

case IV estimation requires correction of the standard errors in the second stage. We <strong>use</strong> 200<br />

i.i.d. resamplings to calculate the standard errors. Andrews and Buchinsky (1999) discuss<br />

the number of resamplings required to attain a prescribed degree of accuracy for different<br />

P-values. It turns out that the original (unbootstrapped) standard errors provide a reliable<br />

guide to statistical significance. Also, the original parameter estimates reported in Table 3<br />

turn out to be close to the means of their bootstrapped counterparts, suggesting that the<br />

crude estimates are unbiased.<br />

It is noteworthy that if the relative price of <strong>cigarette</strong>s is omitted from Table 2, PPS ceases<br />

to be statistically significant in Table 3. This suggests that there is identifying information<br />

in <strong>cigarette</strong> <strong>prices</strong>, and that the significance of PPS in Table 3 is not simply the result of<br />

parametric identification.<br />

We experimented with other instruments, including herd effects and exposure to <strong>cigarette</strong><br />

regulations. In the former case we <strong>use</strong>d “personalized” <strong>cigarette</strong> consumption per capita<br />

(based in Fig. 2) as an additional covariate in Table 2, but it was not statistically significant. 7<br />

In the latter case, we defined exposure to be the number of adult years lived prior to the<br />

introduction of mandatory health warnings on <strong>cigarette</strong> packets. We expected that more<br />

exposed individuals are more likely to smoke, holding other variables constant. However,<br />

this did not turn out to be the case. This was true when we <strong>use</strong>d a variety of health warning<br />

trigger dates (1983 Israel, 1971 UK, 1965 US) both individually and collectively. If anything,<br />

we found that more exposed individuals, as defined, were less likely to smoke. However, this<br />

may be the manifestation of some complex cohort effect; early birth cohorts are less likely<br />

to smoke. In summary, the main instrument for smoking turned out to be the personalized<br />

relative price of <strong>cigarette</strong>s.<br />

Other results in Table 3 include a negative cohort effect; older birth cohorts are less likely<br />

to <strong>use</strong> cannabis, Sabras (native Israelis) are more likely to <strong>use</strong> cannabis, the uneducated and<br />

the highly educated are more likely to <strong>use</strong> cannabis. When controlling for other factors,<br />

women and secular Jews are more likely to <strong>use</strong> cannabis.<br />

As noted in Section 2.2, 2SL is consistent but not efficient, beca<strong>use</strong> it restricts ρ = 0.<br />

Inefficiency may induce bias in non-linear estimators. To investigate this we re-estimated the<br />

model using two-stage probit (2SP), solved for PPS, and then iterated PPS using two-stage<br />

bivariate probit (2SBP). Coefficient estimates for the main variables of interest are shown<br />

in Table 4.<br />

7 It is possible that prevalence data would have produced different results. See Footnote 5.

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