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Pulpal Diagnosis - University at Buffalo

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Eugene A. Pantera, Jr., DDS, MS<br />

Department of Periodontics and Endodontics<br />

Clinical <strong>Diagnosis</strong><br />

<strong>University</strong> <strong>at</strong> <strong>Buffalo</strong><br />

<strong>Pulpal</strong> <strong>Diagnosis</strong><br />

Endodontic Tre<strong>at</strong>ment is NOT Needed<br />

CLINICAL CLASSIFICATION SIGNS AND SYMPTOMS DIAGNOSTIC TEST RESULTS<br />

Normal<br />

A pulpal condition, usually called normal, in<br />

which the pulp responds to thermal and<br />

electrical tests in a manner similar to th<strong>at</strong> of a<br />

corresponding control tooth.<br />

Hypersensitive Dentin<br />

A pulpal condition, with no apparent<br />

histologic changes, in which the p<strong>at</strong>ient feels<br />

pain when the dentin is exposed to touch<br />

from a dental explorer, fingernail or tooth<br />

brush and to thermal or to other stimuli.<br />

However the pain disappears when the<br />

stimulus is removed.<br />

Reversible Pulpitis<br />

Syn: hyperemia, inflamed-reversible.<br />

A pulpal condition commonly induced by<br />

dental caries and oper<strong>at</strong>ive procedures, in<br />

which the p<strong>at</strong>ient responds to thermal or<br />

osmotic stimuli, but the symptoms disappear<br />

when the etiology is elimin<strong>at</strong>ed.<br />

Pain<br />

No history of pain.<br />

None to mild to moder<strong>at</strong>e intensity<br />

Pain is non-spontaneous<br />

No pain with percussion<br />

Moder<strong>at</strong>e to sharp response to thermal, sweet, or<br />

sour stimuli; response subsides when<br />

stimulus is removed.<br />

Etiology<br />

Usually caries, defective restor<strong>at</strong>ion, restor<strong>at</strong>ive<br />

procedures, mechanical pulp exposures,<br />

tooth brush abrasion, tooth fracture,<br />

recent prophylaxis, or subgingival scaling<br />

and curettage.<br />

Radiology<br />

No radiographic evidence of pulp calcific<strong>at</strong>ions,<br />

internal resorption, or periapical changes.<br />

EPT<br />

Response is normal and not in extremes. Is<br />

generally in the same range as control<br />

teeth.<br />

Thermal Tests<br />

Usual, normal response, for dur<strong>at</strong>ion of the<br />

stimulus. Reaction may be severe, but<br />

BRIEF.<br />

Percussion and Palp<strong>at</strong>ion<br />

Neg<strong>at</strong>ive, no response.<br />

1


Eugene A. Pantera, Jr., DDS, MS<br />

Department of Periodontics and Endodontics<br />

<strong>Pulpal</strong> <strong>Diagnosis</strong><br />

Endodontic Tre<strong>at</strong>ment IS Needed<br />

CLINICAL CLASSIFICATION SIGNS AND SYMPTOMS DIAGNOSTIC TEST RESULTS<br />

Irreversible Pulpitis<br />

Irreversible pulpitis without periapical p<strong>at</strong>hosis<br />

A pulpal condition, usually caused by deep<br />

dental caries or restor<strong>at</strong>ions, in which<br />

spontaneous pain may occur or be<br />

precipit<strong>at</strong>ed by thermal or other stimuli.<br />

Radiographs show no periapical changes.<br />

The pain last for several minutes to hours.<br />

Irreversible pulpitis with periapical p<strong>at</strong>hosis<br />

A pulpal condition similar to above, but in<br />

which periapical or l<strong>at</strong>eral radiographic<br />

changes are evident.<br />

Pain<br />

May have acute or chronic symptoms.<br />

Sharp, exagger<strong>at</strong>ed, painful response to thermal<br />

stimulus; pain lingers after stimulus is<br />

removed.<br />

Pain may be spontaneous; maybe past repe<strong>at</strong>ed<br />

episodes of pain, often continuous pain.<br />

Pain with mastic<strong>at</strong>ion.<br />

Etiology<br />

Deep caries and/or restor<strong>at</strong>ions, evidence of<br />

previous pulp cap.<br />

Exposed dentin (<strong>at</strong>trition, abrasion, and erosion).<br />

Traum<strong>at</strong>ic injuries.<br />

Resorption (especially perfor<strong>at</strong>ing).<br />

Orthodontic forces.<br />

EPT<br />

Tooth may test within normal limits.<br />

Response may be markedly different from<br />

control, rapid/delayed onset, persistent, and<br />

may be of severe intensity.<br />

Thermal Test<br />

A key factor in making a diagnosis.<br />

May be abnormal, rapid/delayed onset, gre<strong>at</strong>er<br />

intensity and longer dur<strong>at</strong>ion.<br />

Percussion Test<br />

May or may not be positive.<br />

Palp<strong>at</strong>ion<br />

May or may not be positive.<br />

Radiology<br />

May be normal.<br />

Radiographic evidence may reveal normal pulp,<br />

calcific<strong>at</strong>ions, narrow pulp chamber,<br />

"calcified" canals, , or condensing osteitis.<br />

An enlarged PDL may also be present.<br />

2


Eugene A. Pantera, Jr., DDS, MS<br />

Department of Periodontics and Endodontics<br />

<strong>Pulpal</strong> <strong>Diagnosis</strong><br />

Endodontic Tre<strong>at</strong>ment IS Needed<br />

CLINICAL CLASSIFICATION SIGNS AND SYMPTOMS DIAGNOSTIC TEST RESULTS<br />

Necrotic Pulp<br />

Necrotic pulp without periapical p<strong>at</strong>hosis<br />

A pulpal condition in which there may or<br />

may not be spontaneous moder<strong>at</strong>e to severe<br />

pain or pain elicited by various stimuli.<br />

Response to various testing modalities is<br />

usually absent. Radiographic changes are not<br />

evident.<br />

Necrotic pulp with periapical p<strong>at</strong>hosis<br />

A pulpal condition similar to above, except<br />

th<strong>at</strong> in this c<strong>at</strong>egory periapical or l<strong>at</strong>eral<br />

lesions are evident in radiographs.<br />

Pain<br />

May have acute or chronic symptoms.<br />

Pain may be spontaneous; maybe past repe<strong>at</strong>ed<br />

episodes of pain, often continuous, dull,<br />

throbbing, pain.<br />

Pain with mastic<strong>at</strong>ion.<br />

Etiology<br />

Deep caries and/or restor<strong>at</strong>ions, evidence of<br />

previous pulp cap.<br />

Carious pulp exposures.<br />

Exposed dentin (<strong>at</strong>trition, abrasion, and erosion).<br />

Traum<strong>at</strong>ic injuries.<br />

Resorption (especially perfor<strong>at</strong>ing).<br />

Orthodontic forces.<br />

EPT<br />

No response.<br />

May have false positives.<br />

Thermal Test<br />

No response.<br />

Percussion Test<br />

May or may not be positive.<br />

Palp<strong>at</strong>ion<br />

May or may not be positive.<br />

Radiology<br />

May be normal.<br />

May be periapical lesions or l<strong>at</strong>eral lesions.<br />

An enlarged PDL may also be present.<br />

3


Eugene A. Pantera, Jr., DDS, MS<br />

Department of Periodontics and Endodontics<br />

Periapical <strong>Diagnosis</strong><br />

There MAY be Need for Endodontic Tre<strong>at</strong>ment<br />

PULPAL DIAGNOSIS IS REQUIRED FOR DEFINITIVE DETERMINATION<br />

CLINICAL CLASSIFICATION SIGNS AND SYMPTOMS DIAGNOSTIC TEST RESULTS<br />

Acute Apical Periodontitis<br />

Pain<br />

History of pulpal pain.<br />

Moder<strong>at</strong>e to severe intensity th<strong>at</strong> may be<br />

intermittent.<br />

Periapical pain usually requires stimulus (eg<br />

mastic<strong>at</strong>ion).<br />

Pain can be sharp.<br />

Aggrav<strong>at</strong>ing factors are usually present.<br />

Etiology<br />

Irreversible pulpitis, traum<strong>at</strong>ic injuries,<br />

periodontal disease, orthodontic forces maxillary<br />

sinusitis, pressure from periapical tumors,<br />

restor<strong>at</strong>ion in hyperocclusion.<br />

Pulp Tests<br />

EPT and Thermal tests may be normal, or<br />

similar to irreversible pulpitis or pulpal<br />

necrosis.<br />

Percussion<br />

Moder<strong>at</strong>e to severe pain.<br />

Palp<strong>at</strong>ion<br />

Moder<strong>at</strong>e to severe pain.<br />

Radiology<br />

Usually thickening of PDL, also can have<br />

periapical or l<strong>at</strong>eral radiolucency, or normal.<br />

Chronic Apical Periodontitis<br />

A periapical condition characterized by none<br />

to slight pain on mastic<strong>at</strong>ion but may present<br />

itself with varying degrees of apical swelling.<br />

Radiographs reveal periapical or l<strong>at</strong>eral<br />

radiolucencies.<br />

Pain<br />

History of pain.<br />

Slight intensity to no pain. Pain may be absent or<br />

constant.<br />

Periapical pain can be spontaneous.<br />

Pain is dull throbbing.<br />

Pain can occur with mastic<strong>at</strong>ion.<br />

Etiology<br />

Same as for Acute apical periodontitis.<br />

Pulp Tests<br />

EPT and Thermal tests may be normal, or<br />

similar to irreversible pulpitis or pulpal<br />

necrosis.<br />

Percussion<br />

Moder<strong>at</strong>e to none.<br />

Palp<strong>at</strong>ion<br />

Moder<strong>at</strong>e to none. May be swelling.<br />

Radiology<br />

Periapical or l<strong>at</strong>eral radiolucency.<br />

4


Eugene A. Pantera, Jr., DDS, MS<br />

Department of Periodontics and Endodontics<br />

Periapical <strong>Diagnosis</strong><br />

There IS a Need for Endodontic Tre<strong>at</strong>ment<br />

PULPAL DIAGNOSIS IS REQUIRED FOR DEFINITIVE DETERMINATION<br />

CLINICAL CLASSIFICATION SIGNS AND SYMPTOMS DIAGNOSTIC TEST RESULTS<br />

Chronic Suppur<strong>at</strong>ive Apical Periodontitis<br />

A periapical condition characterized by non<br />

pain on mastic<strong>at</strong>ion. A draining sinus tract or<br />

other evidence of suppur<strong>at</strong>ion is evident.<br />

Radiographs reveal periapical or l<strong>at</strong>eral<br />

radiolucencies.<br />

Acute Alveolar Abscess<br />

Syn: Acute apical abscess<br />

An acute alveolar abscess is a severe clinical<br />

manifest<strong>at</strong>ion of periapical infectious<br />

disease, characterized by severe pain and<br />

swelling.<br />

Pain<br />

History of pain.<br />

Usually no pain present.<br />

Etiology<br />

Irreversible pulpitis, traum<strong>at</strong>ic injuries,<br />

periodontal disease, orthodontic forces,<br />

restor<strong>at</strong>ion in hyperocclusion.<br />

Pain<br />

History of pain.<br />

Severe intensity.<br />

Constant and spontaneous pain.<br />

Pain is pulsing and throbbing.<br />

Pain can occur with mastic<strong>at</strong>ion.<br />

Etiology<br />

The result of coronal apical progression of pulpal<br />

necrosis with resultant release of antigens into<br />

the periapical tissues.<br />

Pulp Tests<br />

EPT and Thermal tests may be normal, or<br />

similar to irreversible pulpitis or pulpal<br />

necrosis. (Is this a lesion of endodontic origin)<br />

Percussion<br />

None to slight pain.<br />

Palp<strong>at</strong>ion<br />

Slightly tender.<br />

Radiology<br />

Periapical or l<strong>at</strong>eral radiolucency.<br />

Visual<br />

Sinus tract present.<br />

Pulp Tests<br />

No response.<br />

Percussion<br />

Moder<strong>at</strong>e to severe.<br />

Palp<strong>at</strong>ion<br />

Moder<strong>at</strong>e to severe. Swelling probable.<br />

Radiology<br />

PDL thickening, periapical or l<strong>at</strong>eral<br />

radiolucency.<br />

5

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