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nanosymposium - Society for Neuroscience

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8:55 311.03 The specificity of synaptic connections underlying<br />

direction-selectivity in the retina. K. L. BRIGGMAN. MPI<br />

(Winfried Denk Laboratory).<br />

9:15 311.04 Anatomy and in vivo physiology from neuronal<br />

network in visual cortex. D. D. BOCK. Harvard - HMS (R.<br />

Clay Reid Laboratory).<br />

9:35 311.05 The diversity of cortical synaptic connections as<br />

revealed by array tomography. K. D. MICHEVA. Stan<strong>for</strong>d<br />

SoM (Stephen J. Smith Laboratory).<br />

9:55 311.06 Dynamics and control of cortical blood flow in<br />

relation to the angiotome. P. BLINDER. UCSD (David<br />

Kleinfeld Laboratory).<br />

10:15 311.07 The 3-D reconstruction of type-specific neuronal<br />

somata in the neocortex. B. SAKMANN. Max Planck Florida<br />

Inst.<br />

10:35 311.08 Closing Remarks.<br />

MINISYMPOSIUM Walter E. Washington Convention Center<br />

312. Emerging Roles <strong>for</strong> Somatostatin-Containing<br />

Cortical Interneurons: Novel Insights From<br />

Transgenic Mice — CME<br />

Mon. 8:30 AM - 11:00 AM — 145B<br />

Chair: A. AGMON<br />

Somatostatin-containing (SOM), inhibitory cortical<br />

interneurons have long been thought to play second fiddle<br />

to the better-known “fast spiking” (FS) interneurons. Studies<br />

using SOM-specific mouse lines are now challenging this<br />

perception, revealing how the unique properties of SOM<br />

interneurons, such as their facilitating inputs, distal dendritic<br />

targeting and sensitivity to neuromodulators, allow them<br />

to sculpt cortical activity in a manner distinct from, but<br />

complementary to, FS interneurons.<br />

8:30 312.01 Introduction.<br />

8:35 312.02 State-dependent contribution of SOM interneurons<br />

to cortical dynamics. H. ADESNIK. UCSD.<br />

8:55 312.03 Delayed visual representation by SOM<br />

interneurons. H. TAO. USC Keck Sch. Med.<br />

9:15 312.04 Layer 2/3 SOM cells act as an ON-switch in<br />

mouse somatosensory cortex in vivo. L. J. GENTET. Ecole<br />

Polytechnique Federale de Lausanne.<br />

9:35 312.05 The role of SOM interneurons in regulating sensory<br />

neocortical activity. U. KNOBLICH. Yale Univ.<br />

9:55 312.06 The roles of different subtypes of SOM<br />

interneurons in cortical in<strong>for</strong>mation processing. H. XU. New<br />

York Univ.<br />

10:15 312.07 Behavioral correlates of SOM and PV interneuron<br />

firing in mouse prefrontal cortex. A. KEPECS. Cold Spring<br />

Harbor Lab.<br />

10:35 312.08 Closing Remarks.<br />

MINISYMPOSIUM Walter E. Washington Convention Center<br />

313. New Insights on Diverse Neural Mechanisms<br />

Underlying Alcohol Dependence/Addiction — CME<br />

Mon. 8:30 AM - 11:00 AM — 202B<br />

Chair: C. CUI<br />

Co-Chair: A. NORONHA<br />

Alcohol dependence/addiction is mediated by complex<br />

neural mechanisms, which involve changes in a variety of<br />

neurotransmitter systems resulting in adaptive changes<br />

in neurocircuits. This minisymposium highlights recent<br />

advances in understanding alcohol addiction from the<br />

perspectives of both reward and stress systems. Speakers<br />

will present studies on metaplasticity, dendritic spines,<br />

optogenetics, and cross-talk of different signaling systems<br />

involved in alcohol dependence/addiction.<br />

8:30 313.01 Introduction.<br />

8:35 313.02 NMDA receptor metaplasticity in the VTA and drug<br />

addiction. H. MORIKAWA. Univ. of Texas at Austin.<br />

8:55 313.03 Parallel changes in synaptic function and structural<br />

in the basal ganglia after chronic ethanol and cocaine<br />

exposure. V. A. ALVAREZ. NIAAA, NIH.<br />

9:15 313.04 Optogenetic deconstruction of brain reward<br />

circuitry. G. STUBER. Univ. of North Carolina at Chapel Hill.<br />

9:35 313.05 mGluR5-mediated signaling within the extended<br />

amygdala circuit is critical <strong>for</strong> binge alcohol drinking. K. K.<br />

SZUMLINSKI. Univ. of Cali<strong>for</strong>nia at Santa Barbara.<br />

9:55 313.06 Binge-like ethanol drinking modulates neuropeptide<br />

stress systems in the extended amygdala. T. KASH. UNC<br />

Sch. of Med.<br />

10:15 313.07 Cellular mechanisms of CRF at the GABAergic<br />

synapses in the central amygdala: Role in ethanol<br />

dependence. M. ROBERTO. The Scripps Res. Inst.<br />

10:35 313.08 Closing Remarks.<br />

MINISYMPOSIUM Walter E. Washington Convention Center<br />

314. Neural Phase Coding and Spike-Field<br />

Coherence — CME<br />

Mon. 8:30 AM - 11:00 AM — 207B<br />

Chair: Z. NADASDY<br />

Co-Chair: M. E. HASSELMO<br />

Phase coding refers to the representation of in<strong>for</strong>mation<br />

by the phase of action potential firing relative to intrinsic<br />

brain rhythms. This minisymposium covers recent<br />

empirical support <strong>for</strong> phase coding in sensory cortical<br />

and prefrontal cortical neurons, as well as the emerging<br />

functions of oscillations in controlling in<strong>for</strong>mation flow in<br />

the hippocampus and in generating entorhinal grid cell<br />

properties. New results provide compelling support <strong>for</strong> the<br />

role of oscillations in neural coding.<br />

8:30 314.01 Introduction.<br />

8:35 314.02 Mechanisms <strong>for</strong> phase coding in entorhinal cortex<br />

grid cells. M. E. HASSELMO. Boston Univ.<br />

8:55 314.03 Phase coding of location and novelty by grid cells<br />

and place cells. N. BURGESS. Univ. Col. London.<br />

9:15 314.04 Different frequencies of gamma rhythms in the<br />

hippocampus. L. L. COLGIN. Univ. of Texas at Austin.<br />

2 | <strong>Society</strong> <strong>for</strong> <strong>Neuroscience</strong> • Indicated a real or perceived conflict of interest, see page 162 <strong>for</strong> details.<br />

� Indicates a high school or undergraduate student presenter.

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