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SPORTS INJURY How to tackle it EXERCISE The power of Pilates ...

SPORTS INJURY How to tackle it EXERCISE The power of Pilates ...

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OCCUPATIONAL DISEASES: Advertising FeatureN E HEALTH23<strong>The</strong> dangers <strong>of</strong> asbes<strong>to</strong>s are welldocumented, but the one-timewonder material wasn’t banneduntil relatively recently. SARAHFRENCH documents the his<strong>to</strong>ry<strong>of</strong> <strong>it</strong>s use and <strong>it</strong>s deadly legacyIT is said that in the 1st Century AD, theRoman his<strong>to</strong>rian Pliny the Elder warnedhis friends not <strong>to</strong> buy slaves from asbes<strong>to</strong>smines because “so many <strong>of</strong> themdie young”.Whether this is true or not lies w<strong>it</strong>hin theannals <strong>of</strong> his<strong>to</strong>ry, but <strong>it</strong> carries added poignancy<strong>to</strong>day when so many people have alreadydied from the effects <strong>of</strong> exposure <strong>to</strong> asbes<strong>to</strong>sand countless more are s<strong>to</strong>ring up fatal healthproblems for the future.Most recently, widower Dr Julian L<strong>it</strong>tle won£175,000 damages from the Department <strong>of</strong>Health after his wife Becky died frommesothelioma which developed decades aftershe was exposed <strong>to</strong> asbes<strong>to</strong>s as a trainee nursein the late 1960s. Mrs L<strong>it</strong>tle was one <strong>of</strong> thousands<strong>of</strong> people <strong>to</strong> have lived w<strong>it</strong>h a killer intheir midst.<strong>The</strong> use <strong>of</strong> asbes<strong>to</strong>s in the UK is relativelynew, yet there is ancient evidence that suggests<strong>it</strong>s many benef<strong>it</strong>s were recognised in Finlandas far back as 3,000 years BC.<strong>The</strong> term asbes<strong>to</strong>s applies <strong>to</strong> a group <strong>of</strong> naturallyoccurring, fibrous silicates – chrysotile(wh<strong>it</strong>e asbes<strong>to</strong>s), crocidol<strong>it</strong>e (blue asbes<strong>to</strong>s)and gruner<strong>it</strong>e or amos<strong>it</strong>e (brown asbes<strong>to</strong>s).Strong yet flexible, resistant <strong>to</strong> chemicals,non-conductive, w<strong>it</strong>h great thermal andacoustic qual<strong>it</strong>ies and fire resistant, asbes<strong>to</strong>swas given <strong>it</strong>s name by the Ancient Greeks becausethe word means “inextinguishable”.It’s l<strong>it</strong>tle wonder that asbes<strong>to</strong>s, processedin<strong>to</strong> sheets, blocks and powder, became sucha widely used material following <strong>it</strong>s introduction<strong>to</strong> the UK in 1857.Industry quickly realised <strong>it</strong>s properties andput <strong>it</strong> <strong>to</strong> use in floor and ro<strong>of</strong> tiles, insulation,sealants, cement pipes, turbines, kilns, fire retardantclothing and even in Second WorldWar gas masks.Shipbuilders used <strong>it</strong> as fire-pro<strong>of</strong> laggingand <strong>to</strong> insulate hot water pipes; railwaymensprayed <strong>it</strong> in<strong>to</strong> the cav<strong>it</strong>y between the innerand outer shell <strong>of</strong> coaches; construction firmsused <strong>it</strong> in schools and other public buildingsas insulation and sound absorption; mechanicshandled <strong>it</strong> in vehicle brakeshoes and clutchpads.BUT by the early part <strong>of</strong> last centuryfears were already being raised aboutthe dangers <strong>of</strong> asbes<strong>to</strong>s.In 1918 the Prudential in New York refused<strong>to</strong> sell life insurance <strong>to</strong> people working in asbes<strong>to</strong>smanufacturing. By the 1930s and 1940sthere was widespread concern about the risks<strong>of</strong> cancer due <strong>to</strong> exposure <strong>to</strong> asbes<strong>to</strong>s yet theuse <strong>of</strong> protective equipment remained inadequate.Its continued use bizarrely included thebroomstick ridden by the Wicked W<strong>it</strong>ch <strong>of</strong> theWest in the 1939 film <strong>The</strong> Wizard <strong>of</strong> Oz.By 1960, asbes<strong>to</strong>s was also related <strong>to</strong>mesothelioma.Nonetheless, as the post-war building boomcontinued, the use <strong>of</strong> asbes<strong>to</strong>s materials continued<strong>to</strong> grow, finally reaching <strong>it</strong>s peak in themid-1970s.Ten years later the Asbes<strong>to</strong>s Licensing Regulationswere established <strong>to</strong> control the mosthazardous asbes<strong>to</strong>s removal operations.In 1987, further tightening <strong>of</strong> the rules camein w<strong>it</strong>h the Control <strong>of</strong> Asbes<strong>to</strong>s at Work Regulations.Meanwhile, in America asbes<strong>to</strong>s wasused as insulation for the solid fuel boosters<strong>of</strong> the Space Shuttle.By 1992, the import, supply and use <strong>of</strong> themore carcinogenic brown and blue asbes<strong>to</strong>swas banned, but the import <strong>of</strong> wh<strong>it</strong>e asbes<strong>to</strong>swasn’t s<strong>to</strong>pped until November 1999.It was <strong>to</strong>o late for many workers in theNorth-East. <strong>The</strong> Tees and Tyne ports anddockyards were breeding grounds formesothelioma. Geographical tracking <strong>of</strong>deaths also highlights Yorkshire as sufferingVictims: Val Stiles, who breathed in the fibres her aunt Edna, centre, brought home.Above right: Becky L<strong>it</strong>tle and, right, her widowed husband Dr Julian L<strong>it</strong>tleAsbes<strong>to</strong>sthe killer inour midstAsbes<strong>to</strong>s fibres under the microscopefor <strong>it</strong>s success in railway engineering and Sunderlandfor <strong>it</strong>s fac<strong>to</strong>ries that used raw asbes<strong>to</strong>sin production.While deaths due <strong>to</strong> asbes<strong>to</strong>s are muchlower among females, they are significantlyhigher for women in the North-East than inother parts <strong>of</strong> the country.Fatal disease is not confined <strong>to</strong> those whoworked w<strong>it</strong>h asbes<strong>to</strong>s.<strong>The</strong> widower <strong>of</strong> Val Stiles, formerly <strong>of</strong> Darling<strong>to</strong>n,received £95,000 in compensation in2002 after his wife died from mesothelioma,aged 52. Val never worked w<strong>it</strong>h asbes<strong>to</strong>s. Infact, she was just a schoolgirl when shebreathed in the fibres that would cause herearly death. Her aunt Edna Dean worked atthe Darling<strong>to</strong>n Chemical and Insulation Companyand would <strong>of</strong>ten collect Val from schoolcovered like “a snowman” in the deadly wh<strong>it</strong>edust.Other victims have included a hairdresserand chemist’s shop assistant, both exposed viatheir work <strong>to</strong> asbes<strong>to</strong>s on the clothes and inthe hair <strong>of</strong> men who worked in nearby fac<strong>to</strong>ries.Nonetheless, men who worked w<strong>it</strong>h asbes<strong>to</strong>sremain the greater victims – 85 per cen<strong>to</strong>f the 1,860 deaths due <strong>to</strong> mesothelioma in2001 were men.Meanwhile, the annual number <strong>of</strong> deathsdue <strong>to</strong> asbes<strong>to</strong>s exposure in Great Br<strong>it</strong>ainrecorded by the Health and Safety Executivecontinues <strong>to</strong> rise. In 1968 (the first completeyear <strong>of</strong> data recorded) there were 153; this rose<strong>to</strong> 1,633 in 2000 and again <strong>to</strong> 1,862 in 2002.Only this summer a new group waslaunched in the North-East in readiness forthe rising numbers <strong>of</strong> asbes<strong>to</strong>s-related deathspredicted in the region in the next decade.<strong>The</strong> Tyne and Wear Asbes<strong>to</strong>s SupportGroup (TWASG) advises sufferers and theirfamilies, co-ordinates access <strong>to</strong> benef<strong>it</strong>s andcampaigns on health and safety issues <strong>to</strong> ensureemployers manage asbes<strong>to</strong>s properly.Nancy Ta<strong>it</strong>, direc<strong>to</strong>r <strong>of</strong> the char<strong>it</strong>y Occupationaland Environmental Diseases Association,claims mortal<strong>it</strong>y figures would be evenhigher if there was standardised diagnosis.She says <strong>it</strong>’s impossible <strong>to</strong> estimate howmany deaths have not been diagnosed as beingdue <strong>to</strong> asbes<strong>to</strong>s following occupational exposuresimply because <strong>of</strong> the arb<strong>it</strong>rary measuremen<strong>to</strong>f fibres found on victims’ lungs.She is delighted that in a report <strong>to</strong> the Departmentfor Work and Pensions in July theIndustrial Injuries Advisory Council recommendedthat a lack <strong>of</strong> fibres “should not beused <strong>to</strong> exclude a diagnosis <strong>of</strong> asbes<strong>to</strong>sis”.<strong>The</strong> report went on: “<strong>The</strong> occupational his<strong>to</strong>ryshould be the primary consideration in allcases.”MRS Ta<strong>it</strong>, whose Darling<strong>to</strong>n-born husbandBill died in 1968 <strong>of</strong> mesothelioma,fears a whole new generation<strong>of</strong> workers may be at risk <strong>of</strong> asbes<strong>to</strong>s relateddiseases.Those considered <strong>to</strong> be at risk includeplumbers, gas f<strong>it</strong>ters, carpenters, electricians,building and maintenance workers, and demol<strong>it</strong>ionworkers, as well as ordinary peopledoing DIY in older homes.Asbes<strong>to</strong>s removal did not develop as a specialised,licensed industry until 1980.<strong>The</strong> latency period also makes <strong>it</strong> impossible<strong>to</strong> measure the effectiveness <strong>of</strong> asbes<strong>to</strong>scontrols; <strong>it</strong> is “still <strong>to</strong>o short for the developmen<strong>to</strong>f mesothelioma amongst these workers<strong>to</strong> be measured” acknowledges an HSE report“Estimating the Future Burden”.<strong>The</strong> report goes on <strong>to</strong> say that, while regulationshave controlled the presence <strong>of</strong> asbes<strong>to</strong>s,<strong>it</strong> is “unreasonable” <strong>to</strong> assume that exposure<strong>to</strong> <strong>it</strong> in the UK is now zero.As long as asbes<strong>to</strong>s still exists and peopleare exposed <strong>to</strong> <strong>it</strong> at work, or at home, the risk<strong>to</strong> health continues.ASBESTOS-RELATED CONDITIONSMalignant mesotheliomaALTHOUGH a rare form <strong>of</strong> cancer, mesothelioma isthe disease most commonly connected w<strong>it</strong>hasbes<strong>to</strong>s because <strong>it</strong>’s almost always caused byexposure <strong>to</strong> the substance, the body’s normaldefences being unable <strong>to</strong> expel <strong>it</strong>s small, sharpfibres. It’s a malignant and aggressive tumour thatgenerally affects the thin surface membrane <strong>of</strong> thelungs (pleura) although <strong>it</strong> can also affect theabdomen (peri<strong>to</strong>neum) or the heart (pericardium). Itcan be caused by only a few months exposure <strong>to</strong>low levels <strong>of</strong> asbes<strong>to</strong>s – even one fibre can kill.Onset is usually longer than ten years from thedate <strong>of</strong> exposure (the ‘latency’ period) but deathusually occurs w<strong>it</strong>hin 12-18 months from diagnosis.<strong>The</strong>re is no cure.Symp<strong>to</strong>ms: pain in the lower back or side <strong>of</strong> thechest, a persistent cough, shortness <strong>of</strong> breath, ahoarse or husky voice, weight loss, sweating andfevers and difficulty swallowing.Asbes<strong>to</strong>sisFIBROSIS scarring <strong>of</strong> the lungs from substantialexposure <strong>to</strong> asbes<strong>to</strong>s resulting in reduced oxygenuptake <strong>to</strong> the bloodstream. Asbes<strong>to</strong>sis can take 20years or more <strong>to</strong> develop following exposure <strong>to</strong>asbes<strong>to</strong>s. It may damage the function <strong>of</strong> the lungsso much that the cond<strong>it</strong>ion progresses <strong>to</strong> respira<strong>to</strong>ryfailure.Symp<strong>to</strong>ms: increased breathlessness, especiallywhen exercising, coughing, chest pain and tightnessin the chest; sometimes nail abnormal<strong>it</strong>ies andclubbing <strong>of</strong> the fingers.Asbes<strong>to</strong>s-related lung cancerIT is <strong>of</strong>ficially recognised that asbes<strong>to</strong>s exposure cancause lung and other cancers becauseepidemiological studies <strong>of</strong> asbes<strong>to</strong>s worker deathsshow a high lung cancer rate. <strong>How</strong>ever, <strong>it</strong>’s verydifficult <strong>to</strong> say whether a specific cancer, other thanmesothelioma, was caused by asbes<strong>to</strong>s exposure.This is complicated further if the sufferer was asmoker at any time in their life.Diffuse pleural thickeningA BENIGN lung cond<strong>it</strong>ion that occurs when the lining<strong>of</strong> the lung, the pleura, hardens as a reaction <strong>to</strong>asbes<strong>to</strong>s fibres. It can develop on one or both <strong>of</strong> thelungs. In severe cases <strong>it</strong> can restrict breathing.<strong>The</strong>re is no cure and <strong>it</strong> can reduce the qual<strong>it</strong>y <strong>of</strong> lifew<strong>it</strong>h extreme cases being life threatening.

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