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50 Annals of Dentistry, University of Malaya, Vol. 16 2009individuals were shown to harbor fimA type I and IIbacteria almost equally in each category of probingpocket depth and attachment loss measurements.However, type 11detection was higher than type 1 in .both these ·measurements. Both genotypes of bacteriawere found only in pocket depths of more than 3 mm.For the GI, BI and PI categories, both genotypes of Pgwere associated with these indices. The frequency ofgenotype fimA type 11 was greater where plaqueaccumulation was greater (> 1)..Conclusions: Pg was detected in periodontallydiseased and healthy subjects. However it was morepredominant in the diseased subjects. PgfimA genotype1 was prevalent in both groups. Pg fimA genotype 11was identified in diseased subjects only. Pg wasassociated significantly with all clinical parameters,except for plaque index and missing teeth. Pg positivefimA genotypes 1 and 11had no significant relationshipto all clinical parameters.Immunolocalization of WNT-l ..in differentclinicopathologic subtypes ~fAnieloblastomaChuah Kee SengDepartment Oral Pathology, Oral Medicineand PeriodontologyBackground: Ameloblastoma is a histological benign,but 10callya,ggTessive non-encapsulated tumor arisingfrom' the odontogenic ectoderm and its derivatives.Despite it being classified as a benign odontogenictumor, the ameloblastoma shows a high tendency of. 'iecurrence and occasional.1y produces metastases.Ameloblastoma is classified into four ciinical types:unicystic, conventional or multi cystic, peripheral andmalignant. Histologically, there are two basic growthpatterns, follicular and plexiform. Wnt-l (Winglesstype protein), belongs to a 'large ~family of 19 secretedglycoprotein that act as signaling transducers thatpromotes cell proliferation and devefopment. Wntprotein signaling pathway plays an important key rolein carcinogenesis and embrYGgenesis. Previou·s.experimental data has shown over