Molecular and Cellular Biology of Plasminogen Activation
Molecular and Cellular Biology of Plasminogen Activation
Molecular and Cellular Biology of Plasminogen Activation
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Tuesday, 19 June 2007<br />
7:00–8:30 Breakfast<br />
8:30–9:55 Session 7—Proteases, Inhibitors, Receptors, <strong>and</strong> Substrates in Tissue Repair <strong>and</strong><br />
Tissue Remodelling<br />
Session Chairs: Katherine Hajjar <strong>and</strong> Dudley Strickl<strong>and</strong><br />
8:30–8:35 Introduction by Session Chairs<br />
8:35–8:55 032 • Pro-fibrinolytic Effects <strong>of</strong> Metalloproteinases during Skin Wound Healing in<br />
the Absence <strong>of</strong> <strong>Plasminogen</strong> • Lund LR*, Green KA, Almholt K, Ploug M, Bugge TH,<br />
Rømer J<br />
8:55–9:15 033 • Complementary Roles <strong>of</strong> Intracellular <strong>and</strong> Pericellular Collagen Degradation<br />
Pathways in Mesenchymal Cell Survival <strong>and</strong> Proliferation • Wagenaar-Miller RA,<br />
Engelholm LH, Gavard J, Yamada S, Gutkind JS, Behrendt N, Holmbeck K, Bugge TH*<br />
9:15–9:35 034 • Interplay between MMPs <strong>and</strong> the Endocytic Collagen Receptor, uPARAP/<br />
Endo180, in Collagen Degradation • Behrendt N*, Madsen DH, Ingvarsen S, Hillig T,<br />
Wagenaar-Miller R, Kjøller L, Gårdsvoll H, Høyer-Hansen G, Bugge TH, Engelholm LH<br />
9:35–9:55 035 • The Urokinase Receptor Ko Mice Have Reduced Keratinocytes Proliferation<br />
<strong>and</strong> Migration during Wound Healing <strong>and</strong> Are Protected in a Skin Carcinogenesis<br />
Protocol • D’Alessio S, Mazzieri, R, Gerasi L, Blasi F*<br />
9:55–10:15 Break<br />
Relevant Poster—088<br />
10:15–12:00 Session 8—Proteases, Inhibitors, Receptors, <strong>and</strong> Substrates in Inflammation <strong>and</strong><br />
Infectious Diseases<br />
Session Chairs: Toni Antalis <strong>and</strong> Vincent Ellis<br />
10:15–10:20 Introduction by Session Chairs<br />
10:20–10:40 036 • Distinct Roles <strong>of</strong> Plasmin in Staphylococcus aureus-induced Sepsis <strong>and</strong><br />
Infection Models • Guo Y*, Li J, Hagström E, Ny T<br />
10:40–11:00 037 • Proteolytic <strong>Activation</strong> <strong>of</strong> the Human Urokinase/Plasmin System by<br />
Staphylococcus aureus • Beaufort N*, Wojciechowski P, Sommerh<strong>of</strong>f CP, Schmitt M,<br />
Potempa J, Magdolen V<br />
11:00–11:20 038 • The Maintenance <strong>of</strong> High Affinity <strong>Plasminogen</strong> Binding by PAM Variants<br />
from Group A Streptococci Is Mediated by Conserved Arg <strong>and</strong> His Residues in Both<br />
the A1 <strong>and</strong> A2 Repeat Domains • Ranson M*, S<strong>and</strong>erson-Smith ML, Walker MJ, Fu Q,<br />
Castellino FJ, Prorok M<br />
11:20–11:40 039 • <strong>Plasminogen</strong> Activator Inhibitor-1 (PAI-1) Is an Inhibitor <strong>of</strong> Factor VII-<br />
Activating Protease in Patients with Acute Respiratory Distress Syndrome •<br />
Wygrecka M*, Morty RE, Markart P, Kanse SM, Andreasen PA, Wind T, Guenther A,<br />
Preissner KT<br />
6 X I t h I n t e r n a t i o n a l W o r k s h o p o n
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