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Clinical Pathways in Neuro-ophthalmology : An ... - E-Lib FK UWKS

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Is the Ptosis Steroid Induced?<br />

Topical steroids have been implicated <strong>in</strong> some cases of ptosis. Discont<strong>in</strong>uation of the<br />

steroids may reverse the ptosis <strong>in</strong> these cases.<br />

Is Aponeurotic Ptosis Present?<br />

Levator aponeurosis th<strong>in</strong>n<strong>in</strong>g and=or dehiscence may occur as a result of trauma,<br />

surgery, lid swell<strong>in</strong>g, patch<strong>in</strong>g, or, most commonly, as an age-related phenomenon<br />

(Frueh, 1996; Liu, 1993; Older, 1995). The characteristic features of aponeurotic ptosis are<br />

listed <strong>in</strong> Table 18–8.<br />

Patients with aponeurotic ptosis may have significant ptosis <strong>in</strong> downgaze more than<br />

primary position (Dryden, 1992; Wojno, 1993). In the absence of f<strong>in</strong>d<strong>in</strong>gs to suggest<br />

mechanical, neurogenic, myogenic, or neuromuscular etiologies for ptosis, no further<br />

evaluation is necessary. Superior visual field loss may occur due to ptosis and may be an<br />

<strong>in</strong>dication for surgical correction. The surgical treatment of aponeurotic ptosis is well<br />

described <strong>in</strong> the literature and is not reviewed here (Frueh, 1996; Liu, 1993; Older, 1995).<br />

Aponeurotic ptosis does not require any neuroimag<strong>in</strong>g (class III–IV, level B).<br />

<strong>An</strong> approach to the evaluation of ptosis is outl<strong>in</strong>ed <strong>in</strong> Figure 18–1.<br />

References<br />

Ptosis 417<br />

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<strong>Neuro</strong>surg Psychiatry 58:508–509.<br />

Afifi AK, Corbett JJ, Thompson HS, Wells KK. (1990). Seizure-<strong>in</strong>duced miosis and ptosis: association with<br />

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Ashizawa T, Hejtmancki JF, Liu J, Perryman MB, Epste<strong>in</strong> MF, Koch DD. (1992). Diagnostic value of ophthalmologic<br />

f<strong>in</strong>d<strong>in</strong>gs <strong>in</strong> myotonic dystrophy: comparison with risks calculated by haplotype analysis of closely<br />

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58:620–624.<br />

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lesions. <strong>An</strong>n <strong>Neuro</strong>l 40:465–468.<br />

Avisar R, Leshem Y, Savir H. (1991). Unilateral congenital ptosis due to plexiform neurofibroma, caus<strong>in</strong>g<br />

refraction error and secondary amblyopia. Metab Pediatr Syst Ophthalmol 14:62–63.<br />

Barton JJ, Kardon RH, Slagel D, Thompson HS. (1995). Bilateral central ptosis <strong>in</strong> acquired immunodeficiency<br />

syndrome (review). Can J <strong>Neuro</strong>l Sci 22:52–55.<br />

Berlit P, Rakicky J. (1992). The Miller Fisher syndrome: review of the literature. J Cl<strong>in</strong> <strong>Neuro</strong>-ophthalmol 12:57–63.<br />

Blumen SC, Nisipeanu P, Sadeh M, Asherov A, Tome F, Korczyn AD. (1993). <strong>Cl<strong>in</strong>ical</strong> features of oculopharyngeal<br />

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Boghen D. (1997). Apraxia of lid open<strong>in</strong>g: a review. <strong>Neuro</strong>logy 48:1491–1503.<br />

Brazis PW. (1997). Enhanced ptosis <strong>in</strong> Lambert-Eaton myasthenic syndrome. J <strong>Neuro</strong>-ophthalmol 17:202–203.

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