Isolated lymphoid follicles in colon - World Journal of Gastroenterology

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on the POU transcription factor Oct4. Cell 1998; 95: 379-391 21 Campbell PA, Perez-Iratxeta C, Andrade-Navarro MA, Rudnicki MA. Oct4 targets regulatory nodes to modulate stem cell function. PLoS One 2007; 2: e553 22 Gidekel S, Pizov G, Bergman Y, Pikarsky E. Oct-3/4 is a dose-dependent oncogenic fate determinant. Cancer Cell 2003; 4: 361-370 23 Hochedlinger K, Yamada Y, Beard C, Jaenisch R. Ectopic expression of Oct-4 blocks progenitor-cell differentiation and causes dysplasia in epithelial tissues. Cell 2005; 121: 465-477 24 Ben-Porath I, Thomson MW, Carey VJ, Ge R, Bell GW, Regev A, Weinberg RA. An embryonic stem cell-like gene expression signature in poorly differentiated aggressive human tumors. Nat Genet 2008; 40: 499-507 25 Zhang L, Hou Y, Ashktorab H, Gao L, Xu Y, Wu K, Zhai J, Zhang L. The impact of C-MYC gene expression on gastric cancer cell. Mol Cell Biochem 2010; 344: 125-135 26 Dang CV, Resar LM, Emison E, Kim S, Li Q, Prescott JE, Wonsey D, Zeller K. Function of the c-Myc oncogenic transcription factor. Exp Cell Res 1999; 253: 63-77 27 Nilsson JA, Cleveland JL. Myc pathways provoking cell suicide and cancer. Oncogene 2003; 22: 9007-9021 28 Milne AN, Sitarz R, Carvalho R, Carneiro F, Offerhaus GJ. Early onset gastric cancer: on the road to unraveling gastric carcinogenesis. Curr Mol Med 2007; 7: 15-28 29 Robinson K, Asawachaicharn N, Galloway DA, Grandori C. c-Myc accelerates S-Phase and requires WRN to avoid replication stress. PLoS One 2009; 4: e5951 30 Orian A, van Steensel B, Delrow J, Bussemaker HJ, Li L, Sawado T, Williams E, Loo LW, Cowley SM, Yost C, Pierce S, Edgar BA, Parkhurst SM, Eisenman RN. Genomic binding by the Drosophila Myc, Max, Mad/Mnt transcription factor WJG|www.wjgnet.com Guo Y et al . 8q24 variants and gastric cancer risk network. Genes Dev 2003; 17: 1101-1114 31 Luo J, Li YN, Wang F, Zhang WM, Geng X. S-adenosylmethionine inhibits the growth of cancer cells by reversing the hypomethylation status of c-myc and H-ras in human gastric cancer and colon cancer. Int J Biol Sci 2010; 6: 784-795 32 Pomerantz MM, Ahmadiyeh N, Jia L, Herman P, Verzi MP, Doddapaneni H, Beckwith CA, Chan JA, Hills A, Davis M, Yao K, Kehoe SM, Lenz HJ, Haiman CA, Yan C, Henderson BE, Frenkel B, Barretina J, Bass A, Tabernero J, Baselga J, Regan MM, Manak JR, Shivdasani R, Coetzee GA, Freedman ML. The 8q24 cancer risk variant rs6983267 shows longrange interaction with MYC in colorectal cancer. Nat Genet 2009; 41: 882-884 33 Haiman CA, Le Marchand L, Yamamato J, Stram DO, Sheng X, Kolonel LN, Wu AH, Reich D, Henderson BE. A common genetic risk factor for colorectal and prostate cancer. Nat Genet 2007; 39: 954-956 34 Wu X, Zeng Z, Chen B, Yu J, Xue L, Hao Y, Chen M, Sung JJ, Hu P. Association between polymorphisms in interleukin- 17A and interleukin-17F genes and risks of gastric cancer. Int J Cancer 2010; 127: 86-92 35 Kim MA, Lee HS, Yang HK, Kim WH. Clinicopathologic and protein expression differences between cardia carcinoma and noncardia carcinoma of the stomach. Cancer 2005; 103: 1439-1446 36 Lu Y, Chen J, Ding Y, Jin G, Wu J, Huang H, Deng B, Hua Z, Zhou Y, Shu Y, Liu P, Hu Z, Shen J, Xu Y, Shen H. Genetic variation of PSCA gene is associated with the risk of both diffuse- and intestinal-type gastric cancer in a Chinese population. Int J Cancer 2010; 127: 2183-2189 37 Crew KD, Neugut AI. Epidemiology of gastric cancer. World J Gastroenterol 2006; 12: 354-362 S- Editor Tian L L- Editor Ma JY E- Editor Ma WH 1765 April 7, 2011|Volume 17|Issue 13|
Online Submissions: http://www.wjgnet.com/1007-9327office wjg@wjgnet.com doi:10.3748/wjg.v17.i13.1766 EUS for choosing best endoscopic treatment of mesenchymal tumors of upper gastrointestinal tract Xin-Xin Zhou, Feng Ji, Liang Xu, Lin Li, Yi-Peng Chen, Jing-Jing Lu, Chun-Wei Wang, Wei Huang Xin-Xin Zhou, Feng Ji, Liang Xu, Lin Li, Yi-Peng Chen, Department of Gastroenterology, the First Affiliated Hospital, College of Medicine, Zhejiang University, Hangzhou 310003, Zhejiang Province, China Jing-Jing Lu, Department of Gastroenterology, Beilun Zongrui Hospital, Ningbo 315806, Zhejiang Province, China Chun-Wei Wang, Department of Gastroenterology, the Traditional Chinese Medicine Hospital of Ninghai, Ninghai 315600, Zhejiang Province, China Wei Huang, Department of Gastroenterology, Jinhua Wenrong Hospital, Jinhua 321017, Zhejiang Province, China Author contributions: Ji F and Zhou XX designed the research and wrote the paper; Ji F, Zhou XX, Xu L, Li L, Chen YP, Lu JJ, Wang CW and Huang W performed the research; Zhou XX and Xu L analyzed the data. Correspondence to: Feng Ji, MD, PhD, Department of Gastroenterology, the First Affiliated Hospital, College of Medicine, Zhejiang University, Hangzhou 310003, Zhejiang Province, China. jifeng1126@sina.com Telephone: +86-571-87236568 Fax: +86-571-87236611 Received: October 19, 2010 Revised: November 17, 2010 Accepted: November 24, 2010 Published online: April 7, 2011 Abstract AIM: To evaluate the value of endoscopic ultrasonography (EUS) in the choice of endoscopic therapy strategies for mesenchymal tumors of the upper gastrointestinal tract. METHODS: From July 2004 to September 2010, 1050 patients with upper gastrointestinal mesenchymal tumors (GIMTs) were diagnosed using EUS. Among them, 201 patients underwent different endoscopic therapies based on the deriving layers, growth patterns and lesion sizes. RESULTS: Using EUS, we found 543 leiomyomas and 507 stromal tumors. One hundred and thirty-three leiomyomas and 24 stromal tumors were treated by snare electrosection, 6 leiomyomas and 20 stromal tumors WJG|www.wjgnet.com 1766 World J Gastroenterol 2011 April 7; 17(13): 1766-1771 ISSN 1007-9327 (print) ISSN 2219-2840 (online) © 2011 Baishideng. All rights reserved. BRIEF ARTICLE were treated by endoloop, 10 stromal tumors were treated by endoscopic mucosal resection and 8 stromal tumors were treated by endoscopic submucosal dissection. Complete resection of the lesion was achieved in all cases. Of the mesenchymal tumors, 90.38% diagnosed by EUS were also identified by pathohistology. All wounds were closed up nicely and no recurrence was found in the follow-up after 2 mo. CONCLUSION: EUS is an effective means of diagnosis for upper GIMTs and is an important tool in choosing the endoscopic therapy for GIMTs, by which the lesions can be treated safely and effectively. © 2011 Baishideng. All rights reserved. Key words: Leiomyoma; Stromal tumor; Endoscopic ultrasonography; Endoscopic therapy Peer reviewer: Massimo Raimondo, Division of Gastroenterology and Hepatology, Mayo Clinic, 4500 San Pablo Road, Jacksonville, FL 32224, United States Zhou XX, Ji F, Xu L, Li L, Chen YP, Lu JJ, Wang CW, Huang W. EUS for choosing best endoscopic treatment of mesenchymal tumors of upper gastrointestinal tract. World J Gastroenterol 2011; 17(13): 1766-1771 Available from: URL: http://www.wjgnet.com/1007-9327/full/v17/i13/1766.htm DOI: http://dx.doi. org/10.3748/wjg.v17.i13.1766 INTRODUCTION Gastrointestinal mesenchymal tumors (GIMTs) originate from mesenchymal cells other than epithelial cells or lymphocytes. They are further classified as stromal tumors, leiomyomas, leiomyosarcomas, neural tumors, fibroblast tumors or liparomphalus. Clinically, mesenchymal tumors are usually incidentally discovered as subepithelial bulges during routine endoscopic examinations for unrelated conditions. The classification and management of these April 7, 2011|Volume 17|Issue 13|
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World Journal of Gastroenterology W
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Robert JL Fraser, Daw Park Jacob Ge
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Italy Donato F Altomare, Bari Piero
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Contents APPENDIX FLYLEAF EDITORS F
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Sipos F et al . Colonic isolated ly
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INTRODUCTION Over the past two deca
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Table 2 Relative incidence of sub-s
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