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THURSDAY, October 15, 2009<br />
4.45<br />
pm<br />
5.00<br />
pm<br />
5.15<br />
pm<br />
5.30<br />
pm<br />
5.45<br />
pm<br />
6.00<br />
pm<br />
6.15<br />
pm<br />
THURSDAY, October 15, 2009<br />
Sala Arancione – 4.45-7.00 pm<br />
Symposium<br />
ROLE OF PURINOCEPTOR IN ACUTE AND CHRONIC INJURIES AT<br />
THE CENTRAL NERVOUS SYSTEM<br />
Chairpersons: Abbracchio M. P. (Milan), Pedata F. (Florence)<br />
A ROLE FOR THE P2Y-LIKE RECEPTOR GPR17 IN THE<br />
DEVELOPMENT OF TISSUE DAMAGE AND IN<br />
REMODELLING/REPAIR PROCESSES: A NEW SENSOR OF DAMAGE<br />
AND A POTENTIAL PHARMACOLOGICAL TARGET IN SPINAL CORD<br />
INJURY<br />
Ceruti S. (Milan)<br />
ACTIVATION OF MICROGLIA BY AMYLOID β REQUIRES P2X7<br />
RECEPTOR EXPRESSION<br />
Ferrari D. (Ferrara)<br />
A SELECTIVE ADENOSINE A2A RECEPTOR ANTAGONIST REDUCES<br />
JNK MAPK ACTIVATION IN OLIGODENDROCYTES AFTER<br />
CEREBRAL ISCHEMIA “IN VIVO”<br />
Melani A. (Florence)<br />
THE PRO-INFLAMMATORY ACTION OF MICROGLIAL P2<br />
RECEPTORS IS ENHANCED IN SOD1 MODELS FOR AMYOTROPHIC<br />
LATERAL SCLEROSIS<br />
D’Ambrosi N. (Rome)<br />
REMODELING OF STRIATAL NMDA RECEPTORS BY CHRONIC A2A<br />
RECEPTOR ACTIVATION OR BLOCKADE IN HUNTINGTON’S<br />
DISEASE MICE<br />
Martire A. (Rome)<br />
NEUROPROTECTION BY A2A RECEPTOR INACTIVATION,: ROLE OF<br />
FOREBRAIN AND GLIAL A2A RECEPTORS IN THE MPTP MOUSE<br />
MODEL OF PARKINSON'S DISEASE<br />
Morelli M. (Cagliari)<br />
NEUROLOGICAL SYNDROMES AND PURINE DISMETABOLISMS:<br />
NEW INSIGHT<br />
Tozzi M. G. (Pisa)<br />
68<br />
6.30<br />
pm<br />
6.45<br />
pm<br />
MICROPARTICLE RELEASE FROM REACTIVE GLIAL CELLS:<br />
POSSIBLE ROLE IN THE CONTROL OF NEURONAL ACTIVITY<br />
Verderio C. (Milan)<br />
FINAL TEST<br />
68 69<br />
THURSDAY, October 15, 2009
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