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S. lycopersicum L. cv. Santa Clara x S. habrochaites f. glabratum accession BGH 6902 ♀ ♂ F 1 x P 1 BC 1 F 1 BC 2 F 1 x P 2 Selffing F 2 Studying of inheritance of resistance to late blight and fruit characterization (Abreu, 2005) F 3 F 4 F 5 Selection of resistance genotypes to late blight (Abreu, 2005) F 6 Inbreed line resistant to late blight with inferior fruit quality F 7 F 8 New selection to late blight resistance (Fiorini, 2008) Half-diallel analysis; commercial varieties with F8 Selection based on GCA and ECA for resistance and fruit quality among the genitors Half –diallel analysis; combination the resistance genes from both lines and testers groups Qualitative (ph-2, ph-3) combined with quantitative resistance genes Study the inheritance of resistance in of F 2 populations: ‘Ikram’ x 163A and ‘Heinz H7155’ x 163A Identification of SSR molecular marker linked to resistance genes and QTLs mapping Figure 1. The main procedures related to the breeding program of late blight during 2005 to 2010 at UFV. Viçosa, MG, 2010. 31
3. RESULTS AND DISCUSSIONS 3.1 The inheritance analysis of resistance to late blight After four days of the inoculation, the disease symptoms began to show up and were very slight. In the following days, heavy rain and low temperature which is considered favorable and optimal natural conditions for disease development followed and from the second evaluation, the lesion expansion of the infected plants was obvious and the symptoms were extreme. Out of the thirty five genotypes (parents and their progenies), none exhibited immune reaction. The females varieties (group I) differed in their resistance expression and susceptibility (Figure 2) while the testers (group II), all the inbred lines were closed in their expression in respect of AUDPC and the 73A recorded the highest values comparing with other testers. All of the crosses between varieties parents and inbred lines are represented in Figures 3, 4, 5, 6 and 7. However, two crosses resulted from crossing both NC 1 CELBR and NC 2 CELBR as females parent to the five inbred lines as source of pollens were found to be resistant in all the three experimental plots. In these crosses, the lesions were very small and there was less sporulation as compared with the susceptible varieties ‘New York’ and ‘Caline’, where the lesions were large with heavy sporulation. The variable behavior of these crosses may be attributed to the differences in the genetic background and 32
Page 1: AHMED YOUSSEF ABDELNABI MOHAMED ELS
Page 5 and 6: ACKNOWLEDGEMENT First of all, to Go
Page 7 and 8: BIOGRAPHY I was born in Dakahlia (E
Page 9 and 10: 2.7.1 Analysis of variance ……
Page 11 and 12: epidemia (Y50), a severidade no fin
Page 13 and 14: halfway epidemic (Y 50 ), severity
Page 15 and 16: and control measures are estimated
Page 17 and 18: dominance. Ramalho et al. (1993) me
Page 19 and 20: fruit to be smaller and green and t
Page 21 and 22: favorable weather, gray to white my
Page 23 and 24: are of the A2 mating type (Reis et
Page 25 and 26: populations throughout the world, e
Page 27 and 28: Heterosis is a biological phenomeno
Page 29 and 30: due to linkage drag. When the paren
Page 31 and 32: By calculating the proportion GCA/S
Page 33 and 34: 2008 in order to increase the amoun
Page 35 and 36: cm inter-row. The experiment was su
Page 37 and 38: spray system was adjusted to turn o
Page 39 and 40: experimental design was randomized
Page 41 and 42: 2 σ b : Component of variance of e
Page 43: Where: Y ij : The mean crossing inv
Page 47 and 48: they differed in respect of the % o
Page 49 and 50: Table 4. Analysis of variance (mean
Page 51 and 52: AUDPC (52.02). The group of these c
Page 53 and 54: crosses and the control group (Cali
Page 55 and 56: Severity (%)
Page 57 and 58: 3.1.2 Pearson’s correlations betw
Page 59 and 60: susceptibility. The genetic paramet
Page 61 and 62: value for the resistant parent 163A
Page 63 and 64: 3.3.1 Combining ability analysis of
Page 65 and 66: Table 9. Partitioning of genotypes
Page 67 and 68: Table 11. Estimation of specific co
Page 69 and 70: The effects of specific combining a
Page 71 and 72: Table 14. Genetic components combin
Page 73 and 74: for average fruit weight, fruit sha
Page 75 and 76: generation is smaller than the arit
Page 77 and 78: However, based on the results of th
Page 79 and 80: genotypes identified as resistant i
Page 81 and 82: 4. CONCLUSIONS The three commercial
Page 83 and 84: Figure 13. Plants hold numbers 12,
Page 85 and 86: Black, L.L., T.C. Wang and Y.H. Hua
Page 87 and 88: Conover, Robert A. and James M. Wal
Page 89 and 90: Fry, W.E. and S.B. Goodwin. Re-emer
Page 91 and 92: Hawksworth, D.L., P.M. Kirk, B.C. S
Page 93 and 94: kim, M. J., Mutschler, M. A.. Trans
Page 95 and 96:
Miranda Filho, J. B. Inbreeding and
Page 97 and 98:
Richards, Mathias C. and Raymond W.
Page 99 and 100:
Thabuis A, Lefebvre V, Bernard G, D
Page 101:
Zentmyer, George A. The world of Ph
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