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Grandmaster Ken MacKenzie - Taekwondo Times

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Stretch Yourself<br />

<br />

Osteoarthritis causes wearing out of a joint’s cartilage,<br />

either from chronic abuse or from a sudden<br />

trauma that unfavorably affected the joint’s mechanics.<br />

Whether from hardening of the subchondral bone* or<br />

from overloading muscles stabilizing the joint, the end<br />

result is destruction of the articular cartilage, pain and<br />

eventually loss of motion.<br />

You can have osteoarthritis and not know it. The<br />

affected joint may be fairly painless—just less stable<br />

or less mobile than it should, and muscles around it<br />

may be sore often. Or the joint may be painful too.<br />

Destruction of the joint’s cartilage can progress quite<br />

far without pain because the cartilage is not innervated<br />

(has no pain receptors). Joint tissues that are<br />

innervated, and send pain signals when irritated, are<br />

the fibrous connective tissue of the joint’s capsule, the<br />

muscles around the joint, and the bone underneath<br />

the cartilage. So, when the cartilage is worn through,<br />

the bone will hurt. Before that happens, the person<br />

may feel pain in some parts of the joint impinging on<br />

others due to poor muscular control (e.g., impingement<br />

of the shoulder joint), as well as the soreness of<br />

muscles overworked by compensating for poor joint<br />

mechanics. Eventually the person may feel tightness<br />

in the joint caused by increased volume of the joint’s<br />

fluid, which distends the joint’s capsule. Distention of<br />

the joint’s capsule causes inhibition (switching off ) of<br />

muscles controlling the joint, and that leads to their<br />

atrophy.<br />

In any case, the pain is easy to deal with—there is<br />

a multitude of painkilling pills and creams. Killing the<br />

pain alone does nothing to stop the arthritic changes<br />

in the joint, but it may permit arthritis sufferers to do<br />

exercises that slow down or stop the progress of the<br />

disease. What concerns the arthritis sufferers most is<br />

the damage to the joint’s cartilage and the<br />

resulting loss of stability and eventually<br />

mobility of the joint. Yes,<br />

at some stage of cartilage damage<br />

the joint loses stability—<br />

becomes lax—and seems<br />

more mobile (e.g., the knee<br />

may bend too much to the<br />

sides or the front). Later<br />

on though, the joint loses<br />

mobility and eventually, in<br />

the worst case, may become<br />

fused. How does this happen?<br />

While in some spots<br />

the cartilage is worn away,<br />

in some others it grows and<br />

eventually blocks the joint.<br />

This is not visible on<br />

X-rays—not until<br />

the overgrown<br />

cartilage calcifies. Before that happens, both the worn-out<br />

and overgrown cartilage can be revealed by MRI. (X-rays<br />

of arthritic joints show only altered position of bones,<br />

which indicates the amount of change in the cartilage but<br />

does not show the cartilage itself.)<br />

But back to the arthritis sufferers…Knowing that the<br />

cartilage in the affected joint or joints is worn out, most<br />

look for ways to restore it. They eat supplements, apply<br />

creams and ointments, even have medication injected into<br />

the joints. Of the supplements, glucosamine and chondroitin<br />

are shown to do no harm, but there is little proof<br />

of them helping. No cream or ointment can penetrate the<br />

joint’s capsule to bring in the building materials, so the<br />

best they can do is lower the pain and reduce inflammation.<br />

After an injection into the joint’s cavity, the cartilage<br />

may begin to grow, but not so selectively. The undamaged<br />

cartilage, growing in the “wrong places,” will grow even<br />

more—and the joint will be further blocked. This excessive,<br />

uneven growth may have striking results in the knee<br />

joints: Not only will their mobility be reduced but also<br />

the legs may bend drastically, even more than 45 degrees,<br />

either out (bow legs) or in (x-legs), and in the worst cases<br />

one leg out and one leg in. Many people fall for miraculous<br />

medicines, ancient or modern, from shamans or space<br />

labs, that promise to selectively grow the cartilage where<br />

it is damaged (and perhaps eat it away where it is not<br />

needed). There are ways of selectively stimulating growth<br />

of worn-out cartilage and removing the overgrown cartilage—but<br />

these are not simple procedures like injections.<br />

These are surgical procedures: Both the prolotherapy to<br />

stimulate growth of the cartilage and the abrading of the<br />

excessive growth require arthroscopy. They are not very<br />

effective either; after all, people still get their knees and<br />

hips replaced.<br />

So what should you do to restore function of an<br />

arthritic joint? First, stop any exercise or activity that<br />

causes pain and inflammation (pain = damage = inflammation).<br />

If an exercise causes any discomfort in the joint<br />

during or after performing it, then it is not good and has<br />

to go. Second, stop the inflammation. Inflammation damages<br />

all tissues of the joint (cartilage, ligaments, tendons)<br />

and causes atrophy of the muscles stabilizing and controlling<br />

the joint. A long-lasting inflammation can cause<br />

permanent destruction of muscles that cannot be brought<br />

back to life by any means (e.g., fatty atrophy—muscle<br />

fibers dying and being replaced by fat). Inflammation may<br />

be stopped by creams, ointments, or gels such as Voltaren,<br />

prescription anti-inflammatory drugs, and in the worst<br />

cases by cortisol injections. Whatever it takes, the inflammation<br />

has to be stopped for two reasons:<br />

* To stop the damage<br />

* To make the patient realize how it feels to not have the<br />

inflammation.<br />

74 November 2009 / taekwondotimes.com<br />

By Thomas Kurz

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