'<strong>1861</strong>.] vmxmksmia. 819composition of the stagnant urine, and its resorption, ft isevident that the decomposition must be hastened by the presence,in the bladder, or catarrhons secretions, blood, or productsof exsudation. At this stage of the disease, the urine,he it evacuated spontaneously or artificially, presents a pungent,often distinctly anmioniacal odor, ana a mucous or purulentsediment, sometimes mixed with blood. Wherever anammoniacal odor is perceivable in recently-voided urine, wemay pretty certainly diagnosticate ammomsemia.3. The carbonate of ammonia is eliminated through the?lungs and skin soon after its reception into the hlood ;this isproved by the distinct ammoniacal odor of the expired air.and other exhalations. Where this odor is not very distinct,the expired air may be tested with humid litmus-paper, orwith a stick of glass, previously immersed in muriatic acid.The chemical analysis of the hlood would, undoubtedly, furnishthe best basis for a correct diagnosis; but the patientsafflicted with ammoimemia are always in a state which doesnot admit of the depletion of blood.•i. Gastric symptoms, as already described, are always present.The evacuations in acute cases do not, at least not inthe first time, present a distinct ammoniacal odor; but this \-always well marked, when emesis and catharsis a<strong>pp</strong>ear withan aggravation of the disease. In chronic ammoniaunia theremay be neither vomiting nor diarrhoea, but rather a tendencyto constipation : the a<strong>pp</strong>etite is, however, with very rare exceptions,disturbed, and a marked aversion to meat present,such as is not observed in any other disease, even not exceptingcancer of the stomach. Other gastric symptoms, for instance,a sensation of dull pressure or burning pain in theach, accumulation of gases, bad taste, furred tongue, etc.,are not characteristic of ammonisemia. But the dry conditionof the month and fauces is important, and may often lead tothe diagnosis when other prominent symptoms are wanting.The discoloration of the face, the emaciation and collapsethe features, finally the continuous depression of spirits, whichis frequently explained as hypochondriacal, will serve toconfirm the diagnosis. The effect on the mind of the patientmay grow >trnir enough to induce the committing of suicide.In the treatment, stricture- of the urethra, which are frequentlypresent, mnst be removed, and the urine regularlyevacuated by means of the catheter. Milk diet, residence inthe country, generous exercise in the fresh air. are often sufficient,in addition to these mean-, to restore the patient.Daring recovery, a vigorous diet is required, which may bo
820 Arnmoniccraia. [<strong>October</strong>,combined with the use of some acidulous or ferruginatedmineral water.Strictures of the urethra have often been overlooked ; Ithink they alone have given rise to the haemorrhoids of thebladder, that we used to hear so much about in times notlong gone. Of equal importance with the strictures arediseases of the prostate in connection with ammonisemia.Amongst them are, atrophy in consequence of precedinginflammation, general or partial hypertrophy and developmentof an accessory tumor, pressing on the urethra or theneck of the bladder. They impede, more or less, theevacuation of the urine, cause torpor of the bladder, hypertrophyof its walls, diseases of the ureters and kidneys,and consequently ammoniamiia, which sometimes a<strong>pp</strong>earssoon, but often only after a long time. The difficulties ofa successful surgical treatment, in such cases, render theprognosis rather unfavorable. Hypertrophy of the prostatewill often yield to a strong ointment of iodide of potassiuma<strong>pp</strong>lied to the perineum, and combined with the internaluse of acidulated waters. With the cautious use of thecatheter, and a decoction of secale cornutum, exhibited internally,the alread} r developed torpor of the bladder, andother symptoms, even of chronic ammonisemia, may be relieved.A long practiced intentional retention of urine is apt toend in the highest development of ammonieemia, withoutany difficulty in, or around the urethra. The greater thechange in the textures, following this cause, the smaller isthe prospect of recovery.That stones in the bladder result fatally through ammoniaemia,has been long known to surgeons; the disease Idesignate by this name is well described, with all its symptomsin their books. It certainly plays a very importantpart in its connection with urinary concretions. Withoutan operation death is certain to follow, while success with asurgical proceeding depends completely upon the presenceand extent of ammoniaBmia. Under the influence of bloodpoisoned by ammonia, diphtheritic processes and exulcerationsin the artificial wounds are unavoidable.Cancer of the bladder is always, in the end, accompaniedby ammonisemia. Medullary carcinoma of the inner surfaceof the bladder induces 'hemorrhage, and thereby decompositionof the urine.Mechanical obstruction of the ureters, or destructive dis-
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