14.02.2018 Views

2017 HCHB_digital

You also want an ePaper? Increase the reach of your titles

YUMPU automatically turns print PDFs into web optimized ePapers that Google loves.

CONTINUING OTC EDUCATION<br />

of Maori or Pacific ancestry. People who drink soft drinks sweetened with high<br />

fructose corn syrup (HFCS) or consume diets rich in fruit or fruit juice are also at<br />

increased risk. In addition, certain medical conditions (eg, type 2 diabetes, kidney<br />

problems, elevated blood pressure), some medicines (eg, diuretics, cytotoxics),<br />

large intakes of purine-rich foods or alcohol (particularly beer) and joint injury<br />

also contribute to the risk.<br />

Treating a gout attack<br />

Non-steroidal anti-inflammatory drugs (NSAIDs) are recommended first line<br />

(unless contraindicated) as pain relief for acute attacks of gout (see Treatment<br />

Options previous page). Corticosteroids or low dose colchicine can be used as<br />

an alternative to NSAIDs.<br />

Any medicine used to treat acute attacks should be taken at the first sign of<br />

an attack at an effective dose. In most people, pain and inflammation caused by<br />

the gout can be controlled within 12–24 hours and treatment discontinued after<br />

a few days.<br />

Drugs used for gout attacks have no effect on reducing uric acid levels and all<br />

customers with symptoms suggestive of gout should be referred to a doctor for<br />

consideration of allopurinol or probenecid. Early prevention of gout, before the<br />

onset of tophi, erosive disease and renal impairment, is vital.<br />

Preventing gout<br />

Most experts agree preventive therapies should aim to reduce serum uric<br />

acid concentration to below 0.36mmol/L (some experts aim for as low as<br />

Hooray! Your file is uploaded and ready to be published.

Saved successfully!

Ooh no, something went wrong!