Espozione Professionale a Metalli Pesanti e Sclerosi ... - Anmil

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Tesi Sperimentale di SpecializzazioneEsposizione Professionale a Metalli Pesanti e Sclerosi Laterale Amiotrofica66[28] Manton WI, Cook JD. High accuracy (stable isotope dilution)measurements of lead in serum and cerebrospinal fluids. Br J IndMed 1984;41:313-319[29] Oliver T. Lead Poisoning: from the industrial, medical and socialpoints of view. London: Lewis, 1914, pp 1-294.[30] Goyer RA, Rhyne BC. Pathological effects of lead. Int Rev Exp Pathol1973;12:1-77[31] Lindgren KN, Masten VL, and Ford DP. Relation of cumulativeexposure to inorganic and neuropsychological test performance.Occup Environ Med 1996;53:472-477[32] Machintosh MJ, Meredith PA, Moore MR, et al. Action of Lead onneurotransmission in rats. Xenobiotica 1989;19:101-113[33] Cooper JR, Bloom FE, and Roth RH. The Biochemical Basis ofNeuropharmacology. New York, NY, 1982: Oxford University Press[34] Barbeito AG, Guelfi N, Varga MR, Pehar M, Beckam J, Barbeito L, etal. Chronic low-level lead exposure increases serviva of G93A SOD-1transgenic mice. Amyotrophic Lateral Sclerosis: beyond the motorneuron; 2005.[35] Kamel F, Umbach DM, Hu H, Munsat TL, Shefner JM, Taylor JA, etal. Lead exposure as a risk factor for amyotrophic lateral sclerosis.Neurodegener Dis 2005;2:195-201[36] Kamel F, Umbach DM, Stallone L, Richards M, Hu H, Sandler DP.Association of lead exposure with survival in amyotrophic lateralsclerosis. Environ Health Perspect 2008;116:943-947
Tesi Sperimentale di SpecializzazioneEsposizione Professionale a Metalli Pesanti e Sclerosi Laterale Amiotrofica67[37] Locktich G. Prespectives on lead toxicity. Clin Biochem 1993;26:371-381[38] Markovac J, Goldstein GW. Lead Activates protein kinase C inimmature rat brain microvessels. Toxicol Appl Farmacol 1998;96:14-23[39] Lasley SM. Regulation of dopaminergic activity, but not tyrosinehydroxylase, is dimished after chronic inorganic lead exposure.Neurotoxicology 1992;13:625-635[40] Apostoli P, Porru S, Alessi L. Are current biomarkers suitable for theassessment of manganese exposure in individual workers? Am J IndMed 2000;37:283-290[41] Greger JL. Dietary and other sources of aluminium intake.Aluminium in Biology and Medicine. New York: Wiley, 1992 pp. 26-49[42] Perl DP, Good PF. Uptake of aluminium intro cenral nervous systemalong nasal olfactory pathways. Lancet 1987;1:1028-1029[43] Jouhanneau P, Raisbeck GM, Viou F, et al. Gastrointestinalabsorption, tissue retention and urinary excretion of dietary levels ofaluminium in rats as determined by using 26 Al. Clin Chem1997;43(pt1):1023-1028[44] Flaten TP, Wakayama I, Sturm MJ, et al. Natural models ofaluminium toxicity in fish from acid-rain lakes, 1993, Nicolini M,Zatta P.F., Corain B. (eds): Alzheimer’s[45] Exley C, Brugess E, Day JP, et al.: Aluminium toxicokinetics. J ToxicolEnviron Health 1996;48:569-584
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