Cong_Nystagmus 2009.pdf - The Private Eye Clinic

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CN c.f. LMLN : L beat on L gaze, R beat on R gaze [Alexander’s Law] • CN: N L on L of null zone, N R on R of null zone • LMLN : • LE is fixing & is in LG: BE have N L • LATENT COMPONENT: Both can have this – ? different mechanisms • RE is fixing & is in RG: BE have N R KOWAL MEACO 2009 18
‘I AM WORSE THAN I USED TO BE’ • LMLN: • Has lost binocularity and LN MLN • CN: • new neurological lesion has caused deterioration, or • Stress – common as a temporary mechanism, rarely permanent KOWAL MEACO 2009 19
Page 1 and 2: CONGENITAL NYSTAGMUS WHEN TO RECORD
Page 3 and 4: Clinical Features CN 1. L beat on L
Page 5 and 6: Why does CN occur? 2 • Genetic re
Page 7 and 8: Whatever caused it, cNwill then deg
Page 9 and 10: Principles of treatment of CN •1.
Page 11 and 12: 2. Improve 2ary effects such as fac
Page 13 and 14: Effects / Treatment of LMLN • Red
Page 15 and 16: Clinical Features CN 1. L beat in L
Page 17: CN c.f. LMLN • STRABISMUS : LMLN
Page 21 and 22: Clinical correlations : Dr Elaine W
Page 23 and 24: Clinical and EMR Correlation KOWAL
Page 25 and 26: Predictive Values of Clinical Featu
Page 27 and 28: Predictive Values of Clinical Featu
Page 29 and 30: Does everyone with N need to be rec
Page 31 and 32: SURGERY IN cN : CN • Eccentric nu
Page 33 and 34: Eccentric null ≤ 20° L face turn
Page 35 and 36: SURGERY IN CN : Convergence null
Page 37 and 38: SURGERY IN cN : PAN • Look for co
Page 39 and 40: THE RECENT PAST AND THE IMMINENT FU

Cong_Nystagmus 2009.pdf - The Private Eye Clinic

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