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Phonological Conditions on Affixation

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ph<strong>on</strong>ological processes but also lack some of those properties. Fortunately, the distincti<strong>on</strong><br />

between suppletive and n<strong>on</strong>-suppletive allomorphy is not crucial to the argument made in<br />

this dissertati<strong>on</strong>. Since the P >> M mechanism in effect extends a ph<strong>on</strong>ological model to<br />

handle some morphological processes, we expect that the mechanism should handle the<br />

purely ph<strong>on</strong>ological (i.e., n<strong>on</strong>-suppletive) examples well and that the problematic cases<br />

(if any) should be the suppletive <strong>on</strong>es. This means that if anything, if we admit any cases<br />

of rule-derived allomorphy into our discussi<strong>on</strong> of suppletive allomorphy, we will err <strong>on</strong><br />

the side of including too many examples that favor the P >> M approach. To offset any<br />

such bias, in making generalizati<strong>on</strong>s about PCSA I will focus <strong>on</strong> the clearest cases of<br />

suppletive allomorphy and not rely too heavily <strong>on</strong> examples that fall into the gray area.<br />

2.1.2 Examples<br />

2.1.2.1 Assimilati<strong>on</strong>/harm<strong>on</strong>y<br />

Given that the P >> M mechanism accounts for suppletive allomorphy using the<br />

same ph<strong>on</strong>ological c<strong>on</strong>straints that drive purely ph<strong>on</strong>ological processes, this model<br />

predicts that every ph<strong>on</strong>ological c<strong>on</strong>straint can show effects in morphology. That is,<br />

c<strong>on</strong>straints that comm<strong>on</strong>ly drive ph<strong>on</strong>ological processes in the world’s languages should<br />

also c<strong>on</strong>diti<strong>on</strong> suppletive allomorph selecti<strong>on</strong>. Assimilati<strong>on</strong> is a very comm<strong>on</strong> type of<br />

ph<strong>on</strong>ological process with roots in coarticulati<strong>on</strong>, and the P >> M model therefore leads<br />

us to expect assimilati<strong>on</strong> to c<strong>on</strong>diti<strong>on</strong> suppletive allomorphy. Several c<strong>on</strong>straints have<br />

been proposed to drive harm<strong>on</strong>y and other assimilati<strong>on</strong> of various types, including<br />

AGREE, SPREAD, and ALIGN (see Pulleyblank 2002 for a summary of approaches to<br />

31

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