Pericarditi

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Circumstances of diagnosis Systematic Acute Chronic Tamponade Echocardiography Pericardial Obvious cause Non obvious cause Volume>200cc or CT scanner Diagnosis compatible Levy PY, Lepidi H, Habib G, Collard F, Raoult D: Etiological diagnosis of pericardial effusion. Future Microbiology. STOP 2006;1:229-39 Systematic non invasive exploration: Serological tests, TSH ACAN, Viral cultures. No diagnosis or inefficacy of treatment Tumors markers Research of primitive cancer Pericardiocentesis - Standard Culture - Viral culture - Mycobacteria - Molecular biology (16SRNA, 18SRNA, Enterovirus, Herpes consensus)
Treatment?Symptomatic treatment with aspirin (up to 650 mg every 4 hours) or nonsteroidal anti-inflammatory drugs are the mainstay treatment. Ibuprofen is generally preferred for its rare side-effects, favourable impact on the coronary flow, and the large dose range 300-800mg every 6-8 hours. It can be continued for days or weeks. Colchicine (0.5mgbid) added or as monotherapy also appears to be effective for the initial attack and the prevention of recurrences. Systemic corticosteroid therapy should be restricted to connective tissues, uremic pericarditis. It is contraindicated in case of infectious pericarditis except in patients with secondary tuberculous pericarditis as an adjunct to tuberculostatic treatment. Quelle est l’évolution du syndrome? Evolution depends from etiology: acute benign pericarditis generally resolves with 3 weeks. Tuberculosis and radiation therapy tends to be responsible of constrictive pericarditis. Possible complications? Possible complications of pericarditis include cardiac tamponade, in which the accumulation of fluids can cause severe compression, recurrent pericarditis with a symptom free interval, and pericardial constriction, in which an adherent pericardium restricts the diastolic filling of the heart. Chronic (>3
Page 1 and 2: PERICARDITIS Summaries -Introductio
Page 3 and 4: 3/ Levy PY, Thuny F, Habib G, Bonne
Page 5 and 6: Risk factors? The diminution of ste
Page 7 and 8: Etiology? Etiology Bacterial , fung
Page 9 and 10: Stade IV: Reversion of T wave to no
Page 11: 1/ Constitution of the kit (specifi
Page 15 and 16: The calcification is responsible of
Page 17 and 18: 2.Levy PY, Corey R, Berger P, et al
Page 19 and 20: Consultations : Sur rendez vous Ven
Page 21: Tuberculose Dysthyroïdie Cancer

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