Pediatric Clinics of North America - CIPERJ

More documents

Recommendations

Info

420 BLANCHETTE & BOLTON-MAGGS [103] Bussel JB, Cheng G, Saleh MN, et al. Eltrombopag for the treatment of chronic idiopathic thrombocytopenic purpura. N Engl J Med 2007;357:2237–47. [104] Bolton-Maggs PHB. Management of immune thrombocytopenic purpura. Paediatr Child Health 2007;17:305–10. [105] Buchanan GR, Adix L. Outcome measures and treatment endpoints other than platelet count in childhood idiopathic thrombocytopenic purpura. Semin Thromb Hemost 2001; 27:277–85. [106] Barnard D, Woloski M, Feeny D, et al. Development of disease-specific health-related quality of life instruments for children with immune thrombocytopenic purpura and their parents. J Pediatr Hematol Oncol 2003;25:56–62. [107] von Mackensen S, Nilsson C, Jankovic M, et al. Development of a disease-specific quality of life questionnaire for children and adolescents with idiopathic thrombocytopenic purpura (ITP-QoL). Pediatr Blood Cancer 2006;47:688–91. [108] Klaassen RJ, Blanchette VS, Barnard D, et al. Validity, reliability and responsiveness of a new measure of health-related quality of life in children with immune thrombocytopenic purpura: the Kids’ ITP Tools. J Pediatr 2007;150:510–5. [109] Buchanan GR, Adix L. Grading of hemorrhage in children with idiopathic thrombocytopenic purpura. J Pediatr 2002;141:683–8. [110] Page KL, Psaila B, Provan D, et al. The immune thrombocytopenic purpura (ITP) bleeding score: assessment of bleeding in patients with ITP. Br J Haematol 2007;138:245–8. [111] O’Brien SH, Ritchey AK, Smith KJ. A cost-utility analysis of treatment for acute childhood idiopathic thrombocytopenic purpura (ITP). Pediatr Blood Cancer 2007;48:173–80. [112] Cines DB, Bussel JB. How I treat idiopathic thrombocytopenic purpura (ITP). Blood 2005; 106:2244–51.
Pediatr Clin N Am 55 (2008) 421–445 Blood Component Therapy Ross Fasano, MD a,c , Naomi L.C. Luban, MD b,c, * a Children’s National Medical Center, Department of Hematology/Oncology, 111 Michigan Avenue NW, Washington, DC 20010, USA b Children’s National Medical Center, Department of Laboratory Medicine, 111 Michigan Avenue, NW, Washington, DC 20010, USA c The George Washington University Medical Center, Washington, DC, USA Blood component transfusion is an integral part of the treatment of many infants and children cared for by general pediatricians, surgeons, intensivists, and hematologists/oncologists. Technologic advances in blood collection, separation, anticoagulation, and preservation have resulted in component preparation of red blood cells (RBCs), platelets, white blood cells (WBCs), and plasma, which are superior to whole blood (WB) used in the past. Recent advances in donor selection, infectious disease testing, use of leukoreduction filters, and gamma irradiation also deem today’s products safer than the past. Physicians prescribing blood components not only should have a basic understanding of the indications (and contraindications) for their use but also should be cognizant of the methods of preparation, the proper storage conditions, and the requirements for further modification of blood products to prevent potential adverse effects. Blood component preparation and modification Blood components are prepared from blood collected by WB or apheresis donations. Transfusion of WB is uncommon in modern medicine. Uses for WB or reconstituted WB units include blood priming for extracorporeal circuits (ie, therapeutic apheresis in small patients, cardiovascular bypass, extracorporeal membrane oxygenation, and continuous hemoperfusion), neonatal exchange transfusions, and patients who have active bleeding and massive volume loss. Given that platelet function is poor after 24 hours * Corresponding author. Children’s National Medical Center, Department of Laboratory Medicine, 111 Michigan Avenue, NW, Washington, DC 20010. E-mail address: nluban@cnmc.org (N.L.C. Luban). 0031-3955/08/$ - see front matter Ó 2008 Elsevier Inc. All rights reserved. doi:10.1016/j.pcl.2008.01.006 pediatric.theclinics.com
Page 2 and 3:
Pediatr Clin N Am 55 (2008) xiii-xi
Page 4 and 5:
Pediatr Clin N Am 55 (2008) 287-304
Page 6 and 7:
DECISION ANALYSIS IN PEDIATRIC HEMA
Page 8 and 9:
Page 10 and 11:
Page 12 and 13:
Page 14 and 15:
Page 16 and 17:
Nietert et al [30] Treatment of sic
Page 18 and 19:
Page 20 and 21:
Page 22 and 23:
Page 24 and 25:
VENOUS THROMBOEMBOLISM IN CHILDREN
Page 26 and 27:
Page 28 and 29:
Page 30 and 31:
Page 32 and 33:
Page 34 and 35:
Page 36 and 37:
Page 38 and 39:
Page 40 and 41:
Page 42 and 43:
PEDIATRIC ARTERIAL ISCHEMIC STROKE
Page 44 and 45:
Page 46 and 47:
Page 48 and 49:
Page 50 and 51:
Page 52 and 53:
Page 54 and 55:
Page 56 and 57:
Page 58 and 59:
CARE OF PATIENTS WITH VASCULAR ANOM
Page 60 and 61:
Page 62 and 63:
Page 64 and 65:
Page 66 and 67:
Page 68 and 69:
Page 70 and 71:
Page 72 and 73:
Page 74 and 75:
358 RODRIGUEZ & HOOTS Fig. 1. X-lin
Page 76 and 77:
Table 1 Clotting factor concentrate
Page 78 and 79:
Table 1 (continued ) Factor VIII (o
Page 80 and 81:
364 RODRIGUEZ & HOOTS was administe
Page 82 and 83:
366 RODRIGUEZ & HOOTS Antifibrinoly
Page 84 and 85:
368 RODRIGUEZ & HOOTS infections an
Page 86 and 87: 370 RODRIGUEZ & HOOTS Fig. 3. Schem
Page 88 and 89: 372 RODRIGUEZ & HOOTS protein is no
Page 90 and 91: 374 RODRIGUEZ & HOOTS [2] Berntorp
Page 92 and 93: 376 RODRIGUEZ & HOOTS [42] Carcao M
Page 94 and 95: 378 ROBERTSON et al von Willebrand
Page 96 and 97: 380 ROBERTSON et al Fig. 2. A propo
Page 98 and 99: 382 ROBERTSON et al a heterogenous
Page 100 and 101: 384 ROBERTSON et al of considering
Page 102 and 103: 386 ROBERTSON et al Type 2M Type 2M
Page 104 and 105: 388 ROBERTSON et al Desmopressin is
Page 106 and 107: 390 ROBERTSON et al References [1]
Page 108 and 109: 392 ROBERTSON et al [46] Nesbitt IM
Page 110 and 111: 394 BLANCHETTE & BOLTON-MAGGS A key
Page 112 and 113: 396 BLANCHETTE & BOLTON-MAGGS recep
Page 114 and 115: 398 BLANCHETTE & BOLTON-MAGGS and p
Page 116 and 117: 400 BLANCHETTE & BOLTON-MAGGS Fig.
Page 118 and 119: 402 BLANCHETTE & BOLTON-MAGGS in di
Page 120 and 121: 404 BLANCHETTE & BOLTON-MAGGS the s
Page 122 and 123: 406 BLANCHETTE & BOLTON-MAGGS findi
Page 124 and 125: 408 BLANCHETTE & BOLTON-MAGGS as a
Page 126 and 127: 410 BLANCHETTE & BOLTON-MAGGS splen
Page 128 and 129: 412 BLANCHETTE & BOLTON-MAGGS with
Page 130 and 131: 414 BLANCHETTE & BOLTON-MAGGS react
Page 132 and 133: 416 BLANCHETTE & BOLTON-MAGGS [16]
Page 134 and 135: 418 BLANCHETTE & BOLTON-MAGGS [57]
Page 138 and 139: 422 FASANO & LUBAN of storage and t
Page 140 and 141: 424 FASANO & LUBAN Leukocyte reduct
Page 142 and 143: 426 FASANO & LUBAN RBCs is decrease
Page 144 and 145: Table 1 Blood component characteris
Page 146 and 147: 430 FASANO & LUBAN Alloimmunization
Page 148 and 149: 432 FASANO & LUBAN of individual un
Page 150 and 151: 434 FASANO & LUBAN patients have an
Page 152 and 153: 436 FASANO & LUBAN adults; therefor
Page 154 and 155: 438 FASANO & LUBAN [21]. Meta-analy
Page 156 and 157: 440 FASANO & LUBAN Pretransfusion m
Page 158 and 159: 442 FASANO & LUBAN [55,56]. This is
Page 160 and 161: 444 FASANO & LUBAN [20] Roseff SD,
Page 162 and 163: Pediatr Clin N Am 55 (2008) 447-460
Page 164 and 165: THALASSEMIA UPDATE 449 hypochromia
Page 166 and 167: THALASSEMIA UPDATE 451 Fig. 1. Chan
Page 168 and 169: THALASSEMIA UPDATE 453 delivery of
Page 170 and 171: THALASSEMIA UPDATE 455 thromboses i
Page 172 and 173: THALASSEMIA UPDATE 457 On the basis
Page 174 and 175: THALASSEMIA UPDATE 459 [31] Kan YW,
Page 176 and 177: Pediatr Clin N Am 55 (2008) 461-482
Page 178 and 179: ORAL IRON CHELATORS 463 large iron-
Page 180 and 181: ORAL IRON CHELATORS 465 of 50 child
Page 182 and 183: ORAL IRON CHELATORS 467 Table 2 Com
Page 184 and 185: ORAL IRON CHELATORS 469 use in Nort
Page 186 and 187:
ORAL IRON CHELATORS 471 needed to d
Page 188 and 189:
ORAL IRON CHELATORS 473 in liver fi
Page 190 and 191:
ORAL IRON CHELATORS 475 in the lowe
Page 192 and 193:
ORAL IRON CHELATORS 477 Other oral
Page 194 and 195:
ORAL IRON CHELATORS 479 [12] Borgna
Page 196 and 197:
ORAL IRON CHELATORS 481 [55] Wonke
Page 198 and 199:
Page 200 and 201:
HYDROXYUREA FOR CHILDREN WITH SICKL
Page 202 and 203:
Page 204 and 205:
Page 206 and 207:
Page 208 and 209:
Fig. 1. Guideline for initiating, m
Page 210 and 211:
Page 212 and 213:
Page 214 and 215:
Page 216 and 217:
Page 218 and 219:
504 TRACY & RICE congenital spheroc
Page 220 and 221:
506 TRACY & RICE and antibodies aga
Page 222 and 223:
508 TRACY & RICE limited splenic vo
Page 224 and 225:
510 TRACY & RICE constitutes the pi
Page 226 and 227:
512 TRACY & RICE German experience
Page 228 and 229:
514 TRACY & RICE Table 1 Effect of
Page 230 and 231:
516 TRACY & RICE decreased bilirubi
Page 232 and 233:
518 TRACY & RICE [13] Bader-Meunier
show all

Pediatric Clinics of North America - CIPERJ

You also want an ePaper? Increase the reach of your titles

Delete template?

Save as template?