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IMATINIB AND BCR - ABL TK<br />
* BCR - ABL exemplifies fusion of receptor and nonreceptor TKs<br />
to various N - term<strong>in</strong>al “partner prote<strong>in</strong>s” as consequence of<br />
chromosomal translocations and deletions [<strong>in</strong> CML, from t(9;22)<br />
chr translocation → Philadelphia chr (→ BCR - ABL)];