IAH AC Gastrointestinal Disease

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Peptic ulcer disease • Duodenal ulceration • Related to hyperacidity • Helicobacter pylori infection often present • Both can be caused by NSAIDs like aspirin, ibuprofen, diclofenac (mostly Cox-1-inhibitors, more than Cox-2) © <strong>IAH</strong> 2007 44 Duodenal ulceration on the other hand, is almost always associated with a high acid secretion and also with a high incidence of Helicobacter Pylori. Both the gastric as well as the duodenal ulceration can contributed to by the use of NSAIDS. These, as we mentioned above regarding the leaky gut, can in fact directly damage the mucosa, but also interfere with the metabolism of the mucosal cells, and therefore can cause tissue breakdown, and subsequently ulceration. 44
Treatment • Often an adjuvant treatment • Mild to moderate forms can be treated with antihomotoxics alone • (Regular follow ups) • Helps to heal ulcer quicker and relapses are longer apart • H. pylori can also be treated without antibiotics • Chronic association connected with cancer © <strong>IAH</strong> 2007 45 Care should be taken not to treat patients with progressive disease with only biological therapy. Often a combination therapy can go a long way to promote tissue healing and to keep the patient in remission for longer periods of time, or even eradicate the disease earlier. Mild to moderate forms deserve a trial of antihomotoxic therapy alone, but there should be regular follow ups in these patients with endoscopy, especially in gastric ulceration the disease progression towards cancer should be a constant concern. With duodenal ulceration other complications, such as perforation, etc must also be kept in mind. Even H. Pylori can be treated with biological therapy alone, as was seen on a study by Karl Heinz Ricken. 45
Page 1 and 2: Gastrointestinal Disease IAH AC Gas
Page 3 and 4: The intestinal mucosa • Has the p
Page 5 and 6: The mucosal barrier • From the ga
Page 7 and 8: The function of the mucosal lining
Page 9 and 10: Breast is best!! Photos: WHO Ethiop
Page 11 and 12: Environment © IAH 2007 11 The gut
Page 13 and 14: Mucosal distress (2) • Examples,
Page 15 and 16: Vicious cycles of changed permeabil
Page 17 and 18: Lactose intolerance, not immune-reg
Page 19 and 20: Food allergies Positive skin test P
Page 21 and 22: II. Liver overload • All the bloo
Page 23 and 24: Gut milieu luminal contents and pH
Page 25 and 26: IV. Low grade pancreatitis liver To
Page 27 and 28: Homotoxicology approach © IAH 2007
Page 29 and 30: Oral tolerance and immunological by
Page 31 and 32: Oral tolerance and immunological by
Page 33 and 34: Systemic Gastroenterology Antihomot
Page 35 and 36: Aphthous ulceration • Painful ulc
Page 37 and 38: Gastroesophageal reflux disease (GE
Page 39 and 40: Treatment of gastroesophageal reflu
Page 41 and 42: The composition of Gastricumeel 4.
Page 43: Peptic ulcer disease • Gastric ul
Page 47 and 48: Peptic ulceration Symptomatic Basic
Page 49 and 50: Cholecystitis and gall stones • I
Page 51 and 52: Spascupreel tablets - injection sol
Page 53 and 54: Viral hepatitis • Hepatitis virus
Page 55 and 56: Hepatitis Symptomatic Basic medicat
Page 57 and 58: Functional disturbances of motility
Page 59 and 60: Diarrhea • Increase secretion of
Page 61 and 62: Actions of Diarrheel Mucosal action

IAH AC Gastrointestinal Disease

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