IAH AC Gastrointestinal Disease

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The antigenic response in the gut • Is always geared towards tolerance and anti-inflammation • Mediated by the Th2, Th3 helper cells and the T suppressor cells • The activation of this is dependent on the • Concentration of the antigen • Type of antigen presenting cell • Specific antigen presenting cells will take up homeopathic dilutions in D1-14 dilution, which will induce TH3 cells and secrete TGF-beta © <strong>IAH</strong> 2007 8 By nature, and for obvious reasons the immune reaction in the gut is geared towards a TH2 type response, thus a tolerant response. Furthermore, the gut has the most of the inducible type T reg cells, namely TH3 cells. These cells are stimulated in the gut lining when the concentration and the frequency of the antigen exposure is very short and very low concentration. This has been seen by Heine and his co workers to be the case with low diluted plant material, such as Traumeel and certain suis organ extracts. Schmolz, Manfred; Heine, Hartmut Homöopathische Substanzen aus der Antihomotoxischen Medizin modulieren die Synthese von TGF-ß1 in menschlichen Vollblutkulturen. Biologische Medizin 2001; nr 2 61-65 This natural reaction towards tolerance ensures that we do not make an inflammatory response to the food we eat, which contains substances which we do not necessarily use. However when the integrity of the gut lining is disturbed, the amount of antigen that could enter into the body is so high that it can cause an inflammatory response. Some patients will be genetically predisposed to generate an inflammatory response, rather than the normal tolerant response in the gut. This will predispose then to diseases like Crohn’s disease. A large body of research is now available regarding these individual differences in the immune response in the gut, and forms the part in the quest to find the aetiology of inflammatory bowel disease as well as to design new treatment strategies. 8
Breast is best!! Photos: WHO Ethiopia © <strong>IAH</strong> 2007 9 The gut lining in an infant is wide open to allow for the absorption of vital factors in breast milk, such as transfer factors amongst others. It closes at about 20 months when intestinal permeability is fully established under physiological conditions. Till then, breast milk will also gear the gut immune response towards tolerance, so that breast fed babies are known to have fewer allergies in later life. If a baby is fed foreign protein, like the casein in cows milk, for instance, too early allergy will ensue. 9
Page 1 and 2: Gastrointestinal Disease IAH AC Gas
Page 3 and 4: The intestinal mucosa • Has the p
Page 5 and 6: The mucosal barrier • From the ga
Page 7: The function of the mucosal lining
Page 11 and 12: Environment © IAH 2007 11 The gut
Page 13 and 14: Mucosal distress (2) • Examples,
Page 15 and 16: Vicious cycles of changed permeabil
Page 17 and 18: Lactose intolerance, not immune-reg
Page 19 and 20: Food allergies Positive skin test P
Page 21 and 22: II. Liver overload • All the bloo
Page 23 and 24: Gut milieu luminal contents and pH
Page 25 and 26: IV. Low grade pancreatitis liver To
Page 27 and 28: Homotoxicology approach © IAH 2007
Page 29 and 30: Oral tolerance and immunological by
Page 31 and 32: Oral tolerance and immunological by
Page 33 and 34: Systemic Gastroenterology Antihomot
Page 35 and 36: Aphthous ulceration • Painful ulc
Page 37 and 38: Gastroesophageal reflux disease (GE
Page 39 and 40: Treatment of gastroesophageal reflu
Page 41 and 42: The composition of Gastricumeel 4.
Page 43 and 44: Peptic ulcer disease • Gastric ul
Page 45 and 46: Treatment • Often an adjuvant tre
Page 47 and 48: Peptic ulceration Symptomatic Basic
Page 49 and 50: Cholecystitis and gall stones • I
Page 51 and 52: Spascupreel tablets - injection sol
Page 53 and 54: Viral hepatitis • Hepatitis virus
Page 55 and 56: Hepatitis Symptomatic Basic medicat
Page 57 and 58: Functional disturbances of motility
Page 59 and 60:
Diarrhea • Increase secretion of
Page 61 and 62:
Actions of Diarrheel Mucosal action
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IAH AC Gastrointestinal Disease

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