What Really Causes Alzheimer's Disease - Soil and Health Library

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poor source of minerals, such as calcium and magnesium. As previously pointed out, the average British and North American diet contains less than half the calcium and magnesium required to avoid the associated deficiency illnesses, including Alzheimer’s disease. The most effective way to address this problem is to eat many of the mineral enriched foods. These include salmon, sardines, broccoli, spinach, and bok choy, for example, which are all high in calcium. 29-30 Pumpkin seeds, almonds, Brazil nuts, and whole grain brown rice are good sources of magnesium. 31 Certain supplements, especially mineral ascorbates, also are excellent sources of both calcium and magnesium. Alacer Corporation, Foothill Ranch, California, a company with which I have no financial associations, provides excellent mineral ascorbate products. One tablet of Super-Gram II, for example, contains 4 percent of calcium and 8 percent of magnesium recommended daily allowance. Emer’gen-C is a fizzing drink mix that is pleasant to take when added to water. It provides 1,000 mg of vitamin C and 32 mineral complexes, including calcium and magnesium. Alacer’s products were used in the joint Committee on World Health and Russian research projects that produced a marked reversal of memory loss in the elderly. 32-34 REFERENCES 1. Genetics and the Alzheimer’s Diseases. Chromosome 19 (AD2). http:// www.macalester.edu/~psych/whathap/UBNRP/alzheimer.chrom 19.html 2. Foster, H.D. (2000). How aluminum causes Alzheimer’s disease: The implications for prevention and treatment of Foster’s multiple antagonist hypothesis. Journal of Orthomolecular Medicine, 15(1), 21-51. 3. Sarin, S., Gupta, V., and Gill, K.D. (1997). Alteration in lipid composition and neuronal injury in primates following chronic aluminum exposure. Biological Trace Element Research, 59(1-3), 133-143. xiii
4. Golub, M.S., and Tarara, R.P. (1999). Morphometric studies of myelination in the spinal cord of mice exposed developmentally to aluminum. Neurotoxicology, 20(6), 953-959. 5. Verstraeten, S.V., Keen, C.L., Golub, M.S., and Oteiza, P.I. (1998). Membrane composition can influence the rate of Al3+-mediated lipid oxidation: Effect of galactolipids. Biochemical Journal, 333(Pt 3), 833-838. 6. Genetics and the Alzheimer’s diseases chromosome, 19(AD2). http://www. macalester.ed~psych/whathap/UBNRP/alzheimer/chrom19.html 7. Velez-Pardo, C., Jimenez Del Rio, M., and Lopera, E. (1998). Familial Alzheimer’s disease: Oxidative stress, beta-amyloid, presenilins, and cell death. General Pharmacology, 31(5), 675-681. 8. Khachaturian, Z.S. (1997). Plundered memories. The Sciences, 37(4), 20-25. 9. Guo, Q., Sebastian, L., Sopher, B.L., Miller, M.W., Ware, C.B., Martin, G.M., and Mattson, M.P. (1999). Increased vulnerability of hippocampal neurons from presenilin-1 mutant knock-in mice to amyloid beta-peptide toxicity: Central roles of superoxide production and caspase activation. Journal of Neurochemistry, 72(3), 1019- 1029. 10. Haass, C., and Baumeister, R. (1998). What do we learn from a few familial Alzheimer’s disease cases? Journal of Neural Transmission. Supplementum, 54, 137-145. 11. Foster, H.D. (1994). Health and the physical environment: The challenge of global change. In M.V. Hayes, L.T. Foster, and H.D. Foster (Eds.), The determinants of population health: A critical assessment (pp. 73-120). Victoria, BC: University of Victoria. Department of Geography, Western Geographical Series Vol. 39. 12. Elsom, D.M. (1992). Atmospheric pollution: A global problem. Oxford: Basil Blackwell. 13. Marier, J.R., with Neri, L.C., and Anderson, T.W. (1979). Water hardness, human health, and the importance of magnesium. Ottawa: National Research Council of Canada. NRC Associate Committee on Scientific Criteria for Environmental Quality. NRCC No. 17581. 14. Peak Performance. Magnesium: Why magnesium matters to athletes. http://www.pponline.co.uk/encyc/magnesium.html 15. Marier et al., op.cit. 16. Vogt, T. (1986). Water quality and health. Study of a possible relationship between aluminum in drinking water and dementia (in Norwegian, English summary). Sosiale Og Okonomiske Studier, 61, Statistisk Oslo-Kongsvinger, 60-63. xiv
Page 2 and 3: What Really Causes Alzheimer’s Di
Page 4 and 5: Dedicated to Grey Friar’s Bobby i
Page 6 and 7: ACKNOWLEDGEMENTS The unusual cover
Page 8 and 9: WHAT REALLY CAUSES ALZHEIMER’S DI
Page 10 and 11: mutations involved in promoting the
Page 12 and 13: the amount of sediment in the water
Page 16 and 17: 17. Flaten, T.P. (1990). Geographic
Page 18 and 19: TABLE OF CONTENTS 1 Who Was I? ....
Page 20 and 21: WHO WAS I? 1 LEAR: FOOL: Does any h
Page 22 and 23: population. 8-9 David Shenk, 10 in
Page 24 and 25: saw this as a death sentence. “Te
Page 26 and 27: patients deteriorate. That is, the
Page 28 and 29: REFERENCES 1. Henderson, A.S. (1986
Page 30 and 31: 35. Garruto, R.M., Swyt, C., Yanagi
Page 32 and 33: IN THE BEGINNING: A HISTORY 2 I am
Page 34 and 35: In 1901 there was certainly nothing
Page 36 and 37: falciparum, which causes malaria, a
Page 38 and 39: techniques, medical researchers mus
Page 40 and 41: USA age-adjusted mortality rates fo
Page 42 and 43: 14. Chiu, H.F.K., and Zhang, M. (20
Page 44 and 45: THE BRAIN DRAIN: PATHOLOGY AND GENE
Page 46 and 47: with other threads of tau and becom
Page 48 and 49: The most extreme of the geneticists
Page 50 and 51: SUMMARY Major pathologic changes oc
Page 52 and 53: PUTTING THE DISEASE IN ITS PLACE 4
Page 54 and 55: smoking, over-consumption of alcoho
Page 56 and 57: eported more commonly from Russia,
Page 58 and 59: These differences in the spatial di
Page 60 and 61: years and older, the dementia preva
Page 62 and 63: REFERENCES 1. Hippocrates On Airs,
Page 64 and 65:
Alzheimer’s disease in two commun
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BIOCHEMICAL ABNORMALITIES 5 What we
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aspartate, normally exist in low co
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types of glutamate receptors in the
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There is also growing evidence that
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of Alzheimer’s disease and so res
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SUMMARY It is apparent from this sh
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14. Kirschmann, J.D. with Dunn, L.J
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39. Symons, M.C.R., and Gutteridge,
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OTHER RISKS, FURTHER HAZARDS 6 I ca
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Cholesterol Whether or not high cho
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Homocysteine The essential amino ac
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placebos. As has been described ear
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may be stronger in women. Gustafson
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disease. The first of these followe
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Head Trauma Controversy continues o
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Osteoporosis A recent longitudinal
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Aging If, as has been previously ar
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REFERENCES 1. Fotuhi, M. (2003). Th
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25. Lim, G.P., Chu, T., Yang, F., B
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54. Ibid. 55. Wilson, R.S., Barnes,
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78. D’Alessandro, R., Gallassi, R
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DOWN TO EARTH: LOCATION, LOCATION,
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oad salt was used extensively and s
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of aluminum and potassium) to staun
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that, within psychiatric nursing ho
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The importance of monomeric (single
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would also promote this form of dem
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correctly able to identify whether
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and teeth. As a consequence, under
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and coworkers 82 also established t
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17. Crapper, D.R., Krishnan, S.S.,
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45. Perl, D.P., and Good, P.F. (198
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78. Jacqmin, H., Commenges, D., Let
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CHECKING OUT ALTERNATIVES 8 Knowing
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in Japan, China, and Korea. Some of
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All appear favourable and Galantami
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changes. These observations may exp
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and behaviour. The reason for this
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15. Thal, L.J., Salmon, D.P., Laske
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43. Pettegrew, J.W., Klunk, W.E., P
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CONVENTIONAL MEDICAL WISDOM 9 We’
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expected, a deficiency of this neur
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There may be a case for the use of
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of Alzheimer’s patients. 28 Birks
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M., Thomas, R., and Thal, L.J. (200
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BACK FROM THE ABYSS 10 Abide with m
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Louis Blank could no longer recogni
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UNDERSTANDING THE SYSTEM: DEMENTIA
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gene that is responsible. 3 There i
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misfolded proteins that have the un
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those who do not carry the APO E4 a
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REFERENCES 1. Hoffman, R. (2003). W
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IT TAKES TWO (OR MORE) TO TANGO 12
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and increased risk for cardiovascul
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of drinking water and this form of
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These high levels of aluminum were
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acid and elevated aluminum. 40 Unde
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accompanied by a build-up of both b
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11. Bishop and Waldholz, op. cit. 1
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changes in calmodulin-implications
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A BARRIER TO ANTAGONISM 13 It all l
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aluminum enters neurons and alone i
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It is at this level that the geneti
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REFERENCES 1. Koestler, A. (1959).
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OTHER ENZYMES - FURTHER PROBLEMS 14
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mechanisms for triggering and direc
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of the phospholipids that concentra
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neurofibrillary tangles of Alzheime
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REFERENCES 1. Heinlein, R. cited by
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lets of patients with Alzheimer’s
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THE ACID TEST: DO THE PIECES FIT? 1
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Table 1: The Pieces of the Jigsaw P
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Chapter 8. Checking Out Alternative
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Alzheimer’s patients are very glu
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There is no doubt that, globally, s
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genetic aberration can remove beta-
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also that diabetics would benefit f
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We do not need a giant, time wastin
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Table 2: Alzheimer’s Disease and
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this process occurs more slowly if
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patients. 83 It has been establishe
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sampled materials established that
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damage before Alzheimer’s symptom
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Not only is the aluminum burden of
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phosphofructokinase. All the necess
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REFERENCES 1. Brown, S.R. (2003). S
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27. Kawahara, M., Kato, M., and Kur
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adenylate cyclase, guanylate cyclas
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adrenergic hypersensitivity and emo
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115. Marlowe, M., and Bliss, L.B. (
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GOOD-BYE TO THE LONG GOOD-BYE 16
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It would seem to be in the best int
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cancer, and diabetes mellitus. 16 S
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in the Developed World, by governme
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poor source of minerals, including
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SUMMARY There is currently a global
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25. Peak Performance, op.cit. 26. L
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A Index A1-calmodulin complex 159 a
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lood pressure 63-66, 80-81, 208 blo
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E Early Onset Alzheimer’s disease
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hardening of the arteries 7 harmful
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miners 97, 108 MIRAGE 74, 85, 223 M
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etired 1 retrogenesis vii-viii, 2-3
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vitamin B 12 67, 70, 147, 203 vitam
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of the boards of the Journal of Ort
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