What Really Causes Alzheimer's Disease - Soil and Health Library

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What Really Causes Alzheimer’s Disease HAROLD D. FOSTER A free copy of this book is available at www.hdfoster.com. “What really causes AIDS” and “What really causes schizophrenia” also can be downloaded at this website. i
© 2004 by Harold D. Foster. All rights reserved. No part of this publication may be reproduced, stored in a retrieval system, or transmitted, in any form or by an means, electronic, mechanical, photocopying, recording, or otherwise, without the written prior permission of the author. ii
Page 4 and 5: Dedicated to Grey Friar’s Bobby i
Page 6 and 7: ACKNOWLEDGEMENTS The unusual cover
Page 8 and 9: WHAT REALLY CAUSES ALZHEIMER’S DI
Page 10 and 11: mutations involved in promoting the
Page 12 and 13: the amount of sediment in the water
Page 14 and 15: poor source of minerals, such as ca
Page 16 and 17: 17. Flaten, T.P. (1990). Geographic
Page 18 and 19: TABLE OF CONTENTS 1 Who Was I? ....
Page 20 and 21: WHO WAS I? 1 LEAR: FOOL: Does any h
Page 22 and 23: population. 8-9 David Shenk, 10 in
Page 24 and 25: saw this as a death sentence. “Te
Page 26 and 27: patients deteriorate. That is, the
Page 28 and 29: REFERENCES 1. Henderson, A.S. (1986
Page 30 and 31: 35. Garruto, R.M., Swyt, C., Yanagi
Page 32 and 33: IN THE BEGINNING: A HISTORY 2 I am
Page 34 and 35: In 1901 there was certainly nothing
Page 36 and 37: falciparum, which causes malaria, a
Page 38 and 39: techniques, medical researchers mus
Page 40 and 41: USA age-adjusted mortality rates fo
Page 42 and 43: 14. Chiu, H.F.K., and Zhang, M. (20
Page 44 and 45: THE BRAIN DRAIN: PATHOLOGY AND GENE
Page 46 and 47: with other threads of tau and becom
Page 48 and 49: The most extreme of the geneticists
Page 50 and 51: SUMMARY Major pathologic changes oc
Page 52 and 53:
PUTTING THE DISEASE IN ITS PLACE 4
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smoking, over-consumption of alcoho
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eported more commonly from Russia,
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These differences in the spatial di
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years and older, the dementia preva
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REFERENCES 1. Hippocrates On Airs,
Page 64 and 65:
Alzheimer’s disease in two commun
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BIOCHEMICAL ABNORMALITIES 5 What we
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aspartate, normally exist in low co
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types of glutamate receptors in the
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There is also growing evidence that
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of Alzheimer’s disease and so res
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SUMMARY It is apparent from this sh
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14. Kirschmann, J.D. with Dunn, L.J
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39. Symons, M.C.R., and Gutteridge,
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OTHER RISKS, FURTHER HAZARDS 6 I ca
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Cholesterol Whether or not high cho
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Homocysteine The essential amino ac
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placebos. As has been described ear
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may be stronger in women. Gustafson
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disease. The first of these followe
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Head Trauma Controversy continues o
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Osteoporosis A recent longitudinal
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Aging If, as has been previously ar
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REFERENCES 1. Fotuhi, M. (2003). Th
Page 102 and 103:
25. Lim, G.P., Chu, T., Yang, F., B
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54. Ibid. 55. Wilson, R.S., Barnes,
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78. D’Alessandro, R., Gallassi, R
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DOWN TO EARTH: LOCATION, LOCATION,
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oad salt was used extensively and s
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of aluminum and potassium) to staun
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that, within psychiatric nursing ho
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The importance of monomeric (single
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would also promote this form of dem
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correctly able to identify whether
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and teeth. As a consequence, under
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and coworkers 82 also established t
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17. Crapper, D.R., Krishnan, S.S.,
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45. Perl, D.P., and Good, P.F. (198
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78. Jacqmin, H., Commenges, D., Let
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CHECKING OUT ALTERNATIVES 8 Knowing
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in Japan, China, and Korea. Some of
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All appear favourable and Galantami
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changes. These observations may exp
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and behaviour. The reason for this
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15. Thal, L.J., Salmon, D.P., Laske
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43. Pettegrew, J.W., Klunk, W.E., P
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CONVENTIONAL MEDICAL WISDOM 9 We’
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expected, a deficiency of this neur
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There may be a case for the use of
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of Alzheimer’s patients. 28 Birks
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M., Thomas, R., and Thal, L.J. (200
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BACK FROM THE ABYSS 10 Abide with m
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Louis Blank could no longer recogni
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UNDERSTANDING THE SYSTEM: DEMENTIA
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gene that is responsible. 3 There i
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misfolded proteins that have the un
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those who do not carry the APO E4 a
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REFERENCES 1. Hoffman, R. (2003). W
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IT TAKES TWO (OR MORE) TO TANGO 12
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and increased risk for cardiovascul
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of drinking water and this form of
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These high levels of aluminum were
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acid and elevated aluminum. 40 Unde
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accompanied by a build-up of both b
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11. Bishop and Waldholz, op. cit. 1
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changes in calmodulin-implications
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A BARRIER TO ANTAGONISM 13 It all l
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aluminum enters neurons and alone i
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It is at this level that the geneti
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REFERENCES 1. Koestler, A. (1959).
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OTHER ENZYMES - FURTHER PROBLEMS 14
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mechanisms for triggering and direc
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of the phospholipids that concentra
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neurofibrillary tangles of Alzheime
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REFERENCES 1. Heinlein, R. cited by
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lets of patients with Alzheimer’s
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THE ACID TEST: DO THE PIECES FIT? 1
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Table 1: The Pieces of the Jigsaw P
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Chapter 8. Checking Out Alternative
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Alzheimer’s patients are very glu
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There is no doubt that, globally, s
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genetic aberration can remove beta-
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also that diabetics would benefit f
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We do not need a giant, time wastin
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Table 2: Alzheimer’s Disease and
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this process occurs more slowly if
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patients. 83 It has been establishe
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sampled materials established that
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damage before Alzheimer’s symptom
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Not only is the aluminum burden of
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phosphofructokinase. All the necess
Page 236 and 237:
REFERENCES 1. Brown, S.R. (2003). S
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27. Kawahara, M., Kato, M., and Kur
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adenylate cyclase, guanylate cyclas
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adrenergic hypersensitivity and emo
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115. Marlowe, M., and Bliss, L.B. (
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GOOD-BYE TO THE LONG GOOD-BYE 16
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It would seem to be in the best int
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cancer, and diabetes mellitus. 16 S
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in the Developed World, by governme
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poor source of minerals, including
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SUMMARY There is currently a global
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25. Peak Performance, op.cit. 26. L
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A Index A1-calmodulin complex 159 a
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lood pressure 63-66, 80-81, 208 blo
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E Early Onset Alzheimer’s disease
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hardening of the arteries 7 harmful
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miners 97, 108 MIRAGE 74, 85, 223 M
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etired 1 retrogenesis vii-viii, 2-3
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vitamin B 12 67, 70, 147, 203 vitam
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of the boards of the Journal of Ort
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