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European Resuscitation Council Guidelines for Resuscitation ... - CPR

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18 de 0ctubre de 2010 www.elsuapdetodos.com1308 C.D. Deakin et al. / <strong>Resuscitation</strong> 81 (2010) 1305–1352patient care and prevent futile <strong>CPR</strong> attempts (see Section 10). 145Medical emergency teams may have an important role in improvingend-of-life and DNAR decision-making. 142,146–148<strong>Guidelines</strong> <strong>for</strong> prevention of in-hospital cardiac arrestHospitals should provide a system of care that includes: (a) staffeducation about the signs of patient deterioration, and the rationale<strong>for</strong> rapid response to illness, (b) appropriate and regular vitalsigns monitoring of patients, (c) clear guidance (e.g., via calling criteriaor early warning scores) to assist staff in the early detectionof patient deterioration, (d) a clear, uni<strong>for</strong>m system of calling <strong>for</strong>assistance, and (e) an appropriate and timely clinical response tocalls <strong>for</strong> assistance. 5 The following strategies may prevent avoidablein-hospital cardiac arrests.1. Provide care <strong>for</strong> patients who are critically ill or at risk of clinicaldeterioration in appropriate areas, with the level of careprovided matched to the level of patient sickness.2. Critically ill patients need regular observations: each patientshould have a documented plan <strong>for</strong> vital signs monitoring thatidentifies which variables need to be measured and the frequencyof measurement according to the severity of illness orthe likelihood of clinical deterioration and cardiopulmonaryarrest. Recent guidance suggests monitoring of simple physiologicalvariables including pulse, blood pressure, respiratoryrate, conscious level, temperature and arterial blood oxygensaturation by pulse oximetry (SpO 2 ). 26,1493. Use a track-and-trigger system (either ‘calling criteria’ or earlywarning system) to identify patients who are critically ill and,or at risk of clinical deterioration and cardiopulmonary arrest.4. Use a patient charting system that enables the regular measurementand recording of vital signs and, where used, earlywarning scores.5. Have a clear and specific policy that requires a clinical responseto abnormal physiology, based on the track and trigger systemused. This should include advice on the further clinical managementof the patient and the specific responsibilities of medicaland nursing staff.6. The hospital should have a clearly identified response to criticalillness. This may include a designated outreach service orresuscitation team (e.g., MET, RRT system) capable of respondingin a timely fashion to acute clinical crises identified by thetrack-and-trigger system or other indicators. This service mustbe available 24 h per day. The team must include staff with theappropriate acute or critical care skills.7. Train all clinical staff in the recognition, monitoring and managementof the critically ill patient. Include advice on clinicalmanagement while awaiting the arrival of more experiencedstaff. Ensure that staff know their role(s) in the rapid responsesystem.8. Hospitals must empower staff of all disciplines to call <strong>for</strong> helpwhen they identify a patient at risk of deterioration or cardiacarrest. Staff should be trained in the use of structured communicationtools to ensure effective handover of in<strong>for</strong>mationbetween doctors, nurses and other healthcare professions.9. Identify patients <strong>for</strong> whom cardiopulmonary arrest is an anticipatedterminal event and in whom <strong>CPR</strong> is inappropriate, andpatients who do not wish to be treated with <strong>CPR</strong>. Hospitalsshould have a DNAR policy, based on national guidance, whichis understood by all clinical staff.10. Ensure accurate audit of cardiac arrest, “false arrest”, unexpecteddeaths and unanticipated ICU admissions usingcommon datasets. Audit also the antecedents and clinicalresponse to these events.Prevention of sudden cardiac death (SCD)out-of-hospitalCoronary artery disease is the commonest cause of SCD. Nonischaemiccardiomyopathy and valvular disease account <strong>for</strong> mostother SCD events. A small percentage of SCDs are caused byinherited abnormalities (e.g., Brugada syndrome, hypertrophic cardiomyopathy)or congenital heart disease.Most SCD victims have a history of cardiac disease and warningsigns, most commonly chest pain, in the hour be<strong>for</strong>e cardiacarrest. 150 In patients with a known diagnosis of cardiac disease, syncope(with or without prodrome—particularly recent or recurrent)is as an independent risk factor <strong>for</strong> increased risk of death. 151–161Chest pain on exertion only, and palpitations associated withsyncope only, are associated with hypertrophic cardiomyopathy,coronary abnormalities, Wolff–Parkinson–White, and arrhythmogenicright ventricular cardiomyopathy.Apparently healthy children and young adults who suffer SCDcan also have signs and symptoms (e.g., syncope/pre-syncope, chestpain and palpitations) that should alert healthcare professionals toseek expert help to prevent cardiac arrest. 162–170Children and young adults presenting with characteristic symptomsof arrhythmic syncope should have a specialist cardiologyassessment, which should include an ECG and in most cases anechocardiogram and exercise test. Characteristics of arrhythmicsyncope include: syncope in the supine position, occurring duringor after exercise, with no or only brief prodromal symptoms,repetitive episodes, or in individuals with a family history ofsudden death. In addition, non-pleuritic chest pain, palpitationsassociated with syncope, seizures (when resistant to treatment,occurring at night or precipitated by exercise, syncope, or loudnoise), and drowning in a competent swimmer should raise suspicionof increased risk. Systematic evaluation in a clinic specializingin the care of those at risk <strong>for</strong> SCD is recommended in family membersof young victims of SCD or those with a known cardiac disorderresulting in an increased risk of SCD. 151,171–175 A family history ofsyncope or SCD, palpitations as a symptom, supine syncope andsyncope associated with exercise and emotional stress are morecommon in patients with long QT syndrome (LQTS). 176 In olderadults 177,178 the absence of nausea and vomiting be<strong>for</strong>e syncopeand ECG abnormalities is an independent predictor of arrhythmicsyncope.Inexplicable drowning and drowning in a strong swimmermay be due to LQTS or catecholaminergic polymorphic ventriculartachycardia (CPVT). 179 There is an association between LQTSand presentation with seizure phenotype. 180,181 Guidance has beenpublished <strong>for</strong> the screening of competitive athletes to identify thoseat risk of sudden death. 182www.elsuapdetodos.com4b Prehospital resuscitationEMS personnelThere is considerable variation across Europe in the structureand process of EMS systems. Some countries have adopted almostexclusively paramedic/emergency medical technician (EMT)-basedsystems while other incorporate prehospital physicians to a greateror lesser extent. In adult cardiac arrest, physician presence duringresuscitation, compared with paramedics alone, has been reportedto increase compliance with guidelines 183,184 and physicians insome systems can per<strong>for</strong>m advanced resuscitation proceduresmore successfully. 183,185–188 When compared within individualsystems, there are contradictory findings with some studies suggestingimproved survival to hospital discharge when physiciansare part of the resuscitation team 189–192 and other studies suggest-

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