Clinical Aspects of Hyponatremia & Hypernatremia - UT Southwestern

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Diabetes Insipidus (DI)• Excessive water loss bythe kidney• Inability of kidney toconcentrate the urine– Deficiency ofAntidiuretic Hormone(ADH)–Central DI– Renal resistance toADHADHNephrogenic DI– Resistance toADHCentral DI– Lack ofADHH 2 O
Nephrogenic Diabetes Insipidus:Common Causes• Many chronic renal diseases• Electrolyte disorders• High Blood Calcium Hypercalcemia• Low Blood Potassium Hypokalemia• Drugs– including Lithium, AMP-B, Demeclocycline, Gentamicin, cisplatin, others• Dietary abnormalities, e.g. decreased NaCl or proteinintake
Page 1 and 2: Clinical Aspects of Hyponatremia &H
Page 3 and 4: Case Presentation: Physical Examina
Page 5 and 6: Case Presentation: Laboratory II•
Page 7 and 8: Polyuria: Differential Diagnosis•
Page 9: Consequences of Hypercalcemia:Renal
Page 13 and 14: Pathogenetic Mechanisms of Hypernat
Page 15 and 16: Nephrogenic Diabetes InsipidusNorma
Page 17 and 18: Hypercalcemia and NephrogenicDiabet
Page 19 and 20: Hypercalcemia and NephrogenicDiabet
Page 21 and 22: [2002] Australian International Law
Page 23 and 24: How to determine the cause of polyu
Page 25: Calculating Water Deficit in Hypern

Clinical Aspects of Hyponatremia & Hypernatremia - UT Southwestern

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