Clinical Aspects of Hyponatremia & Hypernatremia - UT Southwestern

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Differentiating Polyuric States :Dehydration and AVP Stimulation TestsConditionUosm MaxdehydrationUosm Maxafter AVP% Change Uosm IncreaseNormal 1068 ± 69 979 ± 79 -9 ± 3 < 9%PsychogenicPolydipsiaPartialCentral DICompleteCentral DINephrogenicDI738 ± 53 780 ± 73 5 ± 2 < 9%438 ± 34 549 ± 28 28 ± 5 > 9% < 50%168 ± 13 445 ± 52 183 ± 41 >50%124 174 42 < 50%Miller et al. Ann. In. Med 73:721, 1970
Calculating Water Deficit in <strong>Hypernatremia</strong>Due to Pure Water Loss• Serum Na 150, Normal Na 140, 70 kg man• Total Body Water Deficit =BW in kg x 0.6 x [150 -140]14070 x 0.6 x [0.06] = 2.8 LTBW factor:Use 0.6 male0.5 female0.45 elderly
Page 1 and 2: Clinical Aspects of Hyponatremia &H
Page 3 and 4: Case Presentation: Physical Examina
Page 5 and 6: Case Presentation: Laboratory II•
Page 7 and 8: Polyuria: Differential Diagnosis•
Page 9 and 10: Consequences of Hypercalcemia:Renal
Page 11 and 12: Nephrogenic Diabetes Insipidus:Comm
Page 13 and 14: Pathogenetic Mechanisms of Hypernat
Page 15 and 16: Nephrogenic Diabetes InsipidusNorma
Page 17 and 18: Hypercalcemia and NephrogenicDiabet
Page 19 and 20: Hypercalcemia and NephrogenicDiabet
Page 21 and 22: [2002] Australian International Law
Page 23: How to determine the cause of polyu

Clinical Aspects of Hyponatremia & Hypernatremia - UT Southwestern

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