Clinical Aspects of Hyponatremia & Hypernatremia - UT Southwestern

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Urine Concentration(mOsm/L)Effect of Osmotic Diuresis on Uosm16001400120010008006004002000NormalPartial Central DIComplete Central DI0 2 4 6 8 10 12 14Solute Excretion (mOsm/min/1.73 m 2 )
How to determine the cause of polyuric states:Water Deprivation and ADH responsiveness• Step 1: Water deprivation• Step 2: Measure Urine Osmolality• Step 3: Administer exogenous dose of ADH• Step 4: Repeat Urine Osmolality Measurement toevaluate response to ADH
Page 1 and 2: Clinical Aspects of Hyponatremia &H
Page 3 and 4: Case Presentation: Physical Examina
Page 5 and 6: Case Presentation: Laboratory II•
Page 7 and 8: Polyuria: Differential Diagnosis•
Page 9 and 10: Consequences of Hypercalcemia:Renal
Page 11 and 12: Nephrogenic Diabetes Insipidus:Comm
Page 13 and 14: Pathogenetic Mechanisms of Hypernat
Page 15 and 16: Nephrogenic Diabetes InsipidusNorma
Page 17 and 18: Hypercalcemia and NephrogenicDiabet
Page 19 and 20: Hypercalcemia and NephrogenicDiabet
Page 21: [2002] Australian International Law
Page 25: Calculating Water Deficit in Hypern

Clinical Aspects of Hyponatremia & Hypernatremia - UT Southwestern

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