Clinical Aspects of Hyponatremia & Hypernatremia - UT Southwestern

More documents

Recommendations

Info

How does Hypercalcemia causeNephrogenic Diabetes Insipidus?• Decreased delivery of solute to the loop of Henle(reduced GFR)• Inhibition of NaCl transport in thethick ascending limb• Inhibition of vasopressin-mediated waterpermeability in the terminal collecting duct
Hypercalcemia and NephrogenicDiabetes InsipidusCa 2+Ca ++• Direct Effect of Calciumon Renal water Handling• Vasopressin-ResistantAVPResistanceFree H 2 0 Excretion• Independent of GFR,PTH, Vitamin D andCalcitonin
Page 1 and 2: Clinical Aspects of Hyponatremia &H
Page 3 and 4: Case Presentation: Physical Examina
Page 5 and 6: Case Presentation: Laboratory II•
Page 7 and 8: Polyuria: Differential Diagnosis•
Page 9 and 10: Consequences of Hypercalcemia:Renal
Page 11 and 12: Nephrogenic Diabetes Insipidus:Comm
Page 13 and 14: Pathogenetic Mechanisms of Hypernat
Page 15 and 16: Nephrogenic Diabetes InsipidusNorma
Page 17: Hypercalcemia and NephrogenicDiabet
Page 21 and 22: [2002] Australian International Law
Page 23 and 24: How to determine the cause of polyu
Page 25: Calculating Water Deficit in Hypern

Clinical Aspects of Hyponatremia & Hypernatremia - UT Southwestern

Create successful ePaper yourself

Delete template?

Save as template?