Clinical Aspects of Hyponatremia & Hypernatremia - UT Southwestern

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Case Presentation: Laboratory IBlood Tests:Sodium 150 mEq/L (Nl 138-142)Chloride 106 mEq/L (nl 95-100)Kidney Function Decreased 50%Calcium 16.5 mg/dl (Nl 8-10)Urine Tests:Specific gravity 1.008 (Nl for dehydration 1.025-1.035),Urine sodium Na 15 mEq/L (low), Osm 280 mOsm (lowfor dehydration)
Case Presentation: Laboratory II• Serum protein electrophoresis: Abnormal amount of pure(monoclonal) immunoglobulin• Bone Marrow Biopsy: Multiple Myeloma- a malignanttumor of plasma (immune system) cells that causes– Monoclonal immunoglobulin excess– high blood calcium by break down of the skeleton.
Page 1 and 2: Clinical Aspects of Hyponatremia &H
Page 3: Case Presentation: Physical Examina
Page 7 and 8: Polyuria: Differential Diagnosis•
Page 9 and 10: Consequences of Hypercalcemia:Renal
Page 11 and 12: Nephrogenic Diabetes Insipidus:Comm
Page 13 and 14: Pathogenetic Mechanisms of Hypernat
Page 15 and 16: Nephrogenic Diabetes InsipidusNorma
Page 17 and 18: Hypercalcemia and NephrogenicDiabet
Page 19 and 20: Hypercalcemia and NephrogenicDiabet
Page 21 and 22: [2002] Australian International Law
Page 23 and 24: How to determine the cause of polyu
Page 25: Calculating Water Deficit in Hypern

Clinical Aspects of Hyponatremia & Hypernatremia - UT Southwestern

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