Clinical Aspects of Hyponatremia & Hypernatremia - UT Southwestern

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Central Diabetes InsipidusNormal PersonWater restrictedPatient with Central DIWater RestrictedCortexH 20H 20HighAVPH 20H 20ZeroAVPH 20H 20M edullaUrine Flow LowUosm 1000Urine Flow HighUosm 200
Nephrogenic Diabetes InsipidusNormal PersonWater restrictedPatient with Nephrogenic DIWater RestrictedCortexH 20H 20HighAVPH 20H 20HighAVPH 20H 20M edullaUrine Flow LowUosm 1000Urine Flow HighUosm 250
Page 1 and 2: Clinical Aspects of Hyponatremia &H
Page 3 and 4: Case Presentation: Physical Examina
Page 5 and 6: Case Presentation: Laboratory II•
Page 7 and 8: Polyuria: Differential Diagnosis•
Page 9 and 10: Consequences of Hypercalcemia:Renal
Page 11 and 12: Nephrogenic Diabetes Insipidus:Comm
Page 13: Pathogenetic Mechanisms of Hypernat
Page 17 and 18: Hypercalcemia and NephrogenicDiabet
Page 19 and 20: Hypercalcemia and NephrogenicDiabet
Page 21 and 22: [2002] Australian International Law
Page 23 and 24: How to determine the cause of polyu
Page 25: Calculating Water Deficit in Hypern

Clinical Aspects of Hyponatremia & Hypernatremia - UT Southwestern

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