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Disordered Eat<strong>in</strong>g, Anxiety 4<br />

This unchecked anxiety may account for <strong>the</strong> escalation <strong>of</strong> risk factors such as<br />

body dissatisfaction and negative affect <strong>in</strong>to disordered eat<strong>in</strong>g, and later <strong>in</strong>to full-blown<br />

eat<strong>in</strong>g disorders. This unchecked anxiety is also positively correlated with avoidant<br />

cop<strong>in</strong>g strategies whereas approach cop<strong>in</strong>g strategies are negatively correlated with<br />

anxiety (Whatley, Foreman, & Richards, 1998), suggest<strong>in</strong>g that ways to manage anxiety<br />

outside <strong>of</strong> food-related cop<strong>in</strong>g mechanisms need to be addressed. Anxiety is a clear<br />

component <strong>of</strong> eat<strong>in</strong>g disorder pathology and should not be overlooked as prevention<br />

programs are be<strong>in</strong>g developed.<br />

Short-Com<strong>in</strong>gs <strong>of</strong> Eat<strong>in</strong>g Disorder Preventions<br />

Unfortunately, recent efforts to f<strong>in</strong>d a reliable prevention for eat<strong>in</strong>g disorders have<br />

failed to address anxiety—<strong>in</strong>stead, most prevention research focuses on <strong>in</strong>creas<strong>in</strong>g <strong>the</strong><br />

<strong>in</strong>formation about <strong>the</strong> risks <strong>of</strong> eat<strong>in</strong>g disorders ra<strong>the</strong>r than provid<strong>in</strong>g high-risk populations<br />

with an <strong>in</strong>teractive approach that changes behavior (Franko and Orosan-We<strong>in</strong>e, 1998).<br />

To date, many eat<strong>in</strong>g disorder preventions have successfully <strong>in</strong>creased <strong>the</strong> participants’<br />

knowledge <strong>of</strong> <strong>the</strong> eat<strong>in</strong>g disorder pathology. However, this strategy has failed to produce<br />

long-term resilience to disordered eat<strong>in</strong>g behavior (Stice and Shaw, 2004). Carter et al.<br />

(1997) <strong>in</strong>tegrated <strong>in</strong>formation sessions concern<strong>in</strong>g <strong>the</strong> effects <strong>of</strong> diet<strong>in</strong>g, <strong>the</strong> development<br />

<strong>of</strong> eat<strong>in</strong>g disorders, society’s pressures to be th<strong>in</strong>, and <strong>the</strong> benefits <strong>of</strong> healthy eat<strong>in</strong>g <strong>in</strong>to<br />

school curriculum; unfortunately, at <strong>the</strong> 6-month follow-up, scores on <strong>the</strong> Eat<strong>in</strong>g Disorder<br />

Exam (EDE-Q) <strong>in</strong>creased, show<strong>in</strong>g that disordered eat<strong>in</strong>g <strong>in</strong>creased. This suggests that<br />

behavioral risk factors, such as those def<strong>in</strong>ed by Patton et al. (1990) <strong>in</strong>clud<strong>in</strong>g “attenuated<br />

eat<strong>in</strong>g symptoms” (such as emotional eat<strong>in</strong>g) and general psychological symptoms (such<br />

as anxiety), show more promise as a target for future eat<strong>in</strong>g disorder prevention (Franko<br />

and Orosan-We<strong>in</strong>e, 1998).<br />

It is time that a prevention is developed that addresses <strong>the</strong> comorbidity <strong>of</strong> anxiety<br />

and eat<strong>in</strong>g disorders <strong>in</strong> order to give high-risk populations new ways to manage <strong>the</strong>ir<br />

anxiety without resort<strong>in</strong>g to disordered eat<strong>in</strong>g.<br />

Written Emotional Disclosure as a Positive Cop<strong>in</strong>g Mechanism<br />

Written emotional disclosure has emerged <strong>in</strong> <strong>the</strong> last decade as a new <strong>the</strong>rapy that<br />

encourages its participants to release negative emotions, events, and concerns through<br />

writ<strong>in</strong>g. This new <strong>the</strong>rapy has been shown to have positive health benefits, both<br />

physically and psychologically. Pennebaker and Susman (1988) found an <strong>in</strong>crease <strong>in</strong><br />

autonomatic nervous system activity when participants <strong>in</strong>hibited <strong>the</strong>ir thoughts or<br />

feel<strong>in</strong>gs, result<strong>in</strong>g <strong>in</strong> a culm<strong>in</strong>ation <strong>of</strong> stress. Written emotional disclosure showed a<br />

significant alleviation <strong>of</strong> this stress. Pennebaker (1997) also used written emotional<br />

disclosure as a <strong>the</strong>rapeutic practice and aga<strong>in</strong> found promis<strong>in</strong>g results <strong>in</strong> <strong>the</strong> participants’<br />

mental and physical health. Because <strong>of</strong> this past research, written emotional disclosure is<br />

ga<strong>in</strong><strong>in</strong>g credibility as a new and robust <strong>the</strong>rapy.<br />

More specifically, because written emotional disclosure focuses on anxietyproduc<strong>in</strong>g<br />

events, it is a positive cop<strong>in</strong>g mechanism. Fris<strong>in</strong>a, Borod, and Lepore (2004)<br />

conducted a meta-analysis on <strong>the</strong> effects <strong>of</strong> written emotional disclosure on cl<strong>in</strong>ical<br />

populations. <strong>The</strong>y found that, among <strong>the</strong> many psychological benefits associated with<br />

written emotional disclosure, a reduction <strong>in</strong> anxiety occurred. Because anxiety goes hand

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