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than 27 studies reveal that only 45%<br />

of all bulimics experience full<br />

recovery after years of treatment<br />

(Steinhausen & Weber, 2009).<br />

Studies of brain functioning in<br />

individuals with anorexia and bulimia<br />

also have shown impairments in the<br />

functioning of the insula and the<br />

anterior cingulate gyrus (Kaye,<br />

2008), areas responsible for<br />

interoceptive awareness, the<br />

integration of sensory information<br />

and emotions, the regulation of<br />

aggressive impulses, as well as<br />

behavioral motivation and<br />

coordination of motor impulses<br />

(Damasio, 2000; Fogel, 2009;<br />

LeDeux, 2002). The insula is also<br />

responsible for assigning reward<br />

value to foods, a function disrupted in<br />

persons with anorexia who respond<br />

differently to taste stimuli (Kaye,<br />

2008). Furthermore, these same areas<br />

of the brain are largely responsible<br />

for the perception of the body-inspace<br />

and when impaired lead to<br />

symptoms of body dysmorphia, at<br />

worst, or body dissatisfaction, at best<br />

(Saxena & Feusner, 2006).<br />

This article explores the adjunctive<br />

use of somatic approaches to restore<br />

the nervous system’s capacity for<br />

autonomic regulation, thereby<br />

eliminating the need for symptoms<br />

believed to serve that function. Eating<br />

disorders are characterized by deficits<br />

in the self-regulation of food intake,<br />

emotion, cognition, and behaviors.<br />

The body is the battlefield for the<br />

uncontrolled emotions, and motions<br />

of incomplete survival responses—<br />

the autonomic nervous system is<br />

either stuck in a dorsal vagal freeze<br />

or sympathetic over-activation (fight<br />

or flight). The war manifests as selfdestructive<br />

behaviors that are,<br />

paradoxically, conditioned attempts<br />

to maintain the body’s autonomic<br />

homeostasis for the purpose of<br />

survival. Dissociation from bodily<br />

experience, leading to a chronic<br />

disregard of the body’s needs, also is<br />

a hallmark of these disorders<br />

regardless of where they fall on the<br />

spectrum – starvation and emaciation<br />

in anorexia, binge-purge cycles in<br />

bulimia, or binge eating and obesity<br />

in the other extreme. Therefore,<br />

therapies such as Somatic<br />

Experiencing® could be critical in<br />

restoring body awareness and<br />

autonomic regulation.<br />

Eating Disorders and Trauma<br />

A review of the literature indicates<br />

that individuals with eating disorders<br />

are more likely than others to have a<br />

history of trauma and, conversely,<br />

those with trauma are more likely to<br />

report disordered eating patterns –<br />

making trauma a risk factor for eating<br />

disorders. Any history of trauma is<br />

correlated with increased levels of<br />

impulsivity and dissociation, both of<br />

which increase symptom severity,<br />

longer length of illness, and poor<br />

prognosis (Briere, 2007; Everill et al.,<br />

1995; Lockwood et al., 2004;<br />

Mantero & Crippa, 2002).<br />

These rates may be higher when<br />

using a broader definition of trauma<br />

that includes any event that taxes the<br />

person’s organismic capacity to<br />

protect their integrity or is perceived<br />

as life threatening. These may have<br />

been pre- and/or peri-natal events, or<br />

chronic patterns of threat such as<br />

abuse, neglect, and invalidation.<br />

Early histories of persons with eating<br />

disorders include injuries to the<br />

attachment system, without which the<br />

neural pathways that mediate affect<br />

regulation cannot develop (Chassler,<br />

1997; O’Kearney, 1996; Schore,<br />

2000, 2002). Because their pathways<br />

between limbic and cortical areas of<br />

the brain are undeveloped, they live<br />

in a constant flux of dysregulation<br />

that includes a dorsal vagal survival<br />

response that makes persons ‘numb’<br />

to most stimuli coming from the gut<br />

to the brain, e.g., hunger and fullness<br />

signals (Porges, 2010).<br />

Living in a world where hunger is<br />

in constant conflict with the ‘thin<br />

ideal’ and where being ‘fat’ in a ‘thin<br />

world’ oppresses young men and<br />

women every day, can be ‘traumatic’<br />

in the sense that it keeps a person in a<br />

physiologically-stressed state of<br />

constant hyper-vigilance. Wanting to<br />

restrict food intake and ‘failing’ leads<br />

people to feel ‘betrayed’ by their own<br />

bodies. When bodily responses fail to<br />

measure up to these impossible<br />

standards, eating disorders brew.<br />

Case Study: Bulimia<br />

In bulimia, food serves to soothe<br />

sympathetic over-activation but<br />

because satiety responses have been<br />

interrupted a binge occurs, leading to<br />

a purging discharge that serves to<br />

expel uncomfortable sensations from<br />

the body. Once purged, the person<br />

experiences the numbness<br />

characteristic of a dorsal vagal<br />

Somatic Psychotherapy Today | Fall 2014 | Volume 4 Number 2 | page 61

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