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Download Update 11 - Update in Anaesthesia - WFSA

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44<strong>Update</strong> <strong>in</strong> <strong>Anaesthesia</strong>loss. The wound site must also be observed every fewm<strong>in</strong>utes to ensure that any bleed<strong>in</strong>g or haematomaformation is noted early. Dra<strong>in</strong>age from surgical dra<strong>in</strong>sshould also be charted.Conscious level should be monitored by observ<strong>in</strong>g thereturn of reflexes such as the eyelash reflex, swallow<strong>in</strong>gand the start of vocalisation and response to commands.Where the patient has undergone regional anaesthesia(sp<strong>in</strong>al or epidural) the height of the block must be assesseduntil it is seen to be regress<strong>in</strong>g. This is most easily testedby measur<strong>in</strong>g the po<strong>in</strong>t at which cold can no longer beappreciated (us<strong>in</strong>g ethyl chloride or ice). It is safer not tosit these patients up too early as marked posturalhypotension can occur.Once the patient is vocalis<strong>in</strong>g and is reasonably awakepa<strong>in</strong> levels should be assessed. Recovery nurses shouldbe capable of adm<strong>in</strong>ister<strong>in</strong>g <strong>in</strong>travenous analgesia andachiev<strong>in</strong>g adequate analgesia should be a primary goal onceairway reflexes have returned. Pa<strong>in</strong> is most easily treatedby adm<strong>in</strong>ister<strong>in</strong>g morph<strong>in</strong>e 1-2 mg aliquots every 3-5m<strong>in</strong>utes until comfortable. It is very unusual to overdosepatients us<strong>in</strong>g this regime, but <strong>in</strong>travenous naloxone shouldbe available. See also <strong>Update</strong> <strong>in</strong> <strong>Anaesthesia</strong> 1997; 7.Supplemental oxygen therapyWhenever possible all patients recover<strong>in</strong>g from anaesthesiashould be given supplemental oxygen (4l/m<strong>in</strong> by face mask).Where facilities are limited, provided the airway andbreath<strong>in</strong>g is monitored closely, young, fit <strong>in</strong>dividuals hav<strong>in</strong>grelatively m<strong>in</strong>or procedures can often recover withoutsupplemental oxygen. However it should be givenwhenever possible <strong>in</strong> the less fit population hav<strong>in</strong>g majorsurgery. <strong>Anaesthesia</strong>, particularly halothane, obtunds, or<strong>in</strong> some cases abolishes, the hypoxic respiratory drive sothat hypoxia no longer stimulates <strong>in</strong>creased ventilation.Coupled with this is a much <strong>in</strong>creased tendency forhypoxaemia to occur due to a variety of reasons <strong>in</strong>clud<strong>in</strong>gairway obstruction due to an obtunded conscious level,hypoventilation secondary to opioids and anaestheticagents, diffusional hypoxia caused by nitrous oxidediffus<strong>in</strong>g <strong>in</strong>to the lungs faster than nitrogen can diffuse <strong>in</strong>the opposite direction and many different causes ofventilation/perfusion mismatch<strong>in</strong>g. These <strong>in</strong>clude atelectasis(absorption collapse, mucus trapp<strong>in</strong>g, prolongedhypoventilation), a decrease <strong>in</strong> functional residual capacitywith anaesthesia and sup<strong>in</strong>e posture, poor mucus clearance(absent/impaired cough reflex and poor ciliary function),and possibly hypovolaemia or pulmonary oedema. Patientsparticularly at risk of postoperative hypoxia <strong>in</strong>clude thosewho have received nitrous oxide or opioids, and thosewith pre-exist<strong>in</strong>g pulmonary disease. Where oxygen canbe adm<strong>in</strong>istered, even 2 litres per m<strong>in</strong>ute via nasalspectacles or face mask may be sufficient to preventdesaturation associated with most of the causes listedabove. It is also important to realise that the tendency forhypoxaemia extends long <strong>in</strong>to the postoperative periodand is particularly likely to occur on the first postoperativenight. If possible, high risk patients undergo<strong>in</strong>g majorsurgery should receive supplemental oxygen for 48-72hours.Monitor<strong>in</strong>g EquipmentThe most useful monitors <strong>in</strong> the recovery area are the pulseoximeter and the sphygmomanometer. The latter isobviously considerably cheaper, more widely available anddoesn’t need electricity to function. It provides valuable<strong>in</strong>formation about a patient’s cardiovascular status.Postoperatively patients are often mildly hypotensive dueto the sedative effects of drugs and the likelihood of bloodloss or <strong>in</strong>traoperative fluid redistribution (coupled withsome degree of dehydration due to preoperative fast<strong>in</strong>g).More marked levels of hypotension (or even moreseriously a downward trend <strong>in</strong> blood pressure) often heraldan unrecognised blood loss, an adverse cardiac event ormay follow sp<strong>in</strong>al anaesthesia. For these reasons it isprudent to monitor blood pressure every five m<strong>in</strong>utes orso until it is stable and with<strong>in</strong> normal limits. This alsodemonstrates why it is important to document the vitalsigns over a period of time, so that these trends can moreeasily be spotted and acted upon.S<strong>in</strong>ce its widespread <strong>in</strong>troduction <strong>in</strong> the 1980’s the pulseoximeter has become one of the ma<strong>in</strong>stays of postanaesthetic monitor<strong>in</strong>g. Where available these mach<strong>in</strong>eswill give a fairly reliable <strong>in</strong>dicator of systemic oxygenation,together with some <strong>in</strong>dication of cardiovascular status.Oxygen saturation levels should rema<strong>in</strong> above 93% anddesaturation below this level <strong>in</strong> recovery is most commonlycaused by airway obstruction and poor or <strong>in</strong>adequateventilation. The presence of a good quality pulsatile signalusually denotes adequate peripheral circulation, althoughvasodilatation and hypotension can still be present, so theblood pressure should still be monitored. See p<strong>11</strong> forfurther read<strong>in</strong>g regard<strong>in</strong>g the use of pulse oximeters.Apart from the above m<strong>in</strong>imal monitor<strong>in</strong>g, furtherassessment must be tailored to a patient’s particular needs.Ur<strong>in</strong>e output should be assessed where an <strong>in</strong>dwell<strong>in</strong>gur<strong>in</strong>ary catheter is sited and ECG monitor<strong>in</strong>g may benecessary where a patient is at risk of arrhythmias.

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